What is he best known for?
The fields of study he is best known for:
Clemens Steegborn mainly investigates Biochemistry, Sirtuin, Cell biology, Second messenger system and Soluble adenylyl cyclase.
His is doing research in Mitochondrion, Enzyme, Plasma protein binding, Phosphodiesterase and Adenylyl cyclase, both of which are found in Biochemistry.
His research in Sirtuin is mostly concerned with SIRT3.
His SIRT3 research focuses on subjects like SIRT5, which are linked to Peptide sequence and Citric acid cycle.
His Cell biology research includes elements of Autophagy, Neuroprotection and Synucleinopathies.
As part of one scientific family, Clemens Steegborn deals mainly with the area of Second messenger system, narrowing it down to issues related to the Adenosine triphosphate, and often Cyclase, Guanosine triphosphate, Enzyme activator and ADCY9.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Substrates and Regulation Mechanisms for the Human Mitochondrial Sirtuins Sirt3 and Sirt5 (418 citations)
- Specific interaction of Smn, the spinal muscular atrophy determining gene product, with hnRNP-R and gry-rbp/hnRNP-Q: a role for Smn in RNA processing in motor axons? (253 citations)
What are the main themes of his work throughout his whole career to date?
Clemens Steegborn focuses on Biochemistry, Sirtuin, Soluble adenylyl cyclase, Cell biology and Stereochemistry.
His Biochemistry study frequently draws connections to adjacent fields such as Crystal structure.
Sirtuin is a subfield of NAD+ kinase that Clemens Steegborn explores.
His study in the fields of Mitochondrion and Signal transduction under the domain of Cell biology overlaps with other disciplines such as Peptide binding.
His study in the field of Respiratory chain is also linked to topics like Mitochondrial matrix.
His Stereochemistry research incorporates themes from Hydrolase and Lyase.
He most often published in these fields:
- Biochemistry (63.06%)
- Sirtuin (25.48%)
- Soluble adenylyl cyclase (21.02%)
What were the highlights of his more recent work (between 2015-2021)?
- Biochemistry (63.06%)
- Sirtuin (25.48%)
- Cell biology (19.11%)
In recent papers he was focusing on the following fields of study:
His main research concerns Biochemistry, Sirtuin, Cell biology, NAD+ kinase and Soluble adenylyl cyclase.
As a part of the same scientific study, Clemens Steegborn usually deals with the Sirtuin, concentrating on Mitochondrion and frequently concerns with Reactive oxygen species, Cancer, Cancer cell and Pharmacology.
The concepts of his Cell biology study are interwoven with issues in Autophagy and Transcription factor.
His NAD+ kinase study combines topics from a wide range of disciplines, such as Polymerase and DNA Repair Protein.
His Computational biology study incorporates themes from Chaperone-mediated autophagy, MAP1LC3B, Sequestosome 1, Autophagosome and Physiology.
His Physiology study which covers Programmed cell death that intersects with Autolysosome, BECN1 and DNA repair.
Between 2015 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Sirtuin activators and inhibitors : Promises, achievements, and challenges (125 citations)
- The mechanism of sirtuin 2-mediated exacerbation of alpha-synuclein toxicity in models of Parkinson disease (91 citations)
In his most recent research, the most cited papers focused on:
Clemens Steegborn mostly deals with Biochemistry, Sirtuin, Cell biology, NAD+ kinase and Activator.
He merges many fields, such as Biochemistry and Soluble adenylyl cyclase, in his writings.
While the research belongs to areas of Sirtuin, he spends his time largely on the problem of Mitochondrion, intersecting his research to questions surrounding Electron transport chain, SIRT5, SIRT3 and Xenopus.
As a member of one scientific family, Clemens Steegborn mostly works in the field of Cell biology, focusing on Autophagy and, on occasion, Computational biology, Proteotoxicity and Substantia nigra.
His NAD+ kinase research integrates issues from Polymerase, Energy metabolism, Drug discovery and Gene isoform.
His work carried out in the field of Programmed cell death brings together such families of science as Chaperone-mediated autophagy, MAP1LC3B, Sequestosome 1, Autophagosome and Physiology.
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