2012 - Fellow of the American Association for the Advancement of Science (AAAS)
1997 - Fellow of John Simon Guggenheim Memorial Foundation
His main research concerns Molecular biology, Genetics, Biochemistry, Chromatin and Acetylation. He combines subjects such as Acetyltransferase, Topoisomerase and Mutant with his study of Molecular biology. His research links Computational biology with Genetics.
His work deals with themes such as Histone and Gene silencing, which intersect with Chromatin. His research investigates the connection between Acetylation and topics such as Saccharomyces cerevisiae that intersect with issues in Acetyltransferase complex, Ogden Syndrome, Chloramphenicol acetyltransferase and Derepression. His Gene study combines topics from a wide range of disciplines, such as DNA and Protein–protein interaction.
Biochemistry, Genetics, Molecular biology, Saccharomyces cerevisiae and Mutant are his primary areas of study. His is involved in several facets of Biochemistry study, as is seen by his studies on Acetylation, Histone, Acetyltransferase, HAT1 and NAD+ kinase. His study in Genetics concentrates on Gene, Gene silencing, Chromatin, Origin recognition complex and DNA replication.
The Molecular biology study which covers Topoisomerase that intersects with DNA supercoil, Eukaryotic DNA replication, Camptothecin and Schizosaccharomyces pombe. The various areas that Rolf Sternglanz examines in his Saccharomyces cerevisiae study include Transcription factor, DNA-binding protein, Transcription and Protein biosynthesis. His study explores the link between Mutant and topics such as Glycoprotein that cross with problems in Glycosylation.
His primary areas of investigation include Saccharomyces cerevisiae, Biochemistry, Genetics, Histone and Cell biology. To a larger extent, Rolf Sternglanz studies Gene with the aim of understanding Saccharomyces cerevisiae. His study in Chromatin, Sirtuin, Binding site and Deacetylase activity falls under the purview of Biochemistry.
All of his Genetics and Gene silencing and Mutation investigations are sub-components of the entire Genetics study. His work in Histone is not limited to one particular discipline; it also encompasses Acetylation. Mutant is closely connected to Molecular biology in his research, which is encompassed under the umbrella topic of Histone octamer.
Rolf Sternglanz focuses on Biochemistry, Histone, Chromatin, SIRT3 and Acetylation. His research brings together the fields of Rational design and Biochemistry. His studies deal with areas such as Protein biosynthesis, Messenger RNA, Saccharomyces cerevisiae and Kinase activity as well as Chromatin.
Rolf Sternglanz is researching SIRT3 as part of the investigation of Sirtuin and Genetics. Rolf Sternglanz interconnects Lysine and Binding site in the investigation of issues within Acetylation. His research integrates issues of Histone methyltransferase, Histone code, Histone H2A, Histone H1 and Gene silencing in his study of Histone H4.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The two-hybrid system: a method to identify and clone genes for proteins that interact with a protein of interest.
Cheng-Ting Chien;Paul L. Bartel;Rolf Sternglanz;Stanley Fields.
Proceedings of the National Academy of Sciences of the United States of America (1991)
The silencing protein SIR2 and its homologs are NAD-dependent protein deacetylases.
Joseph Landry;Ann Sutton;Stefan T. Tafrov;Ryan C. Heller.
Proceedings of the National Academy of Sciences of the United States of America (2000)
The two-hybrid system: an assay for protein-protein interactions
Stanley Fields;Rolf Sternglanz.
Trends in Genetics (1994)
DNA topoisomerase II mutant of Saccharomyces cerevisiae: topoisomerase II is required for segregation of daughter molecules at the termination of DNA replication.
Stephen Dinardo;Karen Voelkel;Rolf Sternglanz.
Proceedings of the National Academy of Sciences of the United States of America (1984)
Elimination of false positives that arise in using the two-hybrid system.
P. Bartel;Cheng-Ting Chien;R. Sternglanz;S. Fields.
BioTechniques (1993)
Silent information regulator 2 family of NAD- dependent histone/protein deacetylases generates a unique product, 1-O-acetyl-ADP-ribose
Kirk G. Tanner;Joseph Landry;Rolf Sternglanz;John M. Denu.
Proceedings of the National Academy of Sciences of the United States of America (2000)
SirT2 is a histone deacetylase with preference for histone H4 Lys 16 during mitosis
Alejandro Vaquero;Michael B. Scher;Dong Hoon Lee;Ann Sutton.
Genes & Development (2006)
Characterization of a “silencer” in yeast: A DNA sequence with properties opposite to those of a transcriptional enhancer
Andrea H. Brand;Linda Breeden;Judith Abraham;Rolf Sternglanz.
Cell (1985)
Perinuclear localization of chromatin facilitates transcriptional silencing
Erik D. Andrulis;Aaron M. Neiman;David C. Zappulla;Rolf Sternglanz.
Nature (1998)
Escherichia coli DNA topoisomerase I mutants have compensatory mutations in DNA gyrase genes
Stephen Dinardo;Karen A. Voelkel;Rolf Sternglanz;Ann E. Reynolds.
Cell (1982)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Pennsylvania
Chinese Academy of Sciences
Stony Brook University
University of Washington
Osaka University
Structural Genomics Consortium
Harvard University
Stowers Institute for Medical Research
New York University
Structural Genomics Consortium
Lancaster University
Sunway University
Michigan State University
Polytechnic University of Milan
University of Notre Dame
Stanford University
Purdue University West Lafayette
Osaka University
Technion – Israel Institute of Technology
Nagoya University
University of Maine
Tohoku University
University of Sassari
New York University
San Diego State University
Goethe University Frankfurt