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Medicine

D-Index
95
Citations
31327
World Ranking
10112
National Ranking
5207

Overview

Bruce A. Julian is affiliated with the University of Alabama at Birmingham in the United States. Their research primarily focuses on medicine, with a significant concentration in nephrology, immunology, pathology and forensic medicine, rheumatology, and hematology. The scientist has contributed extensively to the study of renal diseases and glomerulopathies, autoimmune bullous skin diseases, systemic lupus erythematosus research, and celiac disease research and management.

Their publication record includes numerous papers, with notable works including:

  • IgA Nephropathy: An Interesting Autoimmune Kidney Disease (2020, The American Journal of the Medical Sciences)
  • Pathogenesis of IgA Nephropathy: Current Understanding and Implications for Development of Disease-Specific Treatment (2021, Journal of Clinical Medicine)
  • Experimental evidence of pathogenic role of IgG autoantibodies in IgA nephropathy (2021, Journal of Autoimmunity)
  • Genetic regulation of serum IgA levels and susceptibility to common immune, infectious, kidney, and cardio-metabolic traits (2022, Nature Communications)
  • Leukemia Inhibitory Factor Signaling Enhances Production of Galactose-Deficient IgA1 in IgA Nephropathy (2020, Kidney Diseases)

The scientist frequently collaborates with other researchers, with coauthors including Jan Novák, Stacy Hall, Dana V. Rizk, Colin Reily, and Zina Moldoveanu.

Bruce A. Julian's work is published mainly in these venues:

  • Journal of the American Society of Nephrology
  • UNC Libraries
  • Journal of Clinical Medicine
  • Kidney International Reports
  • Nature Communications

Their research topics cover a range of areas related to immune and kidney diseases. Key topics include:

  • Renal Diseases and Glomerulopathies
  • Autoimmune Bullous Skin Diseases
  • Systemic Lupus Erythematosus Research
  • Celiac Disease Research and Management
  • Complement system in diseases
  • Vasculitis and related conditions
  • Platelet Disorders and Treatments

Best Publications

  • The Oxford classification of IgA nephropathy: Rationale, clinicopathological correlations, and classification

    Daniel C. Cattran;Rosanna Coppo;H. Terence Cook;John Feehally

  • The Oxford classification of IgA nephropathy: pathology definitions, correlations, and reproducibility

    Ian S.D. Roberts;H. Terence Cook;Stéphan Troyanov;Charles E. Alpers

  • The Pathophysiology of IgA Nephropathy

    Hitoshi Suzuki;Krzysztof Kiryluk;Jan Novak;Zina Moldoveanu

  • Rapid Loss of Vertebral Mineral Density after Renal Transplantation

    B A Julian;D A Laskow;J Dubovsky;E V Dubovsky

  • Genome-wide association study identifies susceptibility loci for IgA nephropathy

    Ali G Gharavi;Krzysztof Kiryluk;Murim Choi;Yifu Li

  • Discovery of new risk loci for IgA nephropathy implicates genes involved in immunity against intestinal pathogens

    Krzysztof Kiryluk;Yifu Li;Francesco Scolari;Simone Sanna-Cherchi

  • Circulating immune complexes in IgA nephropathy consist of IgA1 with galactose-deficient hinge region and antiglycan antibodies

    Milan Tomana;Jan Novak;Bruce A. Julian;Karel Matousovic

  • Naturally Occurring Human Urinary Peptides for Use in Diagnosis of Chronic Kidney Disease

    David M. Good;Petra Zürbig;Àngel Argilés;Hartwig W. Bauer

  • Aberrantly glycosylated IgA1 in IgA nephropathy patients is recognized by IgG antibodies with restricted heterogeneity

    Hitoshi Suzuki;Run Fan;Zhixin Zhang;Rhubell Brown

  • Patients with IgA nephropathy have increased serum galactose-deficient IgA1 levels.

    Z. Moldoveanu;R.J. Wyatt;J.Y. Lee;M. Tomana

  • IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.

    Ali G. Gharavi;Ali G. Gharavi;Yan Yan;Francesco Scolari;F. Paolo Schena

  • Galactose-deficient IgA1 in sera of IgA nephropathy patients is present in complexes with IgG.

    Milan Tomana;Karel Matousovic;Bruce A. Julian;Jiri Radl

  • Geographic Differences in Genetic Susceptibility to IgA Nephropathy: GWAS Replication Study and Geospatial Risk Analysis

    Krzysztof Kiryluk;Yifu Li;Simone Sanna-Cherchi;Simone Sanna-Cherchi;Mersedeh Rohanizadegan

  • Sirolimus reduces the incidence of acute rejection episodes despite lower cyclosporine doses in caucasian recipients of mismatched primary renal allografts: a phase II trial. Rapamune Study Group.

    Barry D. Kahan;Bruce A. Julian;Mark D. Pescovitz;Yves Vanrenterghem

  • Long-term results of a controlled prospective study with transfusion of donor-specific bone marrow in 57 cadaveric renal allograft recipients.

    W. Barber;James Mankin;David Laskow;Mark Deierhoi

  • Angiotensin II stimulates proliferation of normal early erythroid progenitors.

    Michal Mrug;Tomas Stopka;Bruce A. Julian;Jaroslav F. Prchal

  • Clinical proteomics: A need to define the field and to begin to set adequate standards.

    Harald Mischak;Rolf Apweiler;Rosamonde E. Banks;Mark Conaway

  • IgA1-secreting cell lines from patients with IgA nephropathy produce aberrantly glycosylated IgA1

    Hitoshi Suzuki;Zina Moldoveanu;Stacy Hall;Rhubell Brown

  • IgA nephropathy, the most common glomerulonephritis worldwide: A neglected disease in the United States?

    Bruce A. Julian;Bruce A. Julian;F.Bryson Waldo;F.Bryson Waldo;Abdalla Rifai;Abdalla Rifai;Jiri Mestecky;Jiri Mestecky

  • Patients with igA nephropathy have increased serum galactose-deficient igA1 levels. Commentary

    A. Roos;C. Van Kooten;Z. Moldoveanu;R. J. Wyatt

Frequent Co-Authors

Jan Novak
Jan Novak University of Alabama at Birmingham
Jiri Mestecky
Jiri Mestecky University of Alabama at Birmingham
Zina Moldoveanu
Zina Moldoveanu University of Alabama at Birmingham
Ali G. Gharavi
Ali G. Gharavi Columbia University
Milan Tomana
Milan Tomana University of Alabama at Birmingham
Yasuhiko Tomino
Yasuhiko Tomino Juntendo University
Agnes B. Fogo
Agnes B. Fogo Vanderbilt University Medical Center
Barry I. Freedman
Barry I. Freedman Wake Forest University
Daniel C. Cattran
Daniel C. Cattran University Health Network

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