What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in Alzheimer's disease, Amyloid precursor protein, Cell biology, Cell culture and Pathology.
His studies in Alzheimer's disease integrate themes in fields like Pathogenesis and Amyloid.
In his study, P3 peptide is inextricably linked to Biochemistry, which falls within the broad field of Amyloid precursor protein.
His Cell biology study combines topics from a wide range of disciplines, such as Cadherin, Internal medicine, Presenilin and Molecular biology.
His studies in Cell culture integrate themes in fields like Exocytosis, Digitonin, Intracellular and Alpha secretase.
The study incorporates disciplines such as Chromosome and Beta in addition to Pathology.
His most cited work include:
- A presenilin‐1/γ‐secretase cleavage releases the E‐cadherin intracellular domain and regulates disassembly of adherens junctions (613 citations)
- Molecular cloning and characterization of a cDNA encoding the cerebrovascular and the neuritic plaque amyloid peptides. (534 citations)
- A CBP Binding Transcriptional Repressor Produced by the PS1/ϵ-Cleavage of N-Cadherin Is Inhibited by PS1 FAD Mutations (438 citations)
What are the main themes of his work throughout his whole career to date?
Nikolaos K. Robakis mainly focuses on Cell biology, Amyloid precursor protein, Biochemistry, Molecular biology and Presenilin.
The Cell biology study combines topics in areas such as Cadherin, Adherens junction, Cleavage, Receptor and Cell adhesion.
His Amyloid precursor protein research includes themes of Proteoglycan and Amyloid.
His work deals with themes such as Cell culture and Alpha secretase, which intersect with Biochemistry.
Nikolaos K. Robakis interconnects Complementary DNA, Genetics, Gene, Serine and Chromosome 21 in the investigation of issues within Molecular biology.
His work in Presenilin covers topics such as Neurodegeneration which are related to areas like Neuroprotection.
He most often published in these fields:
- Cell biology (37.86%)
- Amyloid precursor protein (30.00%)
- Biochemistry (24.29%)
What were the highlights of his more recent work (between 2007-2021)?
- Cell biology (37.86%)
- Neurodegeneration (10.00%)
- Amyloid precursor protein (30.00%)
In recent papers he was focusing on the following fields of study:
Nikolaos K. Robakis spends much of his time researching Cell biology, Neurodegeneration, Amyloid precursor protein, Neuroscience and Presenilin.
His Cell biology study combines topics from a wide range of disciplines, such as Receptor, Biochemistry and Cadherin.
As a member of one scientific family, he mostly works in the field of Neurodegeneration, focusing on Neuroprotection and, on occasion, Excitotoxicity, PI3K/AKT/mTOR pathway and Protein kinase B.
His research on Amyloid precursor protein focuses in particular on P3 peptide.
His Neuroscience research includes elements of Dementia, Alzheimer's disease, Neuropathology, Neurofibrillary tangle and Amyloid.
His Presenilin study incorporates themes from Messenger RNA and Amyloid precursor protein secretase.
Between 2007 and 2021, his most popular works were:
- Amyloid-Independent Mechanisms in Alzheimer's Disease Pathogenesis (222 citations)
- A Role for Noncoding Variation in Schizophrenia (172 citations)
- The CACNA1C and ANK3 risk alleles impact on affective personality traits and startle reactivity but not on cognition or gating in healthy males. (85 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Cell biology, Alzheimer's disease, Genetics, Genome-wide association study and Enhancer.
His Cell biology research integrates issues from Receptor and Receptor complex.
His Alzheimer's disease research is multidisciplinary, incorporating elements of Neurodegeneration, Neuroscience, Genotype and Amyloid.
His Neurodegeneration research incorporates elements of Biochemistry of Alzheimer's disease, Dementia, Pathogenesis and Neurotransmitter.
His Neuroscience research incorporates themes from P3 peptide, Amyloid precursor protein, Mutation, Wild type and Presenilin.
As a part of the same scientific family, Nikolaos K. Robakis mostly works in the field of Genome-wide association study, focusing on Expression quantitative trait loci and, on occasion, Human brain.
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