His scientific interests lie mostly in Molecular biology, Gene, Transfection, Cancer research and Carcinogenesis. The Molecular biology study combines topics in areas such as Apoptosis, Programmed cell death, Transcription factor, DNA and Gene product. In his work, Cell culture and Cell cycle is strongly intertwined with Basic helix-loop-helix leucine zipper transcription factors, which is a subfield of Programmed cell death.
His work in Gene product addresses subjects such as Cotransformation, which are connected to disciplines such as Fibroblast. He interconnects Antigen and Embryo in the investigation of issues within Gene. His work in Cancer research covers topics such as Cyclin D which are related to areas like Cell biology, Signal transduction, DNA synthesis and Cell cycle checkpoint.
His main research concerns Molecular biology, Cancer research, Cell biology, Gene and Carcinogenesis. His Molecular biology research includes elements of Plasmid, Biochemistry, DNA, Recombinant DNA and Complementary DNA. His Cancer research study integrates concerns from other disciplines, such as Pancreatic cancer, Mutation, Oncogene, Signal transduction and Cell type.
His research integrates issues of Cell cycle, Cyclin A2, Growth factor and Cell growth in his study of Cell biology. The concepts of his Gene study are interwoven with issues in Computational biology and Function. His Carcinogenesis research includes themes of Phenotype, Mediator, Transfection and Chimera.
Hartmut Land mainly focuses on Cancer research, Mutation, Inference, Gene and Carcinogenesis. Hartmut Land has researched Cancer research in several fields, including Pancreatic cancer, Cell growth, Signal transduction, Cell type and Mitochondrion. As part of the same scientific family, Hartmut Land usually focuses on Cell growth, concentrating on Heterochromatin and intersecting with Cell cycle.
His research on Gene frequently connects to adjacent areas such as Computational biology. His studies deal with areas such as Glycolysis, Warburg effect, Glutamine and Citric acid cycle as well as Carcinogenesis. His Regulation of gene expression study combines topics in areas such as Ribosomal RNA, Downregulation and upregulation, Folliculin and Molecular biology.
Cancer research, Cell biology, Carcinogenesis, Gene and Mutation are his primary areas of study. His Cancer research research integrates issues from Cholesterol and Mitochondrion. His Cell biology research incorporates themes from Cancer cell, Cancer, Warburg effect, Glycolysis and Lactate dehydrogenase.
His Carcinogenesis research incorporates elements of Autophagy, Pancreatic cancer, Glutamine, Gene silencing and Citric acid cycle. His Gene study focuses mostly on Cell cycle and Regulation of gene expression. His Mutation research is multidisciplinary, incorporating elements of Cellular differentiation, Bioinformatics, Regulator, Penetrance and Etiology.
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Induction of apoptosis in fibroblasts by c-myc protein
Gerard I. Evan;Andrew H. Wyllie;Christopher S. Gilbert;Trevor D. Littlewood.
Advanced mammalian gene transfer: high titre retroviral vectors with multiple drug selection markers and a complementary helper-free packaging cell line
Jay P. Morgenstern;Hartmut Land.
Nucleic Acids Research (1990)
Tumorigenic conversion of primary embryo fibroblasts requires at least two cooperating oncogenes.
Hartmut Land;Luis F. Parada;Robert A. Weinberg.
Cellular oncogenes and multistep carcinogenesis.
Hartmut Land;Luis F. Parada;Robert A. Weinberg.
Cooperation between gene encoding p53 tumour antigen and ras in cellular transformation
Luis F. Parada;Hartmut Land;Robert A. Weinberg;David Wolf.
Nucleotide sequence of cloned cDNA encoding bovine arginine vasopressin-neurophysin II precursor.
Hartmut Land;Günther Schütz;Hartwig Schmale;Dietmar Richter.
Cooperation between two growth factors promotes extended self-renewal and inhibits differentiation of oligodendrocyte-type-2 astrocyte (O-2A) progenitor cells.
Oliver Bogler;Damian Wren;Susan C. Barnett;Hartmut Land.
Proceedings of the National Academy of Sciences of the United States of America (1990)
High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1.
A Sewing;B Wiseman;A C Lloyd;H Land.
Molecular and Cellular Biology (1997)
Oncogenic activity of the c-Myc protein requires dimerization with Max
Bruno Amati;Mary W. Brooks;Naomi Levy;Trevor D. Littlewood.
Transcriptional activation by the human c-Myc oncoprotein in yeast requires interaction with Max.
Bruno Amati;Stephen Dalton;Mary W. Brooks;Trevor D. Littlewood.
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