David H. Sherr spends much of his time researching Aryl hydrocarbon receptor, Transcription factor, Cancer research, Receptor and Cell biology. His work deals with themes such as Carcinogenesis, Inflammation, Signal transduction and Immune system, which intersect with Aryl hydrocarbon receptor. David H. Sherr has researched Transcription factor in several fields, including Molecular biology, Apoptosis and Immunology.
The Cancer research study combines topics in areas such as Cell culture, Regulation of gene expression and NF-κB. His research integrates issues of Gut flora, Fatty acid synthase, Pharmacology and Fatty liver in his study of Receptor. His research in Cell biology intersects with topics in T cell and Cell growth.
His scientific interests lie mostly in Aryl hydrocarbon receptor, Cell biology, Molecular biology, Immunology and B cell. His studies deal with areas such as Receptor, Cancer research, Signal transduction and Cell growth as well as Aryl hydrocarbon receptor. David H. Sherr interconnects Apoptosis, Cell culture, Induced pluripotent stem cell and Endogeny in the investigation of issues within Cell biology.
The concepts of his Apoptosis study are interwoven with issues in Endocrinology and Internal medicine. His study in Molecular biology is interdisciplinary in nature, drawing from both T cell, Antibody, Hapten, Antigen and Naive B cell. David H. Sherr combines subjects such as Regulation of gene expression and NF-κB with his study of Transcription factor.
The scientist’s investigation covers issues in Aryl hydrocarbon receptor, Cancer research, Neuroscience, Cancer and Metastasis. His work deals with themes such as Receptor, Internal medicine, Kidney disease, Uremia and Immunotherapy, which intersect with Aryl hydrocarbon receptor. His Receptor research includes elements of Translational science and Pharmacology.
His Cancer research research is multidisciplinary, incorporating elements of Cell, Gene knockdown, Carcinogen, Cancer stem cell and Transcription factor. Transcription factor is a subfield of Biochemistry that David H. Sherr studies. His Induced pluripotent stem cell study combines topics in areas such as Regulation of gene expression and Cell biology.
His primary scientific interests are in Aryl hydrocarbon receptor, Cancer research, Neuroscience, Receptor and Political science. His work carried out in the field of Aryl hydrocarbon receptor brings together such families of science as CCL2, Gut flora, Small hairpin RNA, Viral replication and CYP1B1. His research in Gut flora focuses on subjects like Inflammation, which are connected to Pharmacology.
His studies deal with areas such as Cancer, Metastasis and Cell, Matrigel as well as Small hairpin RNA. He interconnects Cell growth, WNT5A, Cancer stem cell, CD24 and Catenin in the investigation of issues within CYP1B1. He has included themes like Endocrinology, Kidney disease and Antithrombotic in his Cancer research study.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation
Shyr-Te Ju;D. J. Panka;Haili Cui;R. Ettinger.
The aryl hydrocarbon receptor interacts with c-Maf to promote the differentiation of type 1 regulatory T cells induced by IL-27
Lionel Apetoh;Francisco J Quintana;Caroline Pot;Nicole Joller.
Nature Immunology (2010)
Inhibition of NF-kappaB/Rel induces apoptosis of murine B cells.
M Wu;H Lee;R E Bellas;S L Schauer.
The EMBO Journal (1996)
Aromatic hydrocarbon receptor-driven Bax gene expression is required for premature ovarian failure caused by biohazardous environmental chemicals.
Tiina Matikainen;Gloria I. Perez;Andrea Jurisicova;Andrea Jurisicova;James K. Pru.
Nature Genetics (2001)
NF-κB and epithelial to mesenchymal transition of cancer†
Chengyin Min;Sean F. Eddy;Sean F. Eddy;David H. Sherr;David H. Sherr;Gail E. Sonenshein;Gail E. Sonenshein.
Journal of Cellular Biochemistry (2008)
The RelA NF-κB subunit and the aryl hydrocarbon receptor (AhR) cooperate to transactivate the c- myc promoter in mammary cells
Dong W. Kim;Lee Gazourian;Shafat A. Quadri;Raphaëlle Romieu-Mourez.
Green tea extracts decrease carcinogen‐induced mammary tumor burden in rats and rate of breast cancer cell proliferation in culture
Kathryn T. Kavanagh;Laurie J. Hafer;Dong W. Kim;Koren K. Mann.
Journal of Cellular Biochemistry (2001)
Gut Microbiota-Derived Tryptophan Metabolites Modulate Inflammatory Response in Hepatocytes and Macrophages.
Smitha Krishnan;Yufang Ding;Nima Saedi;Maria Choi.
Cell Reports (2018)
Aryl Hydrocarbon Receptor Control of Adaptive Immunity
Francisco J. Quintana;David H. Sherr.
Pharmacological Reviews (2013)
Control of tumor-associated macrophages and T cells in glioblastoma via AHR and CD39
Maisa C. Takenaka;Galina Gabriely;Veit Rothhammer;Ivan D. Mascanfroni.
Nature Neuroscience (2019)
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