The scientist’s investigation covers issues in Internal medicine, Endocrinology, Neuropeptide Y receptor, Vasoconstriction and Endothelin receptor. His Internal medicine study which covers Cardiology that intersects with Inflammation. He combines subjects such as Catecholamine, Circulatory system and Stimulation, Guanethidine with his study of Neuropeptide Y receptor.
His Vasoconstriction research is multidisciplinary, relying on both Vas deferens, Spleen, Kidney and Forearm. He has researched Endothelin receptor in several fields, including Brachial artery and Endothelin 1. His studies deal with areas such as Pharmacology and Arginase as well as Nitric oxide.
John Pernow mainly investigates Internal medicine, Endocrinology, Cardiology, Ischemia and Nitric oxide. Internal medicine and Arginase are frequently intertwined in his study. Endocrinology connects with themes related to Neuropeptide Y receptor in his study.
His work on Myocardial infarction, Coronary artery disease, Infarct size and St elevation myocardial infarction as part of general Cardiology research is frequently linked to In patient, bridging the gap between disciplines. His Ischemia research integrates issues from Anesthesia and Pharmacology. His Endothelin receptor research incorporates elements of Blockade and Vasodilation.
John Pernow mainly focuses on Internal medicine, Endothelial dysfunction, Pharmacology, Ischemia and Arginase. His study in Internal medicine is interdisciplinary in nature, drawing from both Endocrinology and Cardiology. His research in Endocrinology intersects with topics in Cardiomyopathy and Function.
His biological study spans a wide range of topics, including Diabetes mellitus, Nitric oxide and Soluble guanylyl cyclase. His work on Reperfusion injury as part of his general Ischemia study is frequently connected to Sinus rhythm, thereby bridging the divide between different branches of science. He has researched Arginase in several fields, including Type 2 diabetes and Endothelin 1.
His primary areas of investigation include Endothelial dysfunction, Arginase, Nitric oxide, Ischemia and Type 2 diabetes. His Endothelial dysfunction study integrates concerns from other disciplines, such as Immunology, Enos, Receptor, Signal transduction and Intracellular. His Arginase research is multidisciplinary, relying on both Internal medicine, Endothelin 1 and Endocrinology.
John Pernow does research in Internal medicine, focusing on Cardiac function curve specifically. His Nitric oxide research is multidisciplinary, incorporating perspectives in Hemoglobin, Plethysmograph, Red blood cell, Reactive oxygen species and Nitrosation. The concepts of his Ischemia study are interwoven with issues in Diabetes mellitus, Hemostatic function and Pharmacology.
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The importance of endothelin-1 for vascular dysfunction in cardiovascular disease.
Felix Böhm;John Pernow.
Cardiovascular Research (2007)
Oxygen Therapy in Suspected Acute Myocardial Infarction
Robin Hofmann;Stefan K James;Stefan K James;Tomas Jernberg;Bertil Lindahl.
The New England Journal of Medicine (2017)
Arginase as a potential target in the treatment of cardiovascular disease: reversal of arginine steal?
John Pernow;Christian Jung;Christian Jung.
Cardiovascular Research (2013)
Myocardial infarction mediated by endothelin receptor signaling in hypercholesterolemic mice
Giuseppina Caligiuri;Bernard Levy;John Pernow;Peter Thorén.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Pharmacological possibilities for protection against myocardial reperfusion injury
Qing-Dong Wang;John Pernow;Per-Ove Sjöquist;Lars Rydén.
Cardiovascular Research (2002)
Baroreflex modulation of sympathetic activity and sympathetic neurotransmitters in humans
D. L. Eckberg;R. F. Rea;O. K. Andersson;T. Hedner.
Acta Physiologica Scandinavica (1988)
Guanethidine-sensitive release of neuropeptide Y-like immunoreactivity in the cat spleen by sympathetic nerve stimulation
Jan.M. Lundberg;Anders Änggård;Elvar Theodorsson-Norheim;John Pernow.
Neuroscience Letters (1984)
Glucagon-like peptide-1 relaxes rat conduit arteries via an endothelium-independent mechanism.
Thomas Nyström;Adrian T. Gonon;Åke Sjöholm;John Pernow.
Regulatory Peptides (2005)
Mechanisms underlying pre- and postjunctional effects of neuropeptide Y in sympathetic vascular control.
John Pernow;Alois Saria;Jan M. Lundberg.
Acta Physiologica Scandinavica (1986)
Plasma neuropeptide Y-like immunoreactivity and catecholamines during various degrees of sympathetic activation in man.
John Pernow;Jan M. Lundberg;Lennart Kaijser;Paul Hjemdahl.
Clinical Physiology (2008)
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