His primary areas of investigation include Internal medicine, Cell biology, Biochemistry, Reactive oxygen species and Endocrinology. His biological study deals with issues like Cardiology, which deal with fields such as Randomized controlled trial. His work in the fields of Cell biology, such as Receptor tyrosine kinase, Proto-oncogene tyrosine-protein kinase Src and Tyrosine kinase, intersects with other areas such as CTGF.
His Biochemistry research focuses on Laminin in particular. His studies in Reactive oxygen species integrate themes in fields like AMP-activated protein kinase, Nitric oxide, Mitochondrion and Protein kinase A. His work in Mitochondrion addresses subjects such as Ischemia, which are connected to disciplines such as Electron Transport Complex I, Heart metabolism and Respiratory chain.
Thomas Krieg spends much of his time researching Internal medicine, Mitochondrion, Pharmacology, Endocrinology and Cell biology. His research on Internal medicine frequently links to adjacent areas such as Cardiology. The study incorporates disciplines such as Reactive oxygen species and Bioinformatics in addition to Mitochondrion.
The concepts of his Pharmacology study are interwoven with issues in Reperfusion injury, Signal transduction, Kinase and Bradykinin. His studies deal with areas such as Cardiomyopathy and Diabetic cardiomyopathy as well as Endocrinology. His Cell biology research is multidisciplinary, incorporating perspectives in Fibroblast and In vitro.
Thomas Krieg focuses on Mitochondrion, Pharmacology, Internal medicine, Reactive oxygen species and Ischemia. His Mitochondrion study necessitates a more in-depth grasp of Biochemistry. His Biochemistry study incorporates themes from Tandem mass spectrometry and Mass spectrometry.
His Internal medicine research is multidisciplinary, relying on both Endocrinology and Cardiology. His study with Reactive oxygen species involves better knowledge in Cell biology. His Reperfusion injury study, which is part of a larger body of work in Ischemia, is frequently linked to Monocarboxylate transporter 1, bridging the gap between disciplines.
Mitochondrion, Pharmacology, Ischemia, Reperfusion injury and Reactive oxygen species are his primary areas of study. Biochemistry covers Thomas Krieg research in Mitochondrion. His research investigates the connection between Pharmacology and topics such as Cardioprotection that intersect with issues in Nitric oxide, Ischemic preconditioning, Kinase and Ligation.
In his study, Malonate and Hypothermia is strongly linked to Myocardial infarction, which falls under the umbrella field of Ischemia. His work on Mitochondrial ROS as part of general Reactive oxygen species study is frequently linked to Hormesis, bridging the gap between disciplines. His Cardiology research incorporates elements of Internal medicine, Venous blood and Cardiac magnetic resonance imaging.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.
Edward T. Chouchani;Edward T. Chouchani;Victoria R. Pell;Edoardo Gaude;Dunja Aksentijević.
Nature (2014)
Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways
Xi-Ming Yang;J. Bradley Proctor;Lin Cui;Thomas Krieg.
Journal of the American College of Cardiology (2004)
Cardioprotection by S-nitrosation of a cysteine switch on mitochondrial complex I
Edward T Chouchani;Carmen Methner;Sergiy M Nadtochiy;Angela Logan.
Nature Medicine (2013)
Recombinant nidogen consists of three globular domains and mediates binding of laminin to collagen type IV.
J W Fox;U Mayer;R Nischt;M Aumailley.
The EMBO Journal (1991)
A Unifying Mechanism for Mitochondrial Superoxide Production during Ischemia-Reperfusion Injury.
Edward T. Chouchani;Victoria R. Pell;Andrew M. James;Lorraine M. Work.
Cell Metabolism (2016)
Bradykinin induces mitochondrial ROS generation via NO, cGMP, PKG, and mitoKATP channel opening and leads to cardioprotection.
Olaf Oldenburg;Qining Qin;Thomas Krieg;Xi-Ming Yang.
American Journal of Physiology-heart and Circulatory Physiology (2004)
Inhibition of skin development by overexpression of transforming growth factor beta 1 in the epidermis of transgenic mice
Klaus Sellheyer;Jackie R. Bickenbach;Joseph A. Rothnagel;Donnie Bundman.
Proceedings of the National Academy of Sciences of the United States of America (1993)
Transcriptional control of high molecular weight keratin gene expression in multistage mouse skin carcinogenesis.
Dennis R. Roop;Thomas M. Krieg;Thomas Mehrel;Christina K. Cheng.
Cancer Research (1988)
European contribution to the study of ROS : a summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS)
Javier Egea;Isabel Fabregat;Yves M Frapart;Pietro Ghezzi.
Redox biology (2017)
EXTRACELLULAR-MATRIX COMPONENTS SYNTHESIZED BY HUMAN AMNIOTIC EPITHELIAL-CELLS IN CULTURE
Kari Alitalo;Markku Kurkinen;Antti Vaheri;Thomas Krieg.
Cell (1980)
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