What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Enzyme
Keith A. Hruska spends much of his time researching Internal medicine, Endocrinology, Kidney disease, Cell biology and Parathyroid hormone.
His research integrates issues of Signal transduction and Bone morphogenetic protein in his study of Internal medicine.
Keith A. Hruska interconnects Calcification and Osteoblast in the investigation of issues within Endocrinology.
His study looks at the intersection of Kidney disease and topics like Pathology with Vascular disease.
His Cell biology research includes themes of Osteoclast, microRNA and Cancer research.
His Parathyroid hormone study integrates concerns from other disciplines, such as Calcium in biology, Hyperparathyroidism, Diacylglycerol kinase and Metabolism.
His most cited work include:
- Osteoporosis prevention, diagnosis, and therapy (3598 citations)
- K/DOQI clinical practice guidelines for bone metabolism and disease in chronic kidney disease (2107 citations)
- Protein transduction: unrestricted delivery into all cells? (550 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Internal medicine, Endocrinology, Parathyroid hormone, Kidney disease and Kidney.
His is doing research in Renal osteodystrophy, Bone remodeling, Renal function, Hyperphosphatemia and Hyperparathyroidism, both of which are found in Internal medicine.
His Endocrinology research is multidisciplinary, relying on both Inositol, Calcium and Osteoblast.
His work carried out in the field of Parathyroid hormone brings together such families of science as Brush border, Protein kinase A, Hormone, Radioimmunoassay and In vivo.
While the research belongs to areas of Kidney disease, Keith A. Hruska spends his time largely on the problem of Pathology, intersecting his research to questions surrounding Bone morphogenetic protein 7.
His studies deal with areas such as Urinary system and Surgery as well as Kidney.
He most often published in these fields:
- Internal medicine (61.61%)
- Endocrinology (57.74%)
- Parathyroid hormone (23.51%)
What were the highlights of his more recent work (between 2011-2021)?
- Internal medicine (61.61%)
- Endocrinology (57.74%)
- Kidney disease (18.45%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Internal medicine, Endocrinology, Kidney disease, Kidney and Renal osteodystrophy.
His study in Internal medicine is interdisciplinary in nature, drawing from both Gastroenterology, Activin Receptor Type-2A and Diabetes mellitus.
The concepts of his Endocrinology study are interwoven with issues in Sclerostin and Pathogenesis.
His studies in Kidney disease integrate themes in fields like Hyperparathyroidism, Klotho, Osteodystrophy and Pathology.
The Kidney study combines topics in areas such as Secondary hyperparathyroidism, Disease and Vascular disease.
Within one scientific family, Keith A. Hruska focuses on topics pertaining to Bioinformatics under Renal osteodystrophy, and may sometimes address concerns connected to Crosstalk.
Between 2011 and 2021, his most popular works were:
- Early chronic kidney disease-mineral bone disorder stimulates vascular calcification. (135 citations)
- CKD-Induced Wingless/Integration1 Inhibitors and Phosphorus Cause the CKD–Mineral and Bone Disorder (105 citations)
- Down-regulation of miR-21 biogenesis by estrogen action contributes to osteoclastic apoptosis. (69 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Enzyme
Internal medicine, Endocrinology, Kidney disease, Kidney and Klotho are his primary areas of study.
His Internal medicine study combines topics from a wide range of disciplines, such as Fas ligand, Genetic linkage and Type 2 Diabetes Mellitus.
Endocrinology is closely attributed to Sclerostin in his work.
His Kidney disease research incorporates themes from Fibrosis, Osteodystrophy, Pathogenesis and Bone resorption.
His Kidney research incorporates elements of Transforming growth factor, Transforming growth factor beta, Clinical trial and Physiology.
His work in Renal osteodystrophy addresses subjects such as Bone remodeling, which are connected to disciplines such as Bone disease and Arterial calcification.
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