What is he best known for?
The fields of study he is best known for:
Paul F. Kenna mainly focuses on Retinitis pigmentosa, Genetics, Gene, Rhodopsin and Mutant.
His Retinitis pigmentosa study incorporates themes from Retinal degeneration, Molecular biology and Cell biology.
His research in Cell biology intersects with topics in Cone cell, Transgene, Opsin and Outer nuclear layer.
His research investigates the link between Rhodopsin and topics such as RNA interference that cross with problems in Cancer research.
His Mutant research includes elements of Peripherin, Base pair and Chromosome.
Paul F. Kenna has included themes like Cell type, Transplantation and Pathology in his Retinal study.
His most cited work include:
- Retinopathy induced in mice by targeted disruption of the rhodopsin gene (470 citations)
- Retinopathy induced in mice by targeted disruption of the rhodopsin gene (470 citations)
- A three-base-pair deletion in the peripherin– RDS gene in one form of retinitis pigmentosa (354 citations)
What are the main themes of his work throughout his whole career to date?
Genetics, Retinitis pigmentosa, Gene, Cell biology and Retinal are his primary areas of study.
Genetics is frequently linked to Rhodopsin in his study.
His Retinitis pigmentosa research includes elements of Retinal degeneration, Mutant, Genetic linkage, Locus and Exon.
His Cell biology study incorporates themes from Retina, Retinal ganglion cell, Knockout mouse and Transgene.
His Retinal research includes themes of Disease, Pathology, Pharmacology and Macular degeneration.
His research integrates issues of Inflammasome and Interleukin 18 in his study of Macular degeneration.
He most often published in these fields:
- Genetics (61.08%)
- Retinitis pigmentosa (48.65%)
- Gene (37.30%)
What were the highlights of his more recent work (between 2017-2021)?
- Gene (37.30%)
- Genetics (61.08%)
- Retinal (24.32%)
In recent papers he was focusing on the following fields of study:
Paul F. Kenna focuses on Gene, Genetics, Retinal, Retinitis pigmentosa and Disease.
His multidisciplinary approach integrates Genetics and Irish in his work.
His Retinal research integrates issues from Atrophy, Pathology, Retina and Tight junction, Claudin.
In his research, Cell biology is intimately related to Programmed cell death, which falls under the overarching field of Retina.
His studies in Retinitis pigmentosa integrate themes in fields like Electroretinography, Compound heterozygosity, Ataxia, Genetic enhancement and splice.
His work is dedicated to discovering how Disease, Genotyping are connected with Exome sequencing, Genetic analysis, Genomics, Retinal Disorder and Computational biology and other disciplines.
Between 2017 and 2021, his most popular works were:
- Dose-dependent expression of claudin-5 is a modifying factor in schizophrenia (54 citations)
- Findings from a Genotyping Study of Over 1000 People with Inherited Retinal Disorders in Ireland. (11 citations)
- Findings from a Genotyping Study of Over 1000 People with Inherited Retinal Disorders in Ireland. (11 citations)
In his most recent research, the most cited papers focused on:
His main research concerns Retina, Claudin, Electroretinography, Exome sequencing and Genetic analysis.
His Retinal degeneration study in the realm of Retina interacts with subjects such as Retinopathy.
His work carried out in the field of Claudin brings together such families of science as Retinal pigment epithelium, Retinal and Atrophy, Pathology.
His Electroretinography research is multidisciplinary, incorporating perspectives in Cell biology, Photoreceptor cell, Rhodopsin, Programmed cell death and NAD+ kinase.
His studies deal with areas such as Proband, Pedigree chart, Genomics, Genotyping and Disease as well as Exome sequencing.
His Genetic analysis research incorporates themes from Computational biology and Retinal Disorder.
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