D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 45 Citations 17,616 49 World Ranking 15447 National Ranking 1130

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Transcription factor

Masahiro Kawabata mostly deals with SMAD, Bone morphogenetic protein, Molecular biology, Cell biology and Mothers against decapentaplegic homolog 3. SMAD is a subfield of Biochemistry that he studies. His work on BMPR2 and Bone morphogenetic protein receptor as part of his general Bone morphogenetic protein study is frequently connected to Bone morphogenetic protein 2 and Osteoblast, thereby bridging the divide between different branches of science.

His Molecular biology research is multidisciplinary, incorporating elements of Receptor, Core binding factor and Mitogen-activated protein kinase kinase. His work on Thioredoxin expands to the thematically related Cell biology. His research in the fields of Kinase activity overlaps with other disciplines such as Inhibitor of apoptosis.

His most cited work include:

  • Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1. (2078 citations)
  • Identification of Smad7, a TGFβ-inducible antagonist of TGF-β signalling (1568 citations)
  • Smad6 inhibits signalling by the TGF-β superfamily (933 citations)

What are the main themes of his work throughout his whole career to date?

Cell biology, Molecular biology, SMAD, Signal transduction and Bone morphogenetic protein are his primary areas of study. His research integrates issues of Receptor, Receptor complex and Endocrinology in his study of Cell biology. His studies in Molecular biology integrate themes in fields like Amino acid, Mothers against decapentaplegic homolog 2, Transcription factor, DNA and Binding site.

His study in the fields of Mothers against decapentaplegic homolog 3 under the domain of SMAD overlaps with other disciplines such as R-SMAD. The Transforming growth factor beta and STAT3 research Masahiro Kawabata does as part of his general Signal transduction study is frequently linked to other disciplines of science, such as Inhibitor of apoptosis, therefore creating a link between diverse domains of science. When carried out as part of a general Bone morphogenetic protein research project, his work on BMPR2 and Bone morphogenetic protein 10 is frequently linked to work in Bone morphogenetic protein 2, GDF2 and Osteoblast, therefore connecting diverse disciplines of study.

He most often published in these fields:

  • Cell biology (52.94%)
  • Molecular biology (50.98%)
  • SMAD (50.98%)

What were the highlights of his more recent work (between 2000-2013)?

  • Cell biology (52.94%)
  • Signal transduction (27.45%)
  • SMAD (50.98%)

In recent papers he was focusing on the following fields of study:

Masahiro Kawabata mainly investigates Cell biology, Signal transduction, SMAD, Cancer research and Molecular biology. His Cell biology research incorporates themes from HMG-box, bZIP domain and Binding domain, Binding site. As a part of the same scientific study, he usually deals with the Signal transduction, concentrating on Transforming growth factor and frequently concerns with Coactivator, Transactivation, Calcitriol receptor, Long-term potentiation and Vitamin D and neurology.

His SMAD research includes themes of Cellular differentiation and Protein kinase A. His study focuses on the intersection of Cancer research and fields such as Regulation of gene expression with connections in the field of STAT3, Activator, STAT3 Transcription Factor, Hepatic stellate cell and Cytokine. In his research on the topic of Molecular biology, Mothers against decapentaplegic homolog 3 is strongly related with Bone morphogenetic protein.

Between 2000 and 2013, his most popular works were:

  • Ligand-dependent degradation of Smad3 by a ubiquitin ligase complex of ROC1 and associated proteins. (205 citations)
  • Smads, TAK1, and their common target ATF-2 play a critical role in cardiomyocyte differentiation. (130 citations)
  • Positive and Negative Modulation of Vitamin D Receptor Function by Transforming Growth Factor-β Signaling through Smad Proteins (89 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Transcription factor

His primary areas of study are Cell biology, Signal transduction, SMAD, Ubiquitin and Ubiquitin ligase complex. Masahiro Kawabata has included themes like Long-term potentiation and Bone morphogenetic protein in his Cell biology study. Masahiro Kawabata combines subjects such as Transforming growth factor, Endocrinology, Coactivator, Transactivation and Calcitriol receptor with his study of Long-term potentiation.

Calcitriol receptor is a subfield of Vitamin D and neurology that Masahiro Kawabata tackles. He has researched Bone morphogenetic protein in several fields, including Molecular biology, Transcription factor, CREB and Protein kinase A. His Ubiquitin research is multidisciplinary, relying on both Transforming growth factor beta, DNA-binding protein and Phosphorylation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

Masao Saitoh;Hideki Nishitoh;Makiko Fujii;Kohsuke Takeda.
The EMBO Journal (1998)

2820 Citations

Identification of Smad7, a TGFβ-inducible antagonist of TGF-β signalling

Atsuhito Nakao;Mozhgan Afrakhte;Anita Morn;Takuya Nakayama.
Nature (1997)

2295 Citations

Smad6 inhibits signalling by the TGF-Beta superfamily

Takeshi Imamura;Masao Takase;Ayako Nishihara;Eiichi Oeda.
Nature (1997)

1365 Citations

TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4.

Atsuhito Nakao;Takeshi Imamura;Takeshi Imamura;Serhiy Souchelnytskyi;Masahiro Kawabata.
The EMBO Journal (1997)

1291 Citations

Synergistic signaling in fetal brain by STAT3-Smad1 complex bridged by p300.

Kinichi Nakashima;Makoto Yanagisawa;Hirokazu Arakawa;Naoki Kimura.
Science (1999)

960 Citations

Signal transduction by bone morphogenetic proteins.

Masahiro Kawabata;Takeshi Imamura;Kohei Miyazono.
Cytokine & Growth Factor Reviews (1998)

764 Citations

Convergence of Transforming Growth Factor-β and Vitamin D Signaling Pathways on SMAD Transcriptional Coactivators

Junn Yanagisawa;Yasuo Yanagi;Yoshikazu Masuhiro;Miyuki Suzawa.
Science (1999)

620 Citations

Roles of Bone Morphogenetic Protein Type I Receptors and Smad Proteins in Osteoblast and Chondroblast Differentiation

M Fujii;K Takeda;T Imamura;H Aoki.
Molecular Biology of the Cell (1999)

590 Citations

BMP type II receptor is required for gastrulation and early development of mouse embryos.

Hideyuki Beppu;Masahiro Kawabata;Toshiaki Hamamoto;Anna Chytil.
Developmental Biology (2000)

572 Citations

Interaction and Functional Cooperation of PEBP2/CBF with Smads SYNERGISTIC INDUCTION OF THE IMMUNOGLOBULIN GERMLINE Cα PROMOTER

Jun Ichi Hanai;Lin Feng Chen;Tomohiko Kanno;Naoko Ohtani-Fujita.
Journal of Biological Chemistry (1999)

554 Citations

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