His primary areas of investigation include Immunology, Antigen, Macrophage migration inhibitory factor, In vitro and Molecular biology. John R. David regularly links together related areas like Cytotoxicity in his Immunology studies. His Antigen research is multidisciplinary, incorporating perspectives in Anticorps monoclonal, Mediator, Lymph node, Microbiology and Sephadex.
His biological study spans a wide range of topics, including Tumor necrosis factor alpha, Innate immune system, Macrophage and Proinflammatory cytokine. His study in In vitro is interdisciplinary in nature, drawing from both Delayed hypersensitivity and Guinea pig. His Molecular biology study combines topics in areas such as Cell culture, Lymphokine and Gene expression.
Immunology, Molecular biology, Antigen, In vitro and Antibody are his primary areas of study. Immune system, Macrophage migration inhibitory factor, Schistosoma mansoni, Immunity and Inflammation are the core of his Immunology study. His work in Immune system tackles topics such as Cytokine which are related to areas like Tumor necrosis factor alpha.
The Molecular biology study which covers Biochemistry that intersects with Guinea pig and Stimulation. His study explores the link between Antigen and topics such as Monoclonal antibody that cross with problems in Virology. His biological study deals with issues like Delayed hypersensitivity, which deal with fields such as Cell migration.
His primary scientific interests are in Internal medicine, Oncology, Macrophage migration inhibitory factor, Immunology and Cancer. The concepts of his Internal medicine study are interwoven with issues in Gastroenterology, Endocrinology and Anti-hiv drugs. His Macrophage migration inhibitory factor research includes themes of Pathogenesis, Obstructive Nephropathy, Osteopontin, Cell biology and Proinflammatory cytokine.
The study incorporates disciplines such as Kinetoplastida and Intravital microscopy in addition to Immunology. The various areas that John R. David examines in his Cancer study include Proportional hazards model and Pathology. His Inflammation research incorporates themes from Innate immune system, Macrophage, Granuloma and Leishmania.
His scientific interests lie mostly in Immunology, Macrophage migration inhibitory factor, Cytokine, Inflammation and Internal medicine. His study in Immunology focuses on Leishmaniasis in particular. John R. David combines subjects such as Proinflammatory cytokine, Erythroid Precursor Cells, Pathogenesis and Tumor necrosis factor alpha with his study of Macrophage migration inhibitory factor.
His Cytokine study integrates concerns from other disciplines, such as Acquired immune system, Innate immune system, Pattern recognition receptor, Immunity and Mycobacterium tuberculosis. John R. David has included themes like Granuloma, Macrophage and Intravital microscopy in his Inflammation study. His Internal medicine study combines topics from a wide range of disciplines, such as Endocrinology, Cellular differentiation and Oncology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Delayed hypersensitivity in vitro: its mediation by cell-free substances formed by lymphoid cell-antigen interaction.
John R. David.
Proceedings of the National Academy of Sciences of the United States of America (1966)
MIF regulates innate immune responses through modulation of Toll-like receptor 4
Thierry Roger;John David;Michel P. Glauser;Thierry Calandra.
DELAYED HYPERSENSITIVITY IN VITRO. I. THE SPECIFICITY OF INHIBITION OF CELL MIGRATION BY ANTIGENS.
John R. David;S. Al-Askari;H. S. Lawrence;L. Thomas.
Journal of Immunology (1964)
Macrophage migration inhibitory factor (MIF) sustains macrophage proinflammatory function by inhibiting p53: Regulatory role in the innate immune response
Robert A. Mitchell;Hong Liao;Jason Chesney;Gunter Fingerle-Rowson.
Proceedings of the National Academy of Sciences of the United States of America (2002)
Targeted disruption of migration inhibitory factor gene reveals its critical role in sepsis.
Marcelo Bozza;Abhay R. Satoskar;Guosheng Lin;Bao Lu.
Journal of Experimental Medicine (1999)
ALTERATIONS OF MACROPHAGE FUNCTIONS BY MEDIATORS FROM LYMPHOCYTES
Carl F. Nathan;Manfred L. Karnovsky;John R. David.
Journal of Experimental Medicine (1971)
Molecular cloning of a cDNA encoding a human macrophage migration inhibitory factor.
Weishui Y. Weiser;Patricia A. Temple;Joann S. Witek-Giannotti;Heinz G. Remold.
Proceedings of the National Academy of Sciences of the United States of America (1989)
Damage to Schistosomula of Schistosoma Mansoni Induced Directly by Eosinophil Major Basic Protein
Anthony E. Butterworth;Donald L. Wassom;Gerald J. Gleich;David A. Loegering.
Journal of Immunology (1979)
rK39: A Cloned Antigen of Leishmania chagasi that Predicts Active Visceral Leishmaniasis
R. Badaró;D. Benson;M. C. Eulálio;M. Freire.
The Journal of Infectious Diseases (1996)
A New Method for the Purification of Human Eosinophils and Neutrophils, and a Comparison of the Ability of These Cells to Damage Schistosomula of Schistosoma Mansoni
Mathew A. Vadas;John R. David;Anthony Butterworth;Nancy T. Pisani.
Journal of Immunology (1979)
Profile was last updated on December 6th, 2021.
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