2012 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary areas of study are Transcription factor, Molecular biology, Cell biology, Biochemistry and Gene. Alvaro Puga studies Aryl hydrocarbon receptor which is a part of Transcription factor. His Molecular biology research incorporates themes from Aromatic hydrocarbon receptor, CYP1A2, Psychological repression, Mutagen and MAP kinase kinase kinase.
His study in Cell biology focuses on Signal transduction in particular. The various areas that Alvaro Puga examines in his Gene study include Drug metabolizing enzymes and Enzyme. His work deals with themes such as Cell cycle, Regulation of gene expression and Receptor, which intersect with Aryl hydrocarbon receptor nuclear translocator.
His primary areas of investigation include Molecular biology, Aryl hydrocarbon receptor, Cell biology, Transcription factor and Gene. Alvaro Puga has researched Molecular biology in several fields, including Cell culture, Reporter gene, Gene expression, Chromatin immunoprecipitation and Messenger RNA. His Aryl hydrocarbon receptor research includes elements of Endocrinology, Internal medicine and Cancer research.
His research investigates the connection with Cell biology and areas like Regulation of gene expression which intersect with concerns in DNA methylation, Epigenetics and Transcription. His work focuses on many connections between Transcription factor and other disciplines, such as Cell cycle, that overlap with his field of interest in Cell growth and Receptor. His Gene study contributes to a more complete understanding of Genetics.
Alvaro Puga mainly investigates Aryl hydrocarbon receptor, Cell biology, Internal medicine, Endocrinology and Signal transduction. His Aryl hydrocarbon receptor study is concerned with Transcription factor in general. His studies deal with areas such as Cancer research, Environmental exposure and Euchromatin as well as Transcription factor.
His Cell biology study combines topics in areas such as Chromatin, Embryonic stem cell, Epigenetics and Cellular differentiation. His Signal transduction research includes themes of AH Receptor and Regulation of gene expression. His Molecular biology research is multidisciplinary, incorporating elements of Histone H3, Gene expression and Gene expression profiling.
His primary scientific interests are in Aryl hydrocarbon receptor, Transcription factor, Cell biology, Molecular biology and Endocrinology. The concepts of his Aryl hydrocarbon receptor study are interwoven with issues in Gene expression, Homeostasis, HDAC10, Histone and Histone methylation. The study incorporates disciplines such as Embryonic stem cell and Mediator in addition to Transcription factor.
His biological study spans a wide range of topics, including Morphogenesis and Transactivation. Alvaro Puga focuses mostly in the field of Molecular biology, narrowing it down to topics relating to DNA methylation and, in certain cases, Tumor suppressor proteins and Insulin-like growth factor-II. Alvaro Puga interconnects Internal medicine, Signal transduction, Growth factor, Zebrafish and Cell cycle in the investigation of issues within Endocrinology.
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REGULATION OF GENE EXPRESSION BY REACTIVE OXYGEN
Timothy P. Dalton;Howard G. Shertzer;Alvaro Puga.
Annual Review of Pharmacology and Toxicology (1999)
Role of the Ah receptor and the dioxin-inducible [Ah] gene battery in toxicity, cancer, and signal transduction.
Daniel W. Nebert;Alvaro Puga;Vasilis Vasiliou.
Annals of the New York Academy of Sciences (1993)
The aryl hydrocarbon receptor cross-talks with multiple signal transduction pathways.
Alvaro Puga;Ci Ma;Jennifer L. Marlowe.
Biochemical Pharmacology (2009)
Human Drug-Metabolizing Enzyme Polymorphisms: Effects on Risk of Toxicity and Cancer
Daniel W. Nebert;Ross A. Mckinnon;Alvaro Puga.
DNA and Cell Biology (1996)
Aryl hydrocarbon receptor, cell cycle regulation, toxicity, and tumorigenesis
Jennifer L. Marlowe;Alvaro Puga.
Journal of Cellular Biochemistry (2005)
Aromatic Hydrocarbon Receptor Interaction with the Retinoblastoma Protein Potentiates Repression of E2F-dependent Transcription and Cell Cycle Arrest
Alvaro Puga;Sonya J. Barnes;Timothy P. Dalton;Ching Yi Chang.
Journal of Biological Chemistry (2000)
Heme oxygenase-1 induction by NRF2 requires inactivation of the transcriptional repressor BACH1
John F. Reichard;Gregory T. Motz;Alvaro Puga.
Nucleic Acids Research (2007)
Long term low-dose arsenic exposure induces loss of DNA methylation.
John F. Reichard;Michael Schnekenburger;Alvaro Puga.
Biochemical and Biophysical Research Communications (2007)
Constitutive Activation of the Aromatic Hydrocarbon Receptor
Ching-Yi Chang;Alvaro Puga.
Molecular and Cellular Biology (1998)
Effects of arsenic exposure on DNA methylation and epigenetic gene regulation
John F Reichard;Alvaro Puga.
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