2016 - Member of the National Academy of Medicine (NAM)
2006 - Fellow of the American Association for the Advancement of Science (AAAS)
1931 - Fellow of the American Association for the Advancement of Science (AAAS)
John H. Morrison spends much of his time researching Neuroscience, Prefrontal cortex, Hippocampus, Dendritic spine and Neocortex. John H. Morrison combines topics linked to Alzheimer's disease with his work on Neuroscience. His Prefrontal cortex research focuses on subjects like Synapse, which are linked to Hippocampus.
His Hippocampus research includes themes of Hippocampal formation, Cognition, Neurotransmission and Cognitive decline. His Dendritic spine study combines topics from a wide range of disciplines, such as Synaptic plasticity, Postsynaptic density, Spine, Pyramidal cell and Estrogen. Tyrosine hydroxylase, Dopaminergic and Cytoarchitecture is closely connected to Sensory system in his research, which is encompassed under the umbrella topic of Neocortex.
John H. Morrison mainly investigates Neuroscience, Internal medicine, Hippocampus, Endocrinology and Neocortex. His Neuroscience study frequently draws parallels with other fields, such as Alzheimer's disease. His biological study deals with issues like NMDA receptor, which deal with fields such as Protein subunit.
His study in the field of Estrogen and Hypothalamus is also linked to topics like Estrogen receptor. He interconnects Glutamate receptor, Cortex and Neurotransmitter in the investigation of issues within Neocortex. His work on Dorsolateral prefrontal cortex as part of general Prefrontal cortex research is often related to Lucifer yellow, thus linking different fields of science.
John H. Morrison mostly deals with Neuroscience, Dendritic spine, Internal medicine, Prefrontal cortex and Endocrinology. The study of Neuroscience is intertwined with the study of Cognitive decline in a number of ways. His study in Dendritic spine is interdisciplinary in nature, drawing from both Synaptic plasticity, Nucleus accumbens, Spine and Primate.
His Prefrontal cortex research integrates issues from Aging brain, Working memory, Neuroplasticity and Pyramidal cell. His Estrogen study in the realm of Endocrinology interacts with subjects such as Estrogen receptor and Lucifer yellow. In his research, Alzheimer's disease and Reproductive senescence is intimately related to Hippocampal formation, which falls under the overarching field of Hippocampus.
His scientific interests lie mostly in Neuroscience, Prefrontal cortex, Dendritic spine, Hippocampus and Cognitive decline. His study in Synapse, Dorsolateral prefrontal cortex, Nucleus accumbens, Cognition and Neuroplasticity falls within the category of Neuroscience. The Prefrontal cortex study combines topics in areas such as Aging brain, Endocrinology, Life course approach and Internal medicine.
His Dendritic spine research is multidisciplinary, incorporating elements of Deconvolution, Neuroanatomy, Spine and Confocal microscopy, Cell biology. His study ties his expertise on Hippocampal formation together with the subject of Hippocampus. His work focuses on many connections between Cognitive decline and other disciplines, such as Riluzole, that overlap with his field of interest in Glutamatergic, NMDA receptor, Alzheimer's disease, Glutamic acid and Psychopharmacology.
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Obesity and the metabolic syndrome in children and adolescents.
Ram Weiss;James Dziura;Tania S. Burgert;William V. Tamborlane.
The New England Journal of Medicine (2004)
Life and Death of Neurons in the Aging Brain
John H. Morrison;Patrick R. Hof.
Enhanced long-term potentiation and impaired learning in mice with mutant postsynaptic density-95 protein
Martine Migaud;Paul Charlesworth;Maureen Dempster;Lorna C. Webster.
Tangle and neuron numbers, but not amyloid load, predict cognitive status in Alzheimer's disease.
Panteleimon Giannakopoulos;François Herrmann;T. Bussiere;T. Bussiere;Constantin Bouras;Constantin Bouras.
Stress-Induced Alterations in Prefrontal Cortical Dendritic Morphology Predict Selective Impairments in Perceptual Attentional Set-Shifting
Conor Liston;Melinda M. Miller;Deena S. Goldwater;Jason J. Radley.
The Journal of Neuroscience (2006)
Chronic behavioral stress induces apical dendritic reorganization in pyramidal neurons of the medial prefrontal cortex
J. J. Radley;J. J. Radley;H. M. Sisti;J. Hao;A. B. Rocher.
Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis
Michael E. Ripps;George W. Huntley;Patrick R. Hof;John H. Morrison.
Proceedings of the National Academy of Sciences of the United States of America (1995)
Repeated Stress Induces Dendritic Spine Loss in the Rat Medial Prefrontal Cortex
Jason J. Radley;Anne B. Rocher;Melinda Miller;William G.M. Janssen.
Cerebral Cortex (2006)
The Brain on Stress: Vulnerability and Plasticity of the Prefrontal Cortex over the Life Course
Bruce S. McEwen;John H. Morrison.
Early-onset behavioral and synaptic deficits in a mouse model of Alzheimer's disease
J. Steven Jacobsen;Chi-Cheng Wu;Jeffrey M. Redwine;Thomas A. Comery.
Proceedings of the National Academy of Sciences of the United States of America (2006)
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