Eli Lilly (United States)
Matthew D. Breyer spends much of his time researching Internal medicine, Endocrinology, Receptor, Renal function and Cyclooxygenase. Kidney, Prostaglandin E, Renal physiology, Kidney disease and Macula densa are subfields of Internal medicine in which his conducts study. His Macula densa research includes themes of Renal glomerulus and Renal cortex.
His research integrates issues of Peroxisome proliferator-activated receptor and Nephrology in his study of Endocrinology. His work on Prostaglandin E2 receptor as part of general Receptor research is frequently linked to Class C GPCR, bridging the gap between disciplines. His Renal function study integrates concerns from other disciplines, such as Nitric oxide synthase, Glomerular basement membrane, Inulin and Streptozotocin.
His primary areas of investigation include Internal medicine, Endocrinology, Kidney, Receptor and Diabetic nephropathy. His Internal medicine research integrates issues from Gene expression and Eicosanoid. As a member of one scientific family, Matthew D. Breyer mostly works in the field of Endocrinology, focusing on Cyclooxygenase and, on occasion, Renal cortex.
His Kidney research incorporates elements of Renin–angiotensin system, Macula densa, Pharmacology and In situ hybridization. His Receptor research incorporates themes from Molecular biology and Signal transduction. Matthew D. Breyer interconnects Nephropathy, Disease, Immunology and Glomerular basement membrane in the investigation of issues within Diabetic nephropathy.
His scientific interests lie mostly in Internal medicine, Kidney disease, Albuminuria, Disease and Diabetic nephropathy. His work on Internal medicine is being expanded to include thematically relevant topics such as Endocrinology. His study in the fields of Kidney and Prostaglandin E2 under the domain of Endocrinology overlaps with other disciplines such as Mean arterial pressure.
His Kidney disease study incorporates themes from Patient satisfaction, Nephrology, Clinical trial, Oncology and Immunology. Matthew D. Breyer works mostly in the field of Disease, limiting it down to concerns involving Diabetic kidney and, occasionally, Creatinine, Nephron and TGF alpha. His Diabetic nephropathy research is multidisciplinary, incorporating perspectives in Disease pathogenesis, Therapeutic trial, Oxidative injury and Intensive care medicine.
His primary areas of study are Internal medicine, Diabetic nephropathy, Kidney disease, Endocrinology and Disease. Many of his research projects under Internal medicine are closely connected to Public attention, Redress and Osteomalacia with Public attention, Redress and Osteomalacia, tying the diverse disciplines of science together. His work carried out in the field of Diabetic nephropathy brings together such families of science as Pathology, Albuminuria, Renal function and Type 2 diabetes.
His Kidney disease study combines topics in areas such as Patient satisfaction, Nephrology and Clinical trial. Endocrinology is often connected to Prostaglandin E2 receptor in his work. His research investigates the connection between Disease and topics such as Fibrosis that intersect with problems in Inflammation.
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Prostanoid receptors: subtypes and signaling.
Richard M Breyer;Carey K Bagdassarian;Scott A Myers;Matthew D Breyer.
Annual Review of Pharmacology and Toxicology (2001)
Cyclooxygenase-2 is associated with the macula densa of rat kidney and increases with salt restriction.
Raymond C. Harris;James A. McKanna;Yoichi Akai;Harry R. Jacobson.
Journal of Clinical Investigation (1994)
Mouse Models of Diabetic Nephropathy
Frank C. Brosius;Charles E. Alpers;Erwin P. Bottinger;Matthew D. Breyer.
Journal of The American Society of Nephrology (2009)
Thiazolidinediones expand body fluid volume through PPARγ stimulation of ENaC-mediated renal salt absorption
You Fei Guan;Chuanming Hao;Dae Ryong Cha;Reena Rao.
Nature Medicine (2005)
Mesangial cell, glomerular and renal vascular responses to endothelin in the rat kidney. Elucidation of signal transduction pathways.
K F Badr;J J Murray;M D Breyer;K Takahashi.
Journal of Clinical Investigation (1989)
Peroxisome proliferator-activated receptors (PPARs): Novel therapeutic targets in renal disease
YouFei Guan;Matthew D. Breyer.
Kidney International (2001)
SALT-SENSITIVE HYPERTENSION AND REDUCED FERTILITY IN MICE LACKING THE PROSTAGLANDIN EP2 RECEPTOR
Christopher R.J. Kennedy;Yahua Zhang;Suzanne Brandon;Youfei Guan.
Nature Medicine (1999)
Physiological regulation of cyclooxygenase-2 in the kidney
Raymond C. Harris;Matthew D. Breyer.
American Journal of Physiology-renal Physiology (2001)
Endothelial Nitric Oxide Synthase Deficiency Produces Accelerated Nephropathy in Diabetic Mice
Hui John Zhao;Suwan Wang;Huifang Cheng;Ming-zhi Zhang.
Journal of The American Society of Nephrology (2006)
Serial determination of glomerular filtration rate in conscious mice using FITC-inulin clearance
Zhonghua Qi;Zhonghua Qi;Irene Whitt;Amit Mehta;Jianping Jin.
American Journal of Physiology-renal Physiology (2004)
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