D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 106 Citations 45,883 404 World Ranking 3795 National Ranking 2157

Research.com Recognitions

Awards & Achievements

2019 - King Faisal Prize

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Enzyme

Steven L. Teitelbaum spends much of his time researching Internal medicine, Osteoclast, Endocrinology, Bone resorption and Cell biology. His Internal medicine study frequently intersects with other fields, such as Connective tissue. His studies deal with areas such as Cancer research, Cellular differentiation, RANKL, Immunology and Bone remodeling as well as Osteoclast.

His Endocrinology research is multidisciplinary, relying on both Osteoprotegerin, Osteoid, Osteoblast, Osteocalcin and Parathyroid hormone. The study incorporates disciplines such as Osteopetrosis, Calcium, Biophysics, Biochemistry and Resorption in addition to Bone resorption. His Cell biology research incorporates elements of Integrin and Osteopontin.

His most cited work include:

  • Bone Resorption by Osteoclasts (2960 citations)
  • Genetic regulation of osteoclast development and function (1318 citations)
  • TNF-α induces osteoclastogenesis by direct stimulation of macrophages exposed to permissive levels of RANK ligand (1063 citations)

What are the main themes of his work throughout his whole career to date?

Steven L. Teitelbaum mainly investigates Internal medicine, Endocrinology, Osteoclast, Bone resorption and Cell biology. In Endocrinology, Steven L. Teitelbaum works on issues like Macrophage, which are connected to Bone marrow. His Osteoclast study incorporates themes from RANKL, RANK Ligand, Integrin and Cancer research.

His work deals with themes such as Tumor necrosis factor alpha and Signal transduction, which intersect with RANKL. The concepts of his Bone resorption study are interwoven with issues in Osteopetrosis, Resorption, Pathology, In vitro and Glucocorticoid. The Cell biology study combines topics in areas such as Cell, Cytoskeleton, Cellular differentiation and Immunology.

He most often published in these fields:

  • Internal medicine (43.12%)
  • Endocrinology (40.63%)
  • Osteoclast (33.86%)

What were the highlights of his more recent work (between 2010-2021)?

  • Osteoclast (33.86%)
  • Internal medicine (43.12%)
  • Endocrinology (40.63%)

In recent papers he was focusing on the following fields of study:

Steven L. Teitelbaum mostly deals with Osteoclast, Internal medicine, Endocrinology, Cell biology and Bone resorption. Steven L. Teitelbaum has included themes like RANK Ligand, RANKL, Immunology, Bone Density Conservation Agents and Signal transduction in his Osteoclast study. His studies link Phenotype with Internal medicine.

His study in Endocrinology is interdisciplinary in nature, drawing from both Osteoarthritis and Inflammatory arthritis. The various areas that Steven L. Teitelbaum examines in his Cell biology study include Integrin, Cytoskeleton and Monocyte. His Bone resorption research also works with subjects such as

  • Bone remodeling that intertwine with fields like Serotonin,
  • Macrophage colony-stimulating factor which intersects with area such as Integrin alphaVbeta3.

Between 2010 and 2021, his most popular works were:

  • Autophagy proteins regulate the secretory component of osteoclastic bone resorption (294 citations)
  • Osteoclasts: New Insights (169 citations)
  • Report on Mortality from Gluteal Fat Grafting: Recommendations from the ASERF Task Force (103 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

His main research concerns Osteoclast, Cell biology, Bone resorption, Endocrinology and Internal medicine. His Osteoclast research integrates issues from Cell, Cellular differentiation, RANKL, Immunology and Signal transduction. His studies in RANKL integrate themes in fields like Peroxisome proliferator-activated receptor and Osteoprotegerin.

His Cell biology study also includes fields such as

  • Cytoskeleton, which have a strong connection to Actin,
  • Integrin which is related to area like Syk. His Bone resorption research incorporates themes from Osteopetrosis, Macrophage colony-stimulating factor, Cathepsin, Bone remodeling and Cytoskeleton organization. His research ties Osteoblast and Endocrinology together.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Bone Resorption by Osteoclasts

Steven L. Teitelbaum.
Science (2000)

5249 Citations

Genetic regulation of osteoclast development and function

Steven L. Teitelbaum;F. Patrick Ross.
Nature Reviews Genetics (2003)

2200 Citations

TNF-α induces osteoclastogenesis by direct stimulation of macrophages exposed to permissive levels of RANK ligand

Jonathan Lam;Sunao Takeshita;Jane E. Barker;Osami Kanagawa.
Journal of Clinical Investigation (2000)

1472 Citations

Osteoclastic bone resorption by a polarized vacuolar proton pump

Harry C. Blair;Steven L. Teitelbaum;Steven L. Teitelbaum;Robert Ghiselli;Stephen Gluck.
Science (1989)

1130 Citations

β3-integrin–deficient mice are a model for Glanzmann thrombasthenia showing placental defects and reduced survival

Kairbaan M. Hodivala-Dilke;Kevin P. McHugh;Dimitrios A. Tsakiris;Helen Rayburn.
Journal of Clinical Investigation (1999)

1061 Citations

IL-1 mediates TNF-induced osteoclastogenesis

Shi Wei;Hideki Kitaura;Ping Zhou;F. Patrick Ross.
Journal of Clinical Investigation (2005)

888 Citations

Osteoclasts: what do they do and how do they do it?

Steven L. Teitelbaum.
American Journal of Pathology (2007)

789 Citations

Mice lacking β3 integrins are osteosclerotic because of dysfunctional osteoclasts

Kevin P. McHugh;Kairbaan Hodivala-Dilke;Ming-Hao Zheng;Noriyuki Namba.
Journal of Clinical Investigation (2000)

789 Citations

Osteopetrosis in mice lacking haematopoietic transcription factor PU.1

M. M. Tondravi;S. R. McKercher;K. Anderson;J. M. Erdmann.
Nature (1997)

719 Citations

Interactions between the bone matrix proteins osteopontin and bone sialoprotein and the osteoclast integrin alpha v beta 3 potentiate bone resorption.

F. P. Ross;J. Chappel;J. I. Alvarez;D. Sander.
Journal of Biological Chemistry (1993)

686 Citations

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