What is he best known for?
The fields of study he is best known for:
Jeremy C. Mottram spends much of his time researching Cell biology, Biochemistry, Leishmania mexicana, Leishmania and Trypanosoma brucei.
His work carried out in the field of Cell biology brings together such families of science as Autophagy and Caspase, Programmed cell death.
His Leishmania mexicana research integrates issues from Cysteine protease, Amastigote, Molecular biology, Malate dehydrogenase and Virulence.
Jeremy C. Mottram focuses mostly in the field of Leishmania, narrowing it down to topics relating to Virology and, in certain cases, Immunity, Parasitology and Proteasome.
His Trypanosoma brucei research includes elements of Cytokinesis, RNA interference, Trypanosoma, Cell cycle and Trypanosoma cruzi.
He has researched Neuroscience in several fields, including MAP1LC3B, Chaperone-mediated autophagy, BECN1 and Autolysosome.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- The genome of the kinetoplastid parasite, Leishmania major. (1152 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Biochemistry, Leishmania mexicana, Cell biology, Gene and Trypanosoma brucei.
His Cysteine, Enzyme, Kinase and Active site study in the realm of Biochemistry connects with subjects such as Cysteine Proteinase Inhibitors.
His Leishmania mexicana research is multidisciplinary, incorporating perspectives in Molecular biology, Amastigote, Mutant and Cysteine protease.
His Cell biology research incorporates elements of Autophagy, Cell cycle, Cyclin-dependent kinase and Kinetoplast.
His Gene study contributes to a more complete understanding of Genetics.
His study in Trypanosoma brucei is interdisciplinary in nature, drawing from both Metacaspase, RNA interference, Trypanosoma cruzi and Trypanosoma.
He most often published in these fields:
- Biochemistry (37.69%)
- Leishmania mexicana (30.22%)
- Cell biology (33.21%)
What were the highlights of his more recent work (between 2017-2021)?
- Cell biology (33.21%)
- Leishmania (23.51%)
- Gene (25.75%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Cell biology, Leishmania, Gene, Computational biology and Visceral leishmaniasis.
His research in Cell biology intersects with topics in Ubiquitin, Amastigote, Trypanosoma brucei and Cell body membrane.
His research investigates the connection with Leishmania and areas like Function which intersect with concerns in Cell cycle checkpoint, Genome engineering, DNA and Cell cycle.
His Gene research is within the category of Genetics.
He interconnects Caspase and Genome, Contig in the investigation of issues within Computational biology.
Many of his studies involve connections with topics such as Leishmania mexicana and Mutant.
Between 2017 and 2021, his most popular works were:
- Genetically Validated Drug Targets in Leishmania: Current Knowledge and Future Prospects (37 citations)
- Leishmania Genome Dynamics during Environmental Adaptation Reveal Strain-Specific Differences in Gene Copy Number Variation, Karyotype Instability, and Telomeric Amplification. (35 citations)
- Drug Discovery for Kinetoplastid Diseases: Future Directions (32 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in Leishmania, Computational biology, Copy-number variation, Cell biology and Function.
His Leishmania study incorporates themes from Morphogenesis, Gene and Genome.
His study on Genome is covered under Genetics.
His studies deal with areas such as Caspase and Drug discovery as well as Computational biology.
His studies in Copy-number variation integrate themes in fields like Genetic marker, Visceral leishmaniasis and Virology.
His biological study spans a wide range of topics, including Flagellum, Cell body membrane, Trypanosoma brucei, Mutant and Endocytosis.
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