2026 Michael Forte: Biology and Biochemistry Researcher – H-Index, Publications & Awards

Discipline name	D-Index	World Ranking	Current World Ranking	National Ranking	Current National Ranking	Publications	Citations
Biology and Biochemistry	68	7768	7165	3539	3181	154	17066

Overview

Michael Forte is affiliated with Oregon Health & Science University in the United States and has contributed extensively to the fields of Biochemistry, Genetics, and Molecular Biology, with additional work in Medicine. Their research spans multiple subfields including Molecular Biology, Physiology, Pharmacology, Neurology, and Infectious Diseases.

Forte's main research topics include:

Mitochondrial Function and Pathology
ATP Synthase and ATPases Research
RNA modifications and cancer
Signaling Pathways in Disease
Muscle Physiology and Disorders
Adipose Tissue and Metabolism
Pharmacology and Obesity Treatment

Recent scholarly output from Forte covers key aspects of mitochondrial biology and pathology among other topics. Notable papers include:

"Defining the molecular mechanisms of the mitochondrial permeability transition through genetic manipulation of F-ATP synthase," 2021, published in Nature Communications
"The Unique Cysteine of F-ATP Synthase OSCP Subunit Participates in Modulation of the Permeability Transition Pore," 2020, published in Cell Reports
"Treatment with a triazole inhibitor of the mitochondrial permeability transition pore fully corrects the pathology of sapje zebrafish lacking dystrophin," 2021, published in Pharmacological Research
"N-Phenylbenzamides as Potent Inhibitors of the Mitochondrial Permeability Transition Pore," 2020, available through UNC Libraries
"Common peroneal neuropathy following significant weight loss induced by tirzepatide therapy," 2025, published in PM&R

Forte frequently collaborates with other researchers. Their common co-authors include:

Paolo Bernardi
Justina Šileikytė
Michela Carraro
Ildikò Szabó
Marco Schiavone

Publication venues where Forte's work appears multiple times include:

UNC Libraries
Nature Communications
Cell Reports
Pharmacological Research
PM&R

Best Publications

Dimers of mitochondrial ATP synthase form the permeability transition pore

Valentina Giorgio;Sophia von Stockum;Manuela Antoniel;Astrid Fabbro

994
Citations
Properties of the Permeability Transition Pore in Mitochondria Devoid of Cyclophilin D

Emy Basso;Emy Basso;Lisa Fante;Jonathan Fowlkes;Valeria Petronilli

973
Citations
The mitochondrial permeability transition from in vitro artifact to disease target

Paolo Bernardi;Alexandra Krauskopf;Emy Basso;Valeria Petronilli

783
Citations
Associative learning disrupted by impaired Gs signaling in Drosophila mushroom bodies

John B. Connolly;Ian J. H. Roberts;J. Douglas Armstrong;Kim Kaiser

585
Citations
The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology

Paolo Bernardi;Andrea Rasola;Andrea Rasola;Andrea Rasola;Michael Forte;Giovanna Lippe;Giovanna Lippe;Giovanna Lippe

582
Citations
A mitochondrial perspective on cell death

Paolo Bernardi;Valeria Petronilli;Fabio Di Lisa;Michael Forte

514
Citations
Heterotrimeric G Proteins Direct Two Modes of Asymmetric Cell Division in the Drosophila Nervous System

Matthias Schaefer;Mark Petronczki;Daniela Dorner;Michael Forte

398
Citations
Hexokinase II Detachment from Mitochondria Triggers Apoptosis through the Permeability Transition Pore Independent of Voltage-Dependent Anion Channels

Federica Chiara;Diego Castellaro;Oriano Marin;Valeria Petronilli

367
Citations
Cyclophilin D Modulates Mitochondrial F0F1-ATP Synthase by Interacting with the Lateral Stalk of the Complex

Valentina Giorgio;Elena Bisetto;Maria Eugenia Soriano;Federica Dabbeni-Sala

356
Citations
Selectivity changes in site-directed mutants of the VDAC ion channel: structural implications.

Elizabeth Blachly-Dyson;Songzhi Peng;Marco Colombini;Michael Forte

325
Citations
Ectopic G-protein expression in dopamine and serotonin neurons blocks cocaine sensitization in Drosophila melanogaster

H. Li;S. Chaney;M. Forte;J. Hirsh

323
Citations
VDAC, a Channel in the Outer Mitochondrial Membrane

Marco Colombini;Elizabeth Blachly-Dyson;Michael Forte

321
Citations
GM1-Ganglioside Accumulation at the Mitochondria-Associated ER Membranes Links ER Stress to Ca2+-Dependent Mitochondrial Apoptosis

Renata Sano;Ida Annunziata;Annette Patterson;Simon Moshiach

311
Citations
Properties of the permeability transition in VDAC1 / mitochondria

Alexandra Krauskopf;Ove Eriksson;William J. Craigen;Michael A. Forte

305
Citations
Cloning and functional expression in yeast of two human isoforms of the outer mitochondrial membrane channel, the voltage-dependent anion channel.

E. Blachly-Dyson;E.B. Zambronicz;W.H. Yu;V. Adams

294
Citations
Cyclophilin D inactivation protects axons in experimental autoimmune encephalomyelitis, an animal model of multiple sclerosis

Michael Forte;Bruce G. Gold;Gail Marracci;Gail Marracci;Priya Chaudhary

251
Citations
NADH regulates the gating of VDAC, the mitochondrial outer membrane channel.

M Zizi;M Forte;E Blachly-Dyson;M Colombini

244
Citations
Biogenesis of Porin of the Outer Mitochondrial Membrane Involves an Import Pathway via Receptors and the General Import Pore of the Tom Complex

Thomas Krimmer;Doron Rapaport;Michael T. Ryan;Chris Meisinger

236
Citations
The role of yeast VDAC genes on the permeability of the mitochondrial outer membrane.

Lee Ac;Xu X;Blachly-Dyson E;Forte M

211
Citations
A primordial dopamine D1-like adenylyl cyclase-linked receptor from Drosophila melanogaster displaying poor affinity for benzazepines.

Kim S. Sugamori;Lidia L. Demchyshyn;Fortunata McConkey;Michael A. Forte

209
Citations