His main research concerns Internal medicine, Ischemia, Cardiology, Anesthesia and Myocardial infarction. Cardioprotection is closely connected to Endocrinology in his research, which is encompassed under the umbrella topic of Internal medicine. His studies in Ischemia integrate themes in fields like Hemodynamics, Superoxide dismutase and Pharmacology.
His Cardiology research is multidisciplinary, incorporating elements of Blood pressure and Heart rate. The concepts of his Anesthesia study are interwoven with issues in Occlusion, Coronary occlusion and Iloprost. His Myocardial infarction research incorporates elements of Inflammation, Perfusion, Coronary circulation and Pathology.
Internal medicine, Cardiology, Anesthesia, Ischemia and Myocardial infarction are his primary areas of study. Internal medicine is closely attributed to Endocrinology in his work. His work carried out in the field of Anesthesia brings together such families of science as Blood flow, Pharmacology and Heart rate.
Benedict R. Lucchesi has researched Pharmacology in several fields, including Ex vivo and Heparin. The Reperfusion injury research he does as part of his general Ischemia study is frequently linked to other disciplines of science, such as Superoxide, therefore creating a link between diverse domains of science. His Reperfusion injury research includes themes of Complement system and Complement membrane attack complex.
His primary scientific interests are in Internal medicine, Pharmacology, Anesthesia, Cardiology and Reperfusion injury. His Internal medicine research is multidisciplinary, relying on both Endocrinology and Surgery. Benedict R. Lucchesi studies Hemodynamics which is a part of Anesthesia.
His Cardiology research focuses on subjects like Evans Blue, which are linked to Pathology. His Reperfusion injury research incorporates themes from Complement membrane attack complex, Complement system and Cardioprotection, Myocardial infarction. Many of his research projects under Ischemia are closely connected to Apolipoprotein B with Apolipoprotein B, tying the diverse disciplines of science together.
Benedict R. Lucchesi spends much of his time researching Reperfusion injury, Pharmacology, Ischemia, Anesthesia and Internal medicine. His Reperfusion injury research includes elements of Chemokine, Cell adhesion molecule, Cytolysis and Pathogenesis. The Pharmacology study combines topics in areas such as Cytokine, Complement system, Channel blocker, Platelet and Heart metabolism.
Benedict R. Lucchesi interconnects Ex vivo, Antagonist, Myocardial infarction, Focused ultrasound and Electrocardiography in the investigation of issues within Anesthesia. His Internal medicine research is multidisciplinary, incorporating perspectives in Endocrinology and Cardiology. His Cardiology study integrates concerns from other disciplines, such as Carnivora, Evans Blue, Microcirculation and Fissipedia.
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Reduction of the extent of ischemic myocardial injury by neutrophil depletion in the dog.
J L Romson;B G Hook;S L Kunkel;G D Abrams.
Canine myocardial reperfusion injury. Its reduction by the combined administration of superoxide dismutase and catalase.
S R Jolly;W J Kane;M B Bailie;G D Abrams.
Circulation Research (1984)
Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody (anti-Mo1, anti-CD11b) that inhibits leukocyte adhesion.
P J Simpson;R F Todd rd;J C Fantone;J K Mickelson.
Journal of Clinical Investigation (1988)
Myocardial protection with preconditioning.
George C. Li;Juan A. Vasquez;Kim P. Gallagher;Benedict R. Lucchesi.
Mechanisms of myocardial reperfusion injury.
James L Park;Benedict R Lucchesi.
The Annals of Thoracic Surgery (1999)
Free radicals and myocardial ischemia and reperfusion injury
Paul J. Simpson;Benedict R. Lucchesi.
Journal of Laboratory and Clinical Medicine (1987)
Leukocytes and ischemia-induced myocardial injury.
B R Lucchesi;K M Mullane.
Annual Review of Pharmacology and Toxicology (1986)
Free radicals and myocardial injury: pharmacologic implications.
S W Werns;M J Shea;B R Lucchesi.
Reduction of myocardial infarct size by neutrophil depletion: effect of duration of occlusion.
Stanley R. Jolly;William J. Kane;Bruce G. Hook;Gerald D. Abrams.
American Heart Journal (1986)
The independent effects of oxygen radical scavengers on canine infarct size. Reduction by superoxide dismutase but not catalase.
S W Werns;M J Shea;E M Driscoll;C Cohen.
Circulation Research (1985)
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