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Fabio Di Lisa

Fabio Di Lisa

D-Index & Metrics

Biology and Biochemistry

D-Index
74
Citations
20056
World Ranking
5588
National Ranking
122

Research.com Recognitions

  • 2017 - Member of Academia Europaea

Overview

Fabio Di Lisa is affiliated with the University of Padua in Italy and has contributed extensively to the fields of Biochemistry, Genetics and Molecular Biology, and Medicine. Their research spans multiple subfields, including Molecular Biology, Cardiology and Cardiovascular Medicine, Physiology, Pathology and Forensic Medicine, and Surgery.

The scientist's main topics of investigation focus on Mitochondrial Function and Pathology, Cardiac Ischemia and Reperfusion, Adipose Tissue and Metabolism, Nitric Oxide and Endothelin Effects, Cardiac Electrophysiology and Arrhythmias, CRISPR and Genetic Engineering, as well as Tissue Engineering and Regenerative Medicine.

Fabio Di Lisa has published prolifically in various scientific journals. Among the frequent venues are:

  • Journal of Molecular and Cellular Cardiology (5 publications)
  • Redox Biology (3 publications)
  • Basic Research in Cardiology (3 publications)
  • Pharmacological Reviews (1 publication)
  • Science Translational Medicine (1 publication)

Recent publications reflect a focus on cardioprotection and mitochondrial pathology, including:

  • On the Clinical Pharmacology of Reactive Oxygen Species, 2020, Pharmacological Reviews
  • Health position paper and redox perspectives on reactive oxygen species as signals and targets of cardioprotection, 2023, Redox Biology
  • Exercise triggers CAPN1-mediated AIF truncation, inducing myocyte cell death in arrhythmogenic cardiomyopathy, 2021, Science Translational Medicine
  • The Determining Role of Mitochondrial Reactive Oxygen Species Generation and Monoamine Oxidase Activity in Doxorubicin-Induced Cardiotoxicity, 2020, Antioxidants and Redox Signaling
  • Biocompatibility studies of macroscopic fibers made from carbon nanotubes: Implications for carbon nanotube macrostructures in biomedical applications, 2020, Carbon

The scientist has collaborated frequently with several researchers, including:

  • Nina Kaludercic (10 joint publications)
  • Salvatore Antonucci (7 joint publications)
  • Moises Di Sante (6 joint publications)
  • Roberta Menabò (6 joint publications)
  • Petra Alánová (4 joint publications)

Fabio Di Lisa has been recognized professionally as a Member of the Academia Europaea since 2017.

Best Publications

  • Mitochondria and cell death. Mechanistic aspects and methodological issues.

    Paolo Bernardi;Luca Scorrano;Raffaele Colonna;Valeria Petronilli

  • Opening of the Mitochondrial Permeability Transition Pore Causes Depletion of Mitochondrial and Cytosolic NAD+and Is a Causative Event in the Death of Myocytes in Postischemic Reperfusion of the Heart

    Fabio Di Lisa;Roberta Menabò;Marcella Canton;Maria Barile

  • The mitochondrial permeability transition from in vitro artifact to disease target

    Paolo Bernardi;Alexandra Krauskopf;Emy Basso;Valeria Petronilli

  • Transient and Long-Lasting Openings of the Mitochondrial Permeability Transition Pore Can Be Monitored Directly in Intact Cells by Changes in Mitochondrial Calcein Fluorescence

    Valeria Petronilli;Giovanni Miotto;Marcella Canton;Marisa Brini

  • Postconditioning and protection from reperfusion injury: where do we stand? Position paper from the Working Group of Cellular Biology of the Heart of the European Society of Cardiology

    Michel Ovize;Gary F. Baxter;Fabio Di Lisa;Péter Ferdinandy

  • The Mitochondrial Permeability Transition, Release of Cytochrome c and Cell Death: CORRELATION WITH THE DURATION OF PORE OPENINGS IN SITU

    Valeria Petronilli;Daniele Penzo;Luca Scorrano;Paolo Bernardi

  • The mitochondrial permeability transition pore: Molecular nature and role as a target in cardioprotection

    Paolo Bernardi;Fabio Di Lisa

  • A mitochondrial perspective on cell death

    Paolo Bernardi;Valeria Petronilli;Fabio Di Lisa;Michael Forte

  • Mitochondria and ischemia–reperfusion injury of the heart: Fixing a hole

    Fabio Di Lisa;Paolo Bernardi

  • The Opa1-Dependent Mitochondrial Cristae Remodeling Pathway Controls Atrophic, Apoptotic, and Ischemic Tissue Damage

    Tatiana Varanita;Maria Eugenia Soriano;Vanina Romanello;Tania Zaglia

  • Practical guidelines for rigor and reproducibility in preclinical and clinical studies on cardioprotection.

    Hans Erik Bøtker;Derek Hausenloy;Ioanna Andreadou;Salvatore Antonucci

  • Connexin 43 in cardiomyocyte mitochondria and its increase by ischemic preconditioning.

    Kerstin Boengler;Giuliano Dodoni;Antonio Rodriguez-Sinovas;Alberto Cabestrero

  • Ischaemic conditioning and targeting reperfusion injury: a 30 year voyage of discovery.

    Derek J. Hausenloy;Jose A. Barrabes;Hans Erik Bøtker;Sean M. Davidson

  • European contribution to the study of ROS : a summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS)

    Javier Egea;Isabel Fabregat;Yves M Frapart;Pietro Ghezzi

  • Crosstalk of mitochondria with NADPH oxidase via reactive oxygen and nitrogen species signalling and its role for vascular function.

    Andreas Daiber;Fabio Di Lisa;Matthias Oelze;Swenja Kröller-Schön

  • Mitochondrial function as a determinant of recovery or death in cell response to injury

    F Di Lisa;P Bernardi

  • Impairment of Diazoxide-Induced Formation of Reactive Oxygen Species and Loss of Cardioprotection in Connexin 43 Deficient Mice

    Frank R. Heinzel;Yukun Luo;Xiaokui Li;Kerstin Boengler

  • Beat‐to‐beat oscillations of mitochondrial [Ca2+] in cardiac cells

    Valérie Robert;Pamela Gurlini;Valeria Tosello;Takeharu Nagai

  • Monoamine oxidases as sources of oxidants in the heart.

    Nina Kaludercic;Jeanne Mialet-Perez;Jeanne Mialet-Perez;Nazareno Paolocci;Angelo Parini;Angelo Parini

  • Monoamine Oxidase A–Mediated Enhanced Catabolism of Norepinephrine Contributes to Adverse Remodeling and Pump Failure in Hearts With Pressure Overload

    Nina Kaludercic;Eiki Takimoto;Takahiro Nagayama;Ning Feng

Frequent Co-Authors

Paolo Bernardi
Paolo Bernardi University of Padua
Rainer Schulz
Rainer Schulz University of Giessen
Valeria Petronilli
Valeria Petronilli University of Padua
Luca Scorrano
Luca Scorrano University of Padua
Gerd Heusch
Gerd Heusch University of Duisburg-Essen
Michael Forte
Michael Forte Oregon Health & Science University
Péter Ferdinandy
Péter Ferdinandy Semmelweis University
Derek J. Hausenloy
Derek J. Hausenloy National University of Singapore
Andreas Daiber
Andreas Daiber Johannes Gutenberg University of Mainz
Tullio Pozzan
Tullio Pozzan University of Padua

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