D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 76 Citations 18,585 199 World Ranking 1104 National Ranking 21
Medicine D-index 76 Citations 18,759 198 World Ranking 14094 National Ranking 213

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Alzheimer's disease

Constantin Bouras mainly investigates Pathology, Alzheimer's disease, Dementia, Neuroscience and Neurofibrillary tangle. Constantin Bouras is involved in the study of Pathology that focuses on Senile plaques in particular. The Alzheimer's disease study combines topics in areas such as Immunohistochemistry and Degenerative disease.

His studies in Dementia integrate themes in fields like Mental deterioration and Central nervous system disease. His Neurofibrillary tangle research incorporates themes from Neocortex and Hippocampal formation, Entorhinal cortex. As a part of the same scientific family, Constantin Bouras mostly works in the field of Entorhinal cortex, focusing on Cognitive decline and, on occasion, Cingulate cortex.

His most cited work include:

  • A mutation in a case of early onset narcolepsy and a generalized absence of hypocretin peptides in human narcoleptic brains (1674 citations)
  • Tangle and neuron numbers, but not amyloid load, predict cognitive status in Alzheimer's disease. (746 citations)
  • Tangle and neuron numbers, but not amyloid load, predict cognitive status in Alzheimer's disease. (746 citations)

What are the main themes of his work throughout his whole career to date?

Pathology, Alzheimer's disease, Neuroscience, Neurofibrillary tangle and Senile plaques are his primary areas of study. His biological study spans a wide range of topics, including Cerebral cortex, Temporal cortex and Hippocampus. His study focuses on the intersection of Alzheimer's disease and fields such as Posterior cingulate with connections in the field of Posterior cortical atrophy and Visual cortex.

His Neuroscience research is multidisciplinary, incorporating perspectives in Neuropathology, Disease, Neurodegeneration and Amyloid. The study incorporates disciplines such as Neocortex, Temporal lobe, Hippocampal formation, Entorhinal cortex and Clinical Dementia Rating in addition to Neurofibrillary tangle. His Senile plaques research includes elements of Tau protein and Atrophy.

He most often published in these fields:

  • Pathology (63.76%)
  • Alzheimer's disease (51.38%)
  • Neuroscience (45.87%)

What were the highlights of his more recent work (between 2006-2016)?

  • Neuroscience (45.87%)
  • Pathology (63.76%)
  • Cognitive decline (13.76%)

In recent papers he was focusing on the following fields of study:

His main research concerns Neuroscience, Pathology, Cognitive decline, Disease and Alzheimer's disease. His Neuroscience study combines topics in areas such as Alzheimer's disease research, Tau protein, Amyloid β and Senile plaques. His work carried out in the field of Pathology brings together such families of science as Peripheral nervous system and Dysphagia.

His study of Clinical Dementia Rating is a part of Alzheimer's disease. His Clinical Dementia Rating study integrates concerns from other disciplines, such as Neurofibrillary tangle and Entorhinal cortex. His work deals with themes such as Central nervous system disease and Degenerative disease, which intersect with Dementia.

Between 2006 and 2016, his most popular works were:

  • Anorexia induced by activation of serotonin 5-HT4 receptors is mediated by increases in CART in the nucleus accumbens (155 citations)
  • Identification of Alzheimer and vascular lesion thresholds for mixed dementia (133 citations)
  • Oxidative stress increases BACE1 protein levels through activation of the PKR-eIF2α pathway (118 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Gene
  • Alzheimer's disease

Constantin Bouras focuses on Cognitive decline, Alzheimer's disease, Pathology, Degenerative disease and Dementia. His Cognitive decline research integrates issues from Neuroscience and Senile plaques. His work on Cognitive disorder as part of general Neuroscience study is frequently linked to Context, therefore connecting diverse disciplines of science.

His Alzheimer's disease research includes themes of Cerebral cortex and Pathological. In his work, Parkinson's disease, Basal ganglia and Progressive supranuclear palsy is strongly intertwined with Neuropathology, which is a subfield of Degenerative disease. The concepts of his Neurofibrillary tangle study are interwoven with issues in Vascular dementia, Gliosis, Central nervous system disease and Subcortical dementia.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A mutation in a case of early onset narcolepsy and a generalized absence of hypocretin peptides in human narcoleptic brains

Christelle Peyron;Juliette Faraco;William Rogers;Beth Ripley.
Nature Medicine (2000)

2223 Citations

Tangle and neuron numbers, but not amyloid load, predict cognitive status in Alzheimer's disease.

Panteleimon Giannakopoulos;François Herrmann;T. Bussiere;T. Bussiere;Constantin Bouras;Constantin Bouras.
Neurology (2003)

1111 Citations

Evidence Supporting the Existence of an Activity-Dependent Astrocyte-Neuron Lactate Shuttle

Luc Pellerin;Giovanni Pellegri;Philippe G. Bittar;Yves Charnay.
Developmental Neuroscience (1998)

768 Citations

Selective Distribution of Lactate Dehydrogenase Isoenzymes in Neurons and Astrocytes of Human Brain

Philippe G. Bittar;Yves Charnay;Luc Pellerin;Constantin Bouras.
Journal of Cerebral Blood Flow and Metabolism (1996)

440 Citations

Regional Distribution of Neurofibrillary Tangles and Senile Plaques in the Cerebral Cortex of Elderly Patients: A Quantitative Evaluation of a One-Year Autopsy Population from a Geriatric Hospital

Constantin Bouras;Patrick R. Hof;Pantèleimon Giannakopoulos;Jean-Pierre Michel.
Cerebral Cortex (1994)

362 Citations

Atypical Form of Alzheimer's Disease With Prominent Posterior Cortical Atrophy: A Review of Lesion Distribution and Circuit Disconnection in Cortical Visual Pathways

Patrick R. Hof;Brent A. Vogt;Constantin Bouras;Constantin Bouras;John H. Morrison.
Vision Research (1997)

320 Citations

Selective disconnection of specific visual association pathways in cases of Alzheimer's disease presenting with Balint's syndrome.

Patrick R. Hof;Constantin Bouras;Jean Constantinidis;John H. Morrison.
Journal of Neuropathology and Experimental Neurology (1990)

304 Citations

Age-related distribution of neuropathologic changes in the cerebral cortex of patients with Down's syndrome. Quantitative regional analysis and comparison with Alzheimer's disease.

P. R. Hof;C. Bouras;D. P. Perl;D. L. Sparks.
JAMA Neurology (1995)

303 Citations

Down patients: extracellular preamyloid deposits precede neuritic degeneration and senile plaques.

Giorgio Giaccone;Fabrizio Tagliavini;Giovanni Linoli;Constantin Bouras.
Neuroscience Letters (1989)

302 Citations

Sensitivity and specificity of newly proposed clinical criteria for possible vascular dementia

G. Gold;P. Giannakopoulos;C. Montes-Paixao;F. R. Herrman.
Neurology (1997)

298 Citations

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