His primary areas of study are Apoptosis, Cell biology, Receptor, Gene expression and Cancer research. The concepts of his Apoptosis study are interwoven with issues in Tumor necrosis factor alpha, Cell, Cell culture, Carcinogenesis and Cancer cell. The study incorporates disciplines such as Cell cycle and Downregulation and upregulation in addition to Cell biology.
The Receptor study combines topics in areas such as Endocrinology, Death domain and Cell growth. His Gene expression research includes themes of Regulation of gene expression and Initiation factor. His Cancer research research incorporates themes from Tumor suppressor gene and Transfection.
His scientific interests lie mostly in Cell biology, Cancer research, Apoptosis, Downregulation and upregulation and Molecular biology. M. Saeed Sheikh has included themes like Carcinogenesis and Suppressor in his Cell biology study. His research integrates issues of Cell culture, Retinoic acid, Cancer cell, Estrogen receptor and Regulation of gene expression in his study of Cancer research.
His Apoptosis study combines topics in areas such as Tumor necrosis factor alpha and Receptor. The Downregulation and upregulation study which covers Immunology that intersects with Cytotoxic T cell. His studies examine the connections between Molecular biology and genetics, as well as such issues in Gene, with regards to DNA damage, DNA and Death Receptors.
M. Saeed Sheikh spends much of his time researching Cancer research, Cell biology, Cancer cell, Mitochondrion and Lung cancer. His biological study spans a wide range of topics, including Pertuzumab, Suppressor, Breast cancer and Monoclonal antibody. His research on Cell biology frequently links to adjacent areas such as Protein subunit.
M. Saeed Sheikh interconnects Cell culture and Programmed cell death in the investigation of issues within Cancer cell. His studies in Mitochondrion integrate themes in fields like Carcinogenesis and Oxidative stress. His study focuses on the intersection of Ligand and fields such as Pharmacology with connections in the field of Apoptosis.
M. Saeed Sheikh mainly investigates Cell biology, Cancer research, Melanoma, Cobimetinib and Cutaneous melanoma. His is doing research in Mitochondrion and Translocase of the outer membrane, both of which are found in Cell biology. M. Saeed Sheikh combines subjects such as Mitosis, Aspartic acid, Phosphorylation, Kinase and Gene isoform with his study of Cancer research.
His study in Melanoma is interdisciplinary in nature, drawing from both Molecular pathogenesis, Malignancy, Bioinformatics, Dabrafenib and Trametinib.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
p53-dependent and -independent Regulation of the Death Receptor KILLER/DR5 Gene Expression in Response to Genotoxic Stress and Tumor Necrosis Factor α
M. Saeed Sheikh;Timothy F. Burns;Ying Huang;Gen Sheng Wu.
Cancer Research (1998)
Mechanisms of regulation of WAF1/Cip1 gene expression in human breast carcinoma: role of p53-dependent and independent signal transduction pathways.
M. Saeed Sheikh;Xiao Su Li;Jian Chyi Chen;Zhi Ming Shao.
Oncogene (1994)
Role of Gadd45 in apoptosis.
M.Saeed Sheikh;M.Christine Hollander;Albert J. Fornace.
Biochemical Pharmacology (2000)
Role of p53 family members in apoptosis.
M. Saeed Sheikh;M. Saeed Sheikh;Albert J. Fornace.
Journal of Cellular Physiology (2000)
Insulin-like growth factor-binding protein enhancement of insulin-like growth factor-I (IGF-I)-mediated DNA synthesis and IGF-I binding in a human breast carcinoma cell line
Jian‐Chyi ‐C Chen;Zhi‐Ming ‐M Shao;M. Saeed Sheikh;Arif Hussain.
Journal of Cellular Physiology (1994)
The Involvement of Aryl Hydrocarbon Receptor in the Activation of Transforming Growth Factor-β and Apoptosis
Hani Zaher;Pedro M. Fernandez-Salguero;John Letterio;M. Saeed Sheikh.
Molecular Pharmacology (1998)
Altered cell cycle control at the G(2)/M phases in aryl hydrocarbon receptor-null embryo fibroblast.
Guillermo Elizondo;Pedro Fernandez-Salguero;M. Saeed Sheikh;Geum-Yi Kim.
Molecular Pharmacology (2000)
Uniform nomenclature for the mitochondrial contact site and cristae organizing system
Nikolaus Pfanner;Martin van der Laan;Paolo Amati;Roderick A. Capaldi.
Journal of Cell Biology (2014)
The antiapoptotic decoy receptor TRID/TRAIL-R3 is a p53-regulated DNA damage-inducible gene that is overexpressed in primary tumors of the gastrointestinal tract
M Saeed Sheikh;Ying Huang;Ester A Fernandez-Salas;Wafik S El-Deiry.
Oncogene (1999)
Ultraviolet-irradiation-induced apoptosis is mediated via ligand independent activation of tumor necrosis factor receptor 1.
M Saeed Sheikh;Michael J Antinore;Ying Huang;Albert J Fornace.
Oncogene (1998)
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