His primary areas of study are Anesthesia, Internal medicine, Cerebral blood flow, Ischemia and Endocrinology. Within one scientific family, Richard J. Traystman focuses on topics pertaining to Blood pressure under Anesthesia, and may sometimes address concerns connected to Catecholamine, Immature animal and Mean Blood Flow Velocity. The concepts of his Internal medicine study are interwoven with issues in Glutamine synthetase, Glutamine and Cardiology.
The Cerebral blood flow study combines topics in areas such as Hemodynamics, Blood flow, Perfusion and Intracranial pressure. His studies in Ischemia integrate themes in fields like Temporal cortex, CATS, Biochemistry, Neuroprotection and Stroke. Richard J. Traystman has researched Endocrinology in several fields, including Glutamate receptor and Enzyme inhibitor.
His scientific interests lie mostly in Anesthesia, Internal medicine, Cerebral blood flow, Endocrinology and Ischemia. His research integrates issues of Blood flow, Blood pressure and Hypoxia in his study of Anesthesia. His research combines Cardiology and Internal medicine.
As a member of one scientific family, Richard J. Traystman mostly works in the field of Cerebral blood flow, focusing on Fissipedia and, on occasion, Carnivora. Endocrinology and Glutamine are commonly linked in his work. His Ischemia study incorporates themes from Central nervous system disease, CATS, Neuroprotection, Stroke and Infarction.
Richard J. Traystman spends much of his time researching Internal medicine, Anesthesia, Endocrinology, Ischemia and Neuroprotection. His Internal medicine study frequently draws parallels with other fields, such as Cardiology. He regularly ties together related areas like Microcirculation in his Endocrinology studies.
Richard J. Traystman combines subjects such as Stroke, Cerebral infarction, Central nervous system disease, Saline and Infarction with his study of Ischemia. His Neuroprotection study integrates concerns from other disciplines, such as Hippocampal formation, Brain ischemia, Biochemistry and Microdialysis. The various areas that Richard J. Traystman examines in his Cerebral blood flow study include Hemodynamics, Blood pressure and Perfusion.
His primary areas of investigation include Ischemia, Internal medicine, Endocrinology, Anesthesia and Cerebral blood flow. His study in Ischemia is interdisciplinary in nature, drawing from both Stroke, Cerebral infarction, Central nervous system disease, Neuroprotection and Infarction. His Internal medicine research is multidisciplinary, incorporating elements of Glutamine and Cardiology.
His Endocrinology study combines topics in areas such as Glutamate receptor and Enzyme inhibitor. His research in Cerebral blood flow intersects with topics in Mean arterial pressure, Perfusion, Intracranial pressure, Hemodynamics and Intracerebral hemorrhage. Richard J. Traystman works mostly in the field of Cerebral perfusion pressure, limiting it down to topics relating to Ventricular fibrillation and, in certain cases, Blood flow.
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Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemia
Mikael J.L. Eliasson;Kenji Sampei;Allen S. Mandir;Patricia D. Hurn.
Nature Medicine (1997)
Oxygen radical mechanisms of brain injury following ischemia and reperfusion
R. J. Traystman;J. R. Kirsch;R. C. Koehler.
Journal of Applied Physiology (1991)
An α-syntrophin-dependent pool of AQP4 in astroglial end-feet confers bidirectional water flow between blood and brain
Mahmood Amiry-Moghaddam;Takashi Otsuka;Patricia D. Hurn;Richard J. Traystman.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Quantitative assessment of blood flow, blood volume and blood oxygenation effects in functional magnetic resonance imaging
P.C.M van Zijl;S.E Eleff;J.A Ulatowski;J.M.E Oja;J.M.E Oja.
Nature Medicine (1998)
Mechanisms by which epinephrine augments cerebral and myocardial perfusion during cardiopulmonary resuscitation in dogs.
J. R. Michael;A. D. Guerci;R. C. Koehler;A. Y. Shi.
Neuroprotective effects of female gonadal steroids in reproductively senescent female rats.
Nabil J. Alkayed;Stephanie J. Murphy;Richard J. Traystman;Patricia D. Hurn.
Early Neurodegeneration after Hypoxia-Ischemia in Neonatal Rat Is Necrosis while Delayed Neuronal Death Is Apoptosis.
Frances J. Northington;Donna M. Ferriero;Ernest M. Graham;Richard J. Traystman.
Neurobiology of Disease (2001)
17β-Estradiol Reduces Stroke Injury in Estrogen-Deficient Female Animals
Renata Rusa;Nabil J. Alkayed;Barbara J. Crain;Richard J. Traystman.
Inhibition of brain glutamine accumulation prevents cerebral edema in hyperammonemic rats
H. Takahashi;R. C. Koehler;S. W. Brusilow;R. J. Traystman.
American Journal of Physiology-heart and Circulatory Physiology (1991)
Selective inhibition of NAALADase, which converts NAAG to glutamate, reduces ischemic brain injury.
Barbara S. Slusher;James J. Vornov;Ajit G. Thomas;Patricia D. Hurn.
Nature Medicine (1999)
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