Cell biology, Fibroblast growth factor, Molecular biology, Cellular differentiation and Receptor are his primary areas of study. The concepts of his Cell biology study are interwoven with issues in Endocrinology, Basic fibroblast growth factor, Immunology, Internal medicine and Leukemia inhibitory factor. His Fibroblast growth factor research integrates issues from Sonic hedgehog, Limb bud, Flank and Limb development.
His Molecular biology research incorporates themes from FGF4 and Embryogenesis, Blastocyst Inner Cell Mass, Blastocyst. His Cellular differentiation research incorporates elements of Embryonic stem cell, Stem cell, Inner cell mass and Oligodendrocyte. His Receptor study combines topics from a wide range of disciplines, such as Signal transduction, Glycoprotein 130 and Caffeine.
John K. Heath mostly deals with Cell biology, Molecular biology, Fibroblast growth factor, Receptor and Leukemia inhibitory factor. His Cell biology research includes elements of Embryonic stem cell, Endocrinology, Internal medicine and Cellular differentiation. His research on Molecular biology also deals with topics like
His studies deal with areas such as Limb development, Embryo and Apical ectodermal ridge as well as Fibroblast growth factor. His Receptor research is multidisciplinary, incorporating elements of Glycoprotein 130 and Cytokine. His work in Leukemia inhibitory factor addresses subjects such as Ciliary neurotrophic factor, which are connected to disciplines such as Oncostatin M.
John K. Heath mainly investigates Cell biology, Signal transduction, Molecular biology, Phosphorylation and Artificial intelligence. His Cell biology study incorporates themes from Cell adhesion, Fibroblast growth factor receptor, Fibroblast growth factor, Cell cycle and Glycoprotein 130. John K. Heath has researched Fibroblast growth factor in several fields, including Vinculin, Apical ectodermal ridge and Myotome, Somite.
His biological study spans a wide range of topics, including WW domain, Gene family, WWOX and Small interfering RNA. His Phosphorylation research is multidisciplinary, incorporating perspectives in Apoptosis, Downregulation and upregulation and Transfection. His work on Differential evolution as part of general Artificial intelligence study is frequently linked to Complex network, Modularity and Biological network, bridging the gap between disciplines.
Cell biology, Molecular biology, Tyrosine-protein kinase CSK, Signal transduction and Fibroblast growth factor receptor are his primary areas of study. John K. Heath has included themes like Cytotoxic T cell, Antigen-presenting cell, CD40, CD1 and Immunology in his Cell biology study. His research in Molecular biology intersects with topics in Myotome, Somite, Cell adhesion, WW domain and WWOX.
John K. Heath combines subjects such as Fibroblast growth factor receptor 4, Fibroblast growth factor receptor 3, Fibroblast Growth Factor Receptor Substrate 2 and Growth factor receptor with his study of Tyrosine-protein kinase CSK. John K. Heath has researched Signal transduction in several fields, including Function and Receptor complex. His Fibroblast growth factor receptor study combines topics in areas such as SH3 domain, Tyrosine kinase, Receptor tyrosine kinase, Fibroblast growth factor receptor 1 and Fibroblast growth factor receptor 2.
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Inhibition of pluripotential embryonic stem cell differentiation by purified polypeptides
Austin G. Smith;John K. Heath;Deborah D. Donaldson;Gordon G. Wong.
Transforming growth factor beta modulates the expression of collagenase and metalloproteinase inhibitor.
D. R. Edwards;G. Murphy;J. J. Reynolds;S. E. Whitham.
The EMBO Journal (1987)
Mesoderm induction in early Xenopus embryos by heparin-binding growth factors.
J. M. W. Slack;B. G. Darlington;J. K. Heath;S. F. Godsave.
Aggressiveness, hypoalgesia and high blood pressure in mice lacking the adenosine A2a receptor.
Catherine Ledent;Jean-Marie Vaugeois;Serge N. Schiffmann;Thierry Pedrazzini.
PDGF-A signaling is a critical event in lung alveolar myofibroblast development and alveogenesis
Hans Boström;Karen Willetts;Karen Willetts;Milos Pekny;Per Levéen.
Targeted disruption of fibroblast growth factor (FGF) receptor 2 suggests a role for FGF signaling in pregastrulation mammalian development.
Esther Arman;Rebecca Haffner-Krausz;Yali Chen;John K. Heath.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Fibroblast growth factors induce additional limb development from the flank of chick embryos
Martin J Cohn;Juan Carlos Izpisúa-Belmonte;Helen Abud;John K Heath;John K Heath.
Developmental expression of 2ar (osteopontin) and SPARC (osteonectin) RNA as revealed by in situ hybridization
S Nomura;AJ Wills;Edwards;JK Heath.
Journal of Cell Biology (1988)
Oligodendrocyte Population Dynamics and the Role of PDGF In Vivo
Andrew R Calver;Anita C Hall;Wei-Ping Yu;Frank S Walsh.
Defective oligodendrocyte development and severe hypomyelination in PDGF-A knockout mice
Marcus Fruttiger;Linda Karlsson;Anita C. Hall;Alexandra Abramsson.
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