His primary scientific interests are in Pathology, Hemangioma, Surgery, Vascular disease and Genetics. His Pathology research integrates issues from Endothelial stem cell, Angiogenesis, Neovascularization and Involution, Anatomy. His Anatomy research focuses on Soft tissue and how it relates to Dentistry and Hemifacial microsomia.
John B. Mulliken has included themes like Birthmark, Vascular endothelial growth factor A, Stem cell, Endothelium and Vascular malformation in his Hemangioma study. His research investigates the connection with Surgery and areas like Pediatrics which intersect with concerns in Chemotherapy. His Vascular disease research incorporates elements of Vein, Complication, Arteriovenous malformation and Vascular anomaly.
John B. Mulliken focuses on Surgery, Pathology, Anatomy, Hemangioma and Dentistry. His Surgery research is multidisciplinary, incorporating perspectives in Pediatrics and Craniofacial. His Pathology study combines topics in areas such as Venous malformation, Angiogenesis and Somatic cell.
As part of his studies on Anatomy, he frequently links adjacent subjects like Soft tissue. His work carried out in the field of Hemangioma brings together such families of science as Vascular malformation and Angioma. John B. Mulliken works mostly in the field of Dentistry, limiting it down to concerns involving Orthodontics and, occasionally, Deformity.
His main research concerns Pathology, Surgery, Retrospective cohort study, Dentistry and Orthodontics. His study focuses on the intersection of Pathology and fields such as Somatic cell with connections in the field of Mutation, Cancer research and Germline mutation. Surgery is a component of his Palatoplasty and Treatment outcome studies.
John B. Mulliken focuses mostly in the field of Retrospective cohort study, narrowing it down to matters related to Pediatrics and, in some cases, Young adult. As part of the same scientific family, he usually focuses on Dentistry, concentrating on Cranioplasty and intersecting with Cranial bone and Frontal bone. His studies in Hemangioma integrate themes in fields like Lesion and Contractility.
His primary areas of study are Pathology, Mutation, Retrospective cohort study, Lymphatic system and Germline mutation. His work deals with themes such as Anatomy and Venous malformation, which intersect with Pathology. His research in Mutation intersects with topics in Vascular anomaly and Somatic cell.
His Retrospective cohort study study is concerned with Surgery in general. His study looks at the relationship between Germline mutation and fields such as Missense mutation, as well as how they intersect with chemical problems. His Genetics study combines topics from a wide range of disciplines, such as Glomuvenous malformation and Arteriovenous malformation.
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Hemangiomas and vascular malformations in infants and children: a classification based on endothelial characteristics.
John B. Mulliken;Julie Glowacki.
Plastic and Reconstructive Surgery (1982)
Mutations Involving the Transcription Factor CBFA1 Cause Cleidocranial Dysplasia
S Mundlos;S Mundlos;F Otto;C Mundlos;C Mundlos;J.B Mulliken.
Interferon Alfa-2a Therapy for Life-Threatening Hemangiomas of Infancy
R.A.B. Ezekowitz;J.B. Mulliken;J Folkman.
The New England Journal of Medicine (1992)
GRAFTING OF BURNS WITH CULTURED EPITHELIUM PREPARED FROM AUTOLOGOUS EPIDERMAL CELLS
NicholasE. O'Connor;JohnB. Mulliken;Susan Banks-Schlegel;Olaniyi Kehinde.
The Lancet (1981)
Vascular Dysmorphogenesis Caused by an Activating Mutation in the Receptor Tyrosine Kinase TIE2
Miikka Vikkula;Laurence M Boon;Kermit L.Carraway;Jennifer T Calvert.
Cellular markers that distinguish the phases of hemangioma during infancy and childhood.
K. Takahashi;J. B. Mulliken;H. P. W. Kozakewich;R. A. Rogers.
Journal of Clinical Investigation (1994)
Donor-site morbidity after harvesting rib and iliac bone.
Simon W. S. Laurie;Leonard B. Kaban;John B. Mulliken;Joseph E. Murray.
Plastic and Reconstructive Surgery (1984)
A mutation in the homeodomain of the human MSX2 gene in a family affected with autosomal dominant craniosynostosis
Ethylin Wang Jabs;Ulrich Müller;Xiang Li;Liang Ma.
Congenital vascular lesions: Clinical application of a new classification
Mary Collins Finn;Julie Glowacki;John B. Mulliken.
Journal of Pediatric Surgery (1983)
Capillary malformation-arteriovenous malformation, a new clinical and genetic disorder caused by RASA1 mutations.
Iiro Eerola;Laurence M. Boon;John B. Mulliken;Patricia E. Burrows.
American Journal of Human Genetics (2003)
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