Ambra Pozzi mainly focuses on Cell biology, Integrin, Receptor, Endocrinology and Internal medicine. Her Cell biology research is multidisciplinary, incorporating elements of Endothelial stem cell, Angiogenesis and Cortactin. Ambra Pozzi mostly deals with Collagen receptor in her studies of Integrin.
Her studies in Receptor integrate themes in fields like Nuclear receptor, Cancer research, Cell migration and Ex vivo. When carried out as part of a general Endocrinology research project, her work on Transforming growth factor, Kidney and Pathophysiology of hypertension is frequently linked to work in Sodium channel blocker, therefore connecting diverse disciplines of study. Her work in the fields of Internal medicine, such as Blood pressure, intersects with other areas such as Epithelial sodium channel, Amiloride, Renal sodium reabsorption and Vascular smooth muscle.
Her primary scientific interests are in Cell biology, Integrin, Internal medicine, Endocrinology and Cancer research. Her Cell biology research includes themes of Receptor and Cell growth. As a part of the same scientific family, Ambra Pozzi mostly works in the field of Integrin, focusing on Molecular biology and, on occasion, Phosphorylation.
Her Internal medicine study incorporates themes from Kinase and Epoxygenase. Her research on Endocrinology frequently connects to adjacent areas such as Endothelial stem cell. She combines subjects such as Carcinogenesis, Peroxisome proliferator-activated receptor, Lung cancer and Epidermal growth factor receptor with her study of Cancer research.
Her primary areas of investigation include Cell biology, Integrin, Internal medicine, Endocrinology and Receptor. She has researched Cell biology in several fields, including Cell migration, Kidney and Cell adhesion. Her work deals with themes such as Biophysics, Knockout mouse, Calcium and Actin cytoskeleton, which intersect with Integrin.
Her Internal medicine study deals with Extracellular matrix intersecting with Glomerulosclerosis. The concepts of her Endocrinology study are interwoven with issues in Hepatocyte and Kinase activity. Her Receptor study integrates concerns from other disciplines, such as Peptide, Nuclear localization sequence and Phosphorylation.
Her primary areas of study are Integrin, Cell biology, Internal medicine, Endocrinology and Cytochrome P450. Her biological study spans a wide range of topics, including Protein kinase B, Cell migration, Cell adhesion and Basement membrane. Much of her study explores Cell biology relationship to Fibrosis.
Insulin resistance and Receptor are the core of her Internal medicine study. Her studies deal with areas such as Extracellular matrix and Signal transduction as well as Receptor. Her Cytochrome P450 research incorporates themes from Carcinogenesis, Arachidonic acid and Biosynthesis.
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Elevated matrix metalloprotease and angiostatin levels in integrin α1 knockout mice cause reduced tumor vascularization
Ambra Pozzi;Philip E. Moberg;Lindsey A. Miles;Simone Wagner.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Soluble Eph A receptors inhibit tumor angiogenesis and progression in vivo.
Dana M Brantley;Nikki Cheng;Erin J Thompson;Qing Lin.
The juxtamembrane region of the EGF receptor functions as an activation domain.
Monica Red Brewer;Sung Hee Choi;Diego Alvarado;Katarina Moravcevic.
Molecular Cell (2009)
Integrin α1β1 Mediates a Unique Collagen-dependent Proliferation Pathway In Vivo
Ambra Pozzi;Kishore K. Wary;Filippo G. Giancotti;Humphrey A. Gardner.
Journal of Cell Biology (1998)
EphA2 receptor tyrosine kinase regulates endothelial cell migration and vascular assembly through phosphoinositide 3-kinase-mediated Rac1 GTPase activation.
Dana M. Brantley-Sieders;Justin Caughron;Donna Hicks;Ambra Pozzi.
Journal of Cell Science (2004)
Absence of integrin alpha1beta1 in the mouse causes loss of feedback regulation of collagen synthesis in normal and wounded dermis.
Humphrey Gardner;Arsi Broberg;Ambra Pozzi;Mattie Laato.
Journal of Cell Science (1999)
Transforming Growth Factor β Receptor Type II Inactivation Induces the Malignant Transformation of Intestinal Neoplasms Initiated by Apc Mutation
Nina M. Muñoz;Melissa Upton;Andres Rojas;Andres Rojas;M. Kay Washington.
Cancer Research (2006)
The nature and biology of basement membranes.
Ambra Pozzi;Ambra Pozzi;Peter D. Yurchenco;Renato V. Iozzo.
Matrix Biology (2017)
Low plasma levels of matrix metalloproteinase 9 permit increased tumor angiogenesis.
Ambra Pozzi;Wendy F LeVine;Humphrey A Gardner.
Characterization of 5,6- and 8,9-Epoxyeicosatrienoic Acids (5,6- and 8,9-EET) as Potent in Vivo Angiogenic Lipids
Ambra Pozzi;Ines Macias-Perez;Tristin Abair;Shouzuo Wei.
Journal of Biological Chemistry (2005)
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