2019 - German National Academy of Sciences Leopoldina - Deutsche Akademie der Naturforscher Leopoldina – Nationale Akademie der Wissenschaften Neurosciences
Hilmar Bading spends much of his time researching Cell biology, Neuroscience, CREB, NMDA receptor and Voltage-dependent calcium channel. His work in Cell biology tackles topics such as Transcription factor which are related to areas like Molecular biology. The concepts of his Neuroscience study are interwoven with issues in R-type calcium channel, T-type calcium channel, P-type calcium channel, Molecular neuroscience and Signalling.
His p300-CBP coactivator family study, which is part of a larger body of work in CREB, is frequently linked to Response element, bridging the gap between disciplines. His NMDA receptor research includes elements of Glutamate receptor, Stimulation and Neuroprotection. His Voltage-dependent calcium channel study is concerned with Calcium in general.
Hilmar Bading mainly investigates Cell biology, Neuroscience, Calcium signaling, Calcium and NMDA receptor. His research in Cell biology intersects with topics in Gene expression, Voltage-dependent calcium channel, Transcription factor, Biochemistry and Regulation of gene expression. His Neuroscience study incorporates themes from Receptor, Neurotransmission and CREB.
His CREB research integrates issues from Coactivator and Endocrinology. His work carried out in the field of Calcium signaling brings together such families of science as Cell nucleus and Nucleus. His studies in NMDA receptor integrate themes in fields like Glutamate receptor and Neuroprotection.
Hilmar Bading focuses on Neuroscience, Cell biology, Neuroprotection, NMDA receptor and Excitotoxicity. He combines subjects such as Synaptic plasticity, Epigenetics and CREB with his study of Neuroscience. The CREB study combines topics in areas such as Calcium in biology, Intracellular, Signal transduction and Drug discovery.
His study in Cell biology is interdisciplinary in nature, drawing from both Gene expression and Calcium, Calcium flux. His Neuroprotection study integrates concerns from other disciplines, such as Glutamate receptor and Membrane potential. His NMDA receptor research is multidisciplinary, relying on both Neurodegeneration, Excitatory postsynaptic potential, Stroke, Neuron death and Brain damage.
His main research concerns Neuroscience, Mitochondrion, Glutamate receptor, Calcium signaling and Cell biology. His work deals with themes such as Synaptic plasticity and FOSB, which intersect with Neuroscience. His Mitochondrion study combines topics in areas such as CREB, Neuroprotection, Neurodegeneration, Pathology and Receptor.
His Glutamate receptor research is multidisciplinary, incorporating elements of Fear conditioning, Central nucleus of the amygdala, Optogenetics, Extinction and Engram. His Calcium signaling research is multidisciplinary, incorporating perspectives in Hippocampal formation, Gating, Memory consolidation and Dendrite. His Cell biology research incorporates elements of Genome, Human genome, Gene expression, Biochemistry and Mitochondrial permeability transition pore.
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Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways
Giles E. Hardingham;Yuko Fukunaga;Hilmar Bading;Hilmar Bading.
Nature Neuroscience (2002)
Regulation of gene expression in hippocampal neurons by distinct calcium signaling pathways
Hilmar Bading;David D. Ginty;Michael E. Greenberg.
Science (1993)
Synaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disorders
Giles E. Hardingham;Hilmar Bading.
Nature Reviews Neuroscience (2010)
Regulation of CREB phosphorylation in the suprachiasmatic nucleus by light and a circadian clock
David D. Ginty;Jon M. Kornhauser;Margaret A. Thompson;Hilmar Bading;Hilmar Bading.
Science (1993)
Distinct functions of nuclear and cytoplasmic calcium in the control of gene expression
Giles E. Hardingham;Sangeeta Chawla;Claire M. Johnson;Hilmar Bading.
Nature (1997)
The Yin and Yang of NMDA receptor signalling.
Giles E. Hardingham;Hilmar Bading.
Trends in Neurosciences (2003)
Stimulation of protein tyrosine phosphorylation by NMDA receptor activation
Hilmar Bading;Michael E. Greenberg.
Science (1991)
Nuclear calcium signaling controls CREB-mediated gene expression triggered by synaptic activity.
Giles E. Hardingham;Fiona J. L. Arnold;Hilmar Bading.
Nature Neuroscience (2001)
CBP: A Signal-Regulated Transcriptional Coactivator Controlled by Nuclear Calcium and CaM Kinase IV
Sangeeta Chawla;Giles E. Hardingham;David R. Quinn;Hilmar Bading.
Science (1998)
Calcium regulation of gene expression in neuronal cells.
Anirvan Ghosh;David D. Ginty;Hilmar Bading;Michael E. Greenberg.
Journal of Neurobiology (1994)
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