D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 46 Citations 18,112 81 World Ranking 12189 National Ranking 902

Research.com Recognitions

Awards & Achievements

2019 - German National Academy of Sciences Leopoldina - Deutsche Akademie der Naturforscher Leopoldina – Nationale Akademie der Wissenschaften Neurosciences

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Neuron
  • Neurotransmitter

Hilmar Bading spends much of his time researching Cell biology, Neuroscience, CREB, NMDA receptor and Voltage-dependent calcium channel. His work in Cell biology tackles topics such as Transcription factor which are related to areas like Molecular biology. The concepts of his Neuroscience study are interwoven with issues in R-type calcium channel, T-type calcium channel, P-type calcium channel, Molecular neuroscience and Signalling.

His p300-CBP coactivator family study, which is part of a larger body of work in CREB, is frequently linked to Response element, bridging the gap between disciplines. His NMDA receptor research includes elements of Glutamate receptor, Stimulation and Neuroprotection. His Voltage-dependent calcium channel study is concerned with Calcium in general.

His most cited work include:

  • Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways (1308 citations)
  • Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways (1308 citations)
  • Regulation of gene expression in hippocampal neurons by distinct calcium signaling pathways (1028 citations)

What are the main themes of his work throughout his whole career to date?

Hilmar Bading mainly investigates Cell biology, Neuroscience, Calcium signaling, Calcium and NMDA receptor. His research in Cell biology intersects with topics in Gene expression, Voltage-dependent calcium channel, Transcription factor, Biochemistry and Regulation of gene expression. His Neuroscience study incorporates themes from Receptor, Neurotransmission and CREB.

His CREB research integrates issues from Coactivator and Endocrinology. His work carried out in the field of Calcium signaling brings together such families of science as Cell nucleus and Nucleus. His studies in NMDA receptor integrate themes in fields like Glutamate receptor and Neuroprotection.

He most often published in these fields:

  • Cell biology (47.37%)
  • Neuroscience (45.11%)
  • Calcium signaling (29.32%)

What were the highlights of his more recent work (between 2016-2021)?

  • Neuroscience (45.11%)
  • Cell biology (47.37%)
  • Neuroprotection (13.53%)

In recent papers he was focusing on the following fields of study:

Hilmar Bading focuses on Neuroscience, Cell biology, Neuroprotection, NMDA receptor and Excitotoxicity. He combines subjects such as Synaptic plasticity, Epigenetics and CREB with his study of Neuroscience. The CREB study combines topics in areas such as Calcium in biology, Intracellular, Signal transduction and Drug discovery.

His study in Cell biology is interdisciplinary in nature, drawing from both Gene expression and Calcium, Calcium flux. His Neuroprotection study integrates concerns from other disciplines, such as Glutamate receptor and Membrane potential. His NMDA receptor research is multidisciplinary, relying on both Neurodegeneration, Excitatory postsynaptic potential, Stroke, Neuron death and Brain damage.

Between 2016 and 2021, his most popular works were:

  • Therapeutic targeting of the pathological triad of extrasynaptic NMDA receptor signaling in neurodegenerations. (72 citations)
  • Networks of Cultured iPSC-Derived Neurons Reveal the Human Synaptic Activity-Regulated Adaptive Gene Program. (34 citations)
  • A Fear Memory Engram and Its Plasticity in the Hypothalamic Oxytocin System. (32 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Neuron
  • Neurotransmitter

His main research concerns Neuroscience, Mitochondrion, Glutamate receptor, Calcium signaling and Cell biology. His work deals with themes such as Synaptic plasticity and FOSB, which intersect with Neuroscience. His Mitochondrion study combines topics in areas such as CREB, Neuroprotection, Neurodegeneration, Pathology and Receptor.

His Glutamate receptor research is multidisciplinary, incorporating elements of Fear conditioning, Central nucleus of the amygdala, Optogenetics, Extinction and Engram. His Calcium signaling research is multidisciplinary, incorporating perspectives in Hippocampal formation, Gating, Memory consolidation and Dendrite. His Cell biology research incorporates elements of Genome, Human genome, Gene expression, Biochemistry and Mitochondrial permeability transition pore.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways

Giles E. Hardingham;Yuko Fukunaga;Hilmar Bading;Hilmar Bading.
Nature Neuroscience (2002)

1728 Citations

Regulation of gene expression in hippocampal neurons by distinct calcium signaling pathways

Hilmar Bading;David D. Ginty;Michael E. Greenberg.
Science (1993)

1370 Citations

Synaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disorders

Giles E. Hardingham;Hilmar Bading.
Nature Reviews Neuroscience (2010)

1335 Citations

Regulation of CREB phosphorylation in the suprachiasmatic nucleus by light and a circadian clock

David D. Ginty;Jon M. Kornhauser;Margaret A. Thompson;Hilmar Bading;Hilmar Bading.
Science (1993)

957 Citations

Distinct functions of nuclear and cytoplasmic calcium in the control of gene expression

Giles E. Hardingham;Sangeeta Chawla;Claire M. Johnson;Hilmar Bading.
Nature (1997)

847 Citations

The Yin and Yang of NMDA receptor signalling.

Giles E. Hardingham;Hilmar Bading.
Trends in Neurosciences (2003)

802 Citations

Stimulation of protein tyrosine phosphorylation by NMDA receptor activation

Hilmar Bading;Michael E. Greenberg.
Science (1991)

660 Citations

Nuclear calcium signaling controls CREB-mediated gene expression triggered by synaptic activity.

Giles E. Hardingham;Fiona J. L. Arnold;Hilmar Bading.
Nature Neuroscience (2001)

579 Citations

CBP: A Signal-Regulated Transcriptional Coactivator Controlled by Nuclear Calcium and CaM Kinase IV

Sangeeta Chawla;Giles E. Hardingham;David R. Quinn;Hilmar Bading.
Science (1998)

524 Citations

Calcium regulation of gene expression in neuronal cells.

Anirvan Ghosh;David D. Ginty;Hilmar Bading;Michael E. Greenberg.
Journal of Neurobiology (1994)

379 Citations

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