D-Index & Metrics Best Publications
Ryuichi Nishinakamura

Ryuichi Nishinakamura

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 49 Citations 8,707 122 World Ranking 10874 National Ranking 774

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Genetics
  • Cellular differentiation

His primary areas of study are Cell biology, Immunology, Embryonic stem cell, Progenitor cell and Endocrinology. His biological study spans a wide range of topics, including Cellular differentiation, Ureteric bud and Induced pluripotent stem cell. The Immunology study combines topics in areas such as Receptor, Gene targeting and Kidney.

His Embryonic stem cell research is multidisciplinary, relying on both Wnt signaling pathway, WNT3A, Wnt3A Protein, Progenitor and Molecular biology. His Endocrinology research includes elements of SALL1, Internal medicine and Homeotic gene. His work deals with themes such as SALL4, Townes–Brocks syndrome and Haploinsufficiency, which intersect with Homeotic gene.

His most cited work include:

  • Redefining the In Vivo Origin of Metanephric Nephron Progenitors Enables Generation of Complex Kidney Structures from Pluripotent Stem Cells (456 citations)
  • Murine homolog of SALL1 is essential for ureteric bud invasion in kidney development. (296 citations)
  • Mice deficient for the IL-3/GM-CSF/IL-5 βc receptor exhibit lung pathology and impaired immune response, while βIL3 receptor-deficient mice are normal (273 citations)

What are the main themes of his work throughout his whole career to date?

Ryuichi Nishinakamura mainly focuses on Cell biology, Kidney, Internal medicine, Kidney development and Progenitor cell. His Cell biology research incorporates themes from Embryonic stem cell, Induced pluripotent stem cell and Nephron. Ryuichi Nishinakamura has included themes like Renal stem cell, Cell type, Anatomy and Pathology in his Kidney study.

In his work, Mesonephros is strongly intertwined with Endocrinology, which is a subfield of Internal medicine. His study looks at the relationship between Kidney development and fields such as Mesenchyme, as well as how they intersect with chemical problems. His Progenitor cell study incorporates themes from Immunology and Directed differentiation.

He most often published in these fields:

  • Cell biology (70.81%)
  • Kidney (27.33%)
  • Internal medicine (26.09%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cell biology (70.81%)
  • Induced pluripotent stem cell (18.63%)
  • Kidney (27.33%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Cell biology, Induced pluripotent stem cell, Kidney, Progenitor cell and Nephron. His studies in Cell biology integrate themes in fields like Embryonic stem cell and Cardiac neural crest cells. His study in the fields of Kidney development under the domain of Embryonic stem cell overlaps with other disciplines such as Microglia.

His Kidney development research integrates issues from Nephrology and Internal medicine. His Kidney research also works with subjects such as

  • Transplantation, which have a strong connection to Kidney disease, Renal function, Regenerative medicine and Interstitial space,
  • Organoid that connect with fields like Stem cell and Cell type. Ryuichi Nishinakamura interconnects In vitro, Wnt signaling pathway and Directed differentiation in the investigation of issues within Progenitor cell.

Between 2017 and 2021, his most popular works were:

  • Organoids from Nephrotic Disease-Derived iPSCs Identify Impaired NEPHRIN Localization and Slit Diaphragm Formation in Kidney Podocytes. (47 citations)
  • Manipulation of Nephron-Patterning Signals Enables Selective Induction of Podocytes from Human Pluripotent Stem Cells (31 citations)
  • Human kidney organoids: progress and remaining challenges (28 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Genetics
  • Cellular differentiation

His primary scientific interests are in Induced pluripotent stem cell, Cell biology, Progenitor cell, Wnt signaling pathway and Transplantation. In his study, which falls under the umbrella issue of Induced pluripotent stem cell, Renal function, Multipotent Stem Cell and Kidney transplantation is strongly linked to Regenerative medicine. Cell biology and Phenotype are commonly linked in his work.

He combines subjects such as Cancer research and Nephron with his study of Progenitor cell. Ryuichi Nishinakamura focuses mostly in the field of Wnt signaling pathway, narrowing it down to matters related to Directed differentiation and, in some cases, Kidney development, Bone morphogenetic protein 7 and Fibroblast growth factor. His Transplantation research is multidisciplinary, relying on both Cellular differentiation, Kidney disease, Regeneration, Kidney and Mesenchymal stem cell.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Redefining the In Vivo Origin of Metanephric Nephron Progenitors Enables Generation of Complex Kidney Structures from Pluripotent Stem Cells

Atsuhiro Taguchi;Yusuke Kaku;Tomoko Ohmori;Sazia Sharmin.
Cell Stem Cell (2014)

544 Citations

Murine homolog of SALL1 is essential for ureteric bud invasion in kidney development.

Ryuichi Nishinakamura;Yuko Matsumoto;Kazuki Nakao;Kenji Nakamura.
Development (2001)

454 Citations

Mice deficient for the IL-3/GM-CSF/IL-5 βc receptor exhibit lung pathology and impaired immune response, while βIL3 receptor-deficient mice are normal

Ryuichi Nishinakamura;Naoki Nakayama;Yoko Hirabayashi;Tohru Inoue.
Immunity (1995)

344 Citations

The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development.

Masayo Sakaki-Yumoto;Chiyoko Kobayashi;Akira Sato;Sayoko Fujimura.
Development (2006)

344 Citations

Synergistic action of Wnt and LIF in maintaining pluripotency of mouse ES cells

Kazuya Ogawa;Ryuichi Nishinakamura;Yuko Iwamatsu;Daisuke Shimosato.
Biochemical and Biophysical Research Communications (2006)

299 Citations

Hematopoiesis in Mice Lacking the Entire Granulocyte-Macrophage Colony-Stimulating Factor/Interleukin-3/Interleukin-5 Functions

R Nishinakamura;A Miyajima;PJ Mee;VL Tybulewicz.
Blood (1996)

286 Citations

Sall1 is a transcriptional regulator defining microglia identity and function

Anne Buttgereit;Iva Lelios;Xueyang Yu;Melissa Vrohlings.
Nature Immunology (2016)

284 Citations

Generation of kidney from pluripotent stem cells via blastocyst complementation.

Jo Ichi Usui;Toshihiro Kobayashi;Tomoyuki Yamaguchi;A. S. Knisely.
American Journal of Pathology (2012)

204 Citations

Identification of multipotent progenitors in the embryonic mouse kidney by a novel colony-forming assay.

Kenji Osafune;Minoru Takasato;Minoru Takasato;Andreas Kispert;Makoto Asashima.
Development (2006)

195 Citations

The pulmonary alveolar proteinosis in granulocyte macrophage colony-stimulating factor/interleukins 3/5 beta c receptor-deficient mice is reversed by bone marrow transplantation.

Ryuichi Nishinakamura;Rhonda Wiler;Uta Dirksen;Yoshihiro Morikawa.
Journal of Experimental Medicine (1996)

186 Citations

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