His primary areas of investigation include Superoxide dismutase, SOD2, Mitochondrion, Molecular biology and Biochemistry. Endocrinology and Internal medicine are the areas that his Superoxide dismutase study falls under. Ting-Ting Huang works mostly in the field of SOD2, limiting it down to topics relating to Superoxide and, in certain cases, SOD1 and Apoptosis.
As part of one scientific family, he deals mainly with the area of Mitochondrion, narrowing it down to issues related to the Gene, and often Cancer, Incidence and Immunology. His research in Molecular biology intersects with topics in Glutathione peroxidase, Glutathione, Paraquat and Cytosol. His work on Oxidative stress, Antioxidant and Reactive oxygen species as part of general Biochemistry research is frequently linked to Subgranular zone, bridging the gap between disciplines.
His primary areas of study are Superoxide dismutase, Oxidative stress, Molecular biology, Internal medicine and Endocrinology. Ting-Ting Huang mostly deals with SOD2 in his studies of Superoxide dismutase. His research integrates issues of Reactive oxygen species, Genetically modified mouse and Mitochondrion in his study of Oxidative stress.
His studies deal with areas such as Glutathione peroxidase, Cell, Gene, Transgene and Cancer cell as well as Molecular biology. His studies examine the connections between Internal medicine and genetics, as well as such issues in Genetics, with regards to Morris water navigation task. His work on Adiponectin receptor 1, Adiponectin and Obesity as part of general Endocrinology study is frequently linked to Weight gain, bridging the gap between disciplines.
Ting-Ting Huang mainly investigates Hippocampal formation, Neurogenesis, Neuroscience, Hippocampus and Central nervous system. His Neurogenesis research is multidisciplinary, incorporating elements of Dentate gyrus and Neurotrophic factors, Tropomyosin receptor kinase B. His work deals with themes such as Genetically modified mouse, Neuroplasticity, Superoxide dismutase and Pharmacology, which intersect with Dentate gyrus.
His study looks at the relationship between Central nervous system and fields such as Cell biology, as well as how they intersect with chemical problems. Internal medicine and Endocrinology are all intrinsically tied to his study in Oxidative stress. The various areas that Ting-Ting Huang examines in his Internal medicine study include Mitochondrion and In vivo.
The scientist’s investigation covers issues in Neuroscience, Complex regional pain syndrome, Hippocampus, Sensitization and Chronic pain. His study on Neurogenesis, Prevention of dementia and Cognitive impairment is often connected to Mechanism as part of broader study in Neuroscience. His Complex regional pain syndrome research is multidisciplinary, incorporating perspectives in Ketamine, Neuroplasticity, Central nervous system and Follow up studies.
He interconnects Synaptic plasticity, Oxidative stress, Cognition, Hippocampal formation and Cognitive decline in the investigation of issues within Hippocampus. His Sensitization study incorporates themes from Glutamate receptor, Central sensitization, Nociception and Spinal cord. The study incorporates disciplines such as Working memory, Physical medicine and rehabilitation, Bioinformatics and Amygdala in addition to Chronic pain.
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Dilated Cardiomyopathy and Neonatal Lethality in Mutant Mice Lacking Manganese Superoxide Dismutase
Yibing Li;Ting-Ting Huang;Elaine J. Carlson;Simon Melov.
Nature Genetics (1995)
Life-long reduction in MnSOD activity results in increased DNA damage and higher incidence of cancer but does not accelerate aging
Holly Van Remmen;Holly Van Remmen;Yuji Ikeno;Yuji Ikeno;Michelle Hamilton;Mohammad Pahlavani.
Physiological Genomics (2003)
CuZnSOD deficiency leads to persistent and widespread oxidative damage and hepatocarcinogenesis later in life
Sailaja Elchuri;Terry D. Oberley;Wenbo Qi;Richard S. Eisenstein.
Mitochondrial disease in superoxide dismutase 2 mutant mice
Simon Melov;Pinar Coskun;Manisha Patel;Robbyn Tuinstra.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Reduction of CuZn-Superoxide Dismutase Activity Exacerbates Neuronal Cell Injury and Edema Formation after Transient Focal Cerebral Ischemia
Takeo Kondo;Andrew G. Reaume;Ting Ting Huang;Elaine Carlson.
The Journal of Neuroscience (1997)
Increased Oxidative Damage Is Correlated to Altered Mitochondrial Function in Heterozygous Manganese Superoxide Dismutase Knockout Mice
Melissa D. Williams;Holly Van Remmen;Holly Van Remmen;Craig C. Conrad;Craig C. Conrad;Ting Ting Huang.
Journal of Biological Chemistry (1998)
Ts1Cje, a partial trisomy 16 mouse model for Down syndrome, exhibits learning and behavioral abnormalities
Haruhiko Sago;Elaine J. Carlson;Desmond J. Smith;Joshua Kilbridge.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Absence of CuZn superoxide dismutase leads to elevated oxidative stress and acceleration of age-dependent skeletal muscle atrophy
Florian L. Muller;Wook Song;Yuhong Liu;Asish Chaudhuri;Asish Chaudhuri.
Free Radical Biology and Medicine (2006)
A Biologically Effective Fullerene (C60) Derivative With Superoxide Dismutase Mimetic Properties
Sameh S. Ali;Joshua I. Hardt;Kevin L. Quick;Jeong Sook Kim-Han.
Free Radical Biology and Medicine (2004)
Knockout mice heterozygous for Sod2 show alterations in cardiac mitochondrial function and apoptosis
Holly Van Remmen;Holly Van Remmen;Melissa D. Williams;Zhongmao Guo;Larry Estlack.
American Journal of Physiology-heart and Circulatory Physiology (2001)
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