D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 132 Citations 60,803 603 World Ranking 1317 National Ranking 780

Overview

What is he best known for?

The fields of study he is best known for:

  • Surgery
  • Internal medicine
  • Pathology

His primary scientific interests are in Pathology, Hemangioma, Surgery, Vascular disease and Genetics. His Pathology research integrates issues from Endothelial stem cell, Angiogenesis, Neovascularization and Involution, Anatomy. His Anatomy research focuses on Soft tissue and how it relates to Dentistry and Hemifacial microsomia.

John B. Mulliken has included themes like Birthmark, Vascular endothelial growth factor A, Stem cell, Endothelium and Vascular malformation in his Hemangioma study. His research investigates the connection with Surgery and areas like Pediatrics which intersect with concerns in Chemotherapy. His Vascular disease research incorporates elements of Vein, Complication, Arteriovenous malformation and Vascular anomaly.

His most cited work include:

  • Hemangiomas and vascular malformations in infants and children: a classification based on endothelial characteristics. (2492 citations)
  • Mutations Involving the Transcription Factor CBFA1 Cause Cleidocranial Dysplasia (1313 citations)
  • Interferon Alfa-2a Therapy for Life-Threatening Hemangiomas of Infancy (930 citations)

What are the main themes of his work throughout his whole career to date?

John B. Mulliken focuses on Surgery, Pathology, Anatomy, Hemangioma and Dentistry. His Surgery research is multidisciplinary, incorporating perspectives in Pediatrics and Craniofacial. His Pathology study combines topics in areas such as Venous malformation, Angiogenesis and Somatic cell.

As part of his studies on Anatomy, he frequently links adjacent subjects like Soft tissue. His work carried out in the field of Hemangioma brings together such families of science as Vascular malformation and Angioma. John B. Mulliken works mostly in the field of Dentistry, limiting it down to concerns involving Orthodontics and, occasionally, Deformity.

He most often published in these fields:

  • Surgery (30.92%)
  • Pathology (19.65%)
  • Anatomy (13.85%)

What were the highlights of his more recent work (between 2011-2021)?

  • Pathology (19.65%)
  • Surgery (30.92%)
  • Retrospective cohort study (9.02%)

In recent papers he was focusing on the following fields of study:

His main research concerns Pathology, Surgery, Retrospective cohort study, Dentistry and Orthodontics. His study focuses on the intersection of Pathology and fields such as Somatic cell with connections in the field of Mutation, Cancer research and Germline mutation. Surgery is a component of his Palatoplasty and Treatment outcome studies.

John B. Mulliken focuses mostly in the field of Retrospective cohort study, narrowing it down to matters related to Pediatrics and, in some cases, Young adult. As part of the same scientific family, he usually focuses on Dentistry, concentrating on Cranioplasty and intersecting with Cranial bone and Frontal bone. His studies in Hemangioma integrate themes in fields like Lesion and Contractility.

Between 2011 and 2021, his most popular works were:

  • Somatic Mosaic Activating Mutations in PIK3CA Cause CLOVES Syndrome (320 citations)
  • Lymphatic and Other Vascular Malformative/Overgrowth Disorders Are Caused by Somatic Mutations in PIK3CA (236 citations)
  • Kaposiform Hemangioendothelioma: Atypical Features and Risks of Kasabach-Merritt Phenomenon in 107 Referrals (188 citations)

In his most recent research, the most cited papers focused on:

  • Surgery
  • Internal medicine
  • Gene

His primary areas of study are Pathology, Mutation, Retrospective cohort study, Lymphatic system and Germline mutation. His work deals with themes such as Anatomy and Venous malformation, which intersect with Pathology. His research in Mutation intersects with topics in Vascular anomaly and Somatic cell.

His Retrospective cohort study study is concerned with Surgery in general. His study looks at the relationship between Germline mutation and fields such as Missense mutation, as well as how they intersect with chemical problems. His Genetics study combines topics from a wide range of disciplines, such as Glomuvenous malformation and Arteriovenous malformation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Hemangiomas and vascular malformations in infants and children: a classification based on endothelial characteristics.

John B. Mulliken;Julie Glowacki.
Plastic and Reconstructive Surgery (1982)

4291 Citations

Mutations Involving the Transcription Factor CBFA1 Cause Cleidocranial Dysplasia

S Mundlos;S Mundlos;F Otto;C Mundlos;C Mundlos;J.B Mulliken.
Cell (1997)

1815 Citations

Interferon Alfa-2a Therapy for Life-Threatening Hemangiomas of Infancy

R.A.B. Ezekowitz;J.B. Mulliken;J Folkman.
The New England Journal of Medicine (1992)

1492 Citations

GRAFTING OF BURNS WITH CULTURED EPITHELIUM PREPARED FROM AUTOLOGOUS EPIDERMAL CELLS

NicholasE. O'Connor;JohnB. Mulliken;Susan Banks-Schlegel;Olaniyi Kehinde.
The Lancet (1981)

1216 Citations

Vascular Dysmorphogenesis Caused by an Activating Mutation in the Receptor Tyrosine Kinase TIE2

Miikka Vikkula;Laurence M Boon;Kermit L.Carraway;Jennifer T Calvert.
Cell (1996)

896 Citations

Cellular markers that distinguish the phases of hemangioma during infancy and childhood.

K. Takahashi;J. B. Mulliken;H. P. W. Kozakewich;R. A. Rogers.
Journal of Clinical Investigation (1994)

877 Citations

Donor-site morbidity after harvesting rib and iliac bone.

Simon W. S. Laurie;Leonard B. Kaban;John B. Mulliken;Joseph E. Murray.
Plastic and Reconstructive Surgery (1984)

823 Citations

A mutation in the homeodomain of the human MSX2 gene in a family affected with autosomal dominant craniosynostosis

Ethylin Wang Jabs;Ulrich Müller;Xiang Li;Liang Ma.
Cell (1993)

770 Citations

Congenital vascular lesions: Clinical application of a new classification

Mary Collins Finn;Julie Glowacki;John B. Mulliken.
Journal of Pediatric Surgery (1983)

714 Citations

Capillary malformation-arteriovenous malformation, a new clinical and genetic disorder caused by RASA1 mutations.

Iiro Eerola;Laurence M. Boon;John B. Mulliken;Patricia E. Burrows.
American Journal of Human Genetics (2003)

713 Citations

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