Olivia M. Pereira-Smith spends much of her time researching Molecular biology, Senescence, Cell culture, Gene and DNA synthesis. Much of her study explores Molecular biology relationship to DNA repair. Her work deals with themes such as Cell division and Telomerase, which intersect with Senescence.
In Cell culture, Olivia M. Pereira-Smith works on issues like Cell cycle, which are connected to Carcinogenesis, Immunology, Tumor suppressor gene and Cell type. In her research on the topic of Gene, RNA and Gene expression is strongly related with Cell growth. Her DNA synthesis study deals with Cyclin-dependent kinase intersecting with DNA replication and Proliferating cell nuclear antigen.
Olivia M. Pereira-Smith mainly focuses on Gene, Senescence, Molecular biology, Cell biology and Cell culture. Her study in the field of Chromosome 4, Gene expression and Complementary DNA is also linked to topics like Acquired immunodeficiency syndrome. Her Senescence study combines topics in areas such as Phenotype, Complementation, Transcription factor and Telomere.
Her studies in Molecular biology integrate themes in fields like DNA synthesis, Chromosome, Intracellular, DNA repair and Ploidy. Her Cell biology research includes themes of Cell cycle, Cell type, Telomerase and Cellular senescence. She combines subjects such as Cell and Cell growth with her study of Cell culture.
Cell biology, Gene, Molecular biology, Senescence and Genetics are her primary areas of study. Her Cell biology research includes elements of Cell culture, In vitro, In vivo and Cellular senescence. The Cell culture study combines topics in areas such as Cell division, Endoplasmic reticulum and Organelle.
Her work on Leucine zipper, Transcriptional repression and Downregulation and upregulation is typically connected to Chromodomain and Nuclear matrix as part of general Gene study, connecting several disciplines of science. Her Molecular biology research is multidisciplinary, relying on both NAB2, Zinc finger, Coding region, Chromosome and Cloning. The various areas that Olivia M. Pereira-Smith examines in her Senescence study include Transcription factor, Telomerase, Telomerase reverse transcriptase, DNA repair and Telomere.
Her main research concerns Molecular biology, Cell biology, Chromodomain, Sequence alignment and Gene family. Her study in Molecular biology is interdisciplinary in nature, drawing from both Telomere, Cell culture, Telomerase reverse transcriptase and DNA repair. Her Cell biology study frequently draws connections between adjacent fields such as Telomerase.
She integrates many fields in her works, including Chromodomain, Mutant, Gene, Leucine zipper, SUV39H1 and Histone acetyltransferase activity. Her research integrates issues of Drosophila melanogaster, Protein domain, Conserved sequence, Protein family and Subfamily in her study of Sequence alignment. Her Gene family research is within the category of Genetics.
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A biomarker that identifies senescent human cells in culture and in aging skin in vivo
G P Dimri;X Lee;G Basile;M Acosta.
Proceedings of the National Academy of Sciences of the United States of America (1995)
Cloning of senescent cell-derived inhibitors of DNA synthesis using an expression screen.
Asao Noda;Yi Ning;Susan F. Venable;Olivia M. Pereira-Smith.
Experimental Cell Research (1994)
A role for both RB and p53 in the regulation of human cellular senescence
Jerry W. Shay;Olivia M. Pereira-Smith;Woodring E. Wright.
Experimental Cell Research (1991)
Replicative senescence: implications for in vivo aging and tumor suppression.
James R. Smith;Olivia M. Pereira-Smith.
Genetic analysis of indefinite division in human cells: identification of four complementation groups.
Olivia M. Pereira-Smith;James R. Smith.
Proceedings of the National Academy of Sciences of the United States of America (1988)
Replicative senescence of human skin fibroblasts correlates with a loss of regulation and overexpression of collagenase activity
Michael D. West;Olivia M. Pereira-Smith;James R. Smith.
Experimental Cell Research (1989)
Expression of human telomerase (hTERT) does not prevent stress-induced senescence in normal human fibroblasts but protects the cells from stress-induced apoptosis and necrosis.
Vera Gorbunova;Andrei Seluanov;Olivia M. Pereira-Smith.
Journal of Biological Chemistry (2002)
DNA end joining becomes less efficient and more error-prone during cellular senescence
Andrei Seluanov;David Mittelman;Olivia M. Pereira-Smith;John H. Wilson.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Upregulation of mortalin/mthsp70/Grp75 contributes to human carcinogenesis.
Renu Wadhwa;Syuichi Takano;Kamaljit Kaur;Custer C. Deocaris;Custer C. Deocaris.
International Journal of Cancer (2006)
Existence of high abundance antiproliferative mRNA's in senescent human diploid fibroblasts.
CK Lumpkin;JK McClung;OM Pereira-Smith;Smith.
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