CTGF, Growth factor, Connective tissue, Internal medicine and Endocrinology are his primary areas of study. His CTGF research is multidisciplinary, relying on both Fibrosis, Pathology, Molecular biology and Immunology. His work deals with themes such as Cancer research, Angiogenesis, Vascular endothelial growth factor A and Extracellular matrix, Cell biology, which intersect with Growth factor.
Masaharu Takigawa interconnects Gene expression, Basic fibroblast growth factor, Messenger RNA and Cartilage, Endochondral ossification in the investigation of issues within Cell biology. His study looks at the intersection of Internal medicine and topics like Immediate early protein with Dermatomyositis, Systemic disease and Gastroenterology. His Endocrinology research is multidisciplinary, incorporating elements of Phenotype, Congenital muscular dystrophy, Parathyroid hormone and Ornithine decarboxylase.
His main research concerns Cell biology, CTGF, Molecular biology, Growth factor and Internal medicine. His Cell biology research incorporates elements of Immunology and Chondrocyte, Cartilage, Endochondral ossification. His study in CTGF is interdisciplinary in nature, drawing from both Extracellular matrix, Connective tissue, Regeneration, Fibrosis and Regulation of gene expression.
His Molecular biology research integrates issues from Cell culture, Messenger RNA, Gene expression, Northern blot and Monoclonal antibody. As a member of one scientific family, Masaharu Takigawa mostly works in the field of Growth factor, focusing on Cancer research and, on occasion, Metastasis and Cancer cell. As part of his studies on Internal medicine, he frequently links adjacent subjects like Endocrinology.
His primary areas of investigation include Cell biology, Growth factor, CTGF, Cartilage and Molecular biology. His Cell biology study combines topics in areas such as Chondrocyte, Cell culture and Endochondral ossification. His work carried out in the field of Growth factor brings together such families of science as Endocrinology, Connective tissue and Fibroblast growth factor.
His biological study spans a wide range of topics, including Regulation of gene expression, Signal transduction and Matricellular protein. The Cartilage study combines topics in areas such as Immunology, Pathology, Regeneration, Extracellular matrix and Genetically modified mouse. His study on Molecular biology also encompasses disciplines like
Masaharu Takigawa focuses on Cell biology, Growth factor, Endocrinology, Internal medicine and Cancer research. His Cell biology research includes themes of Chondrocyte, Cartilage, Endochondral ossification and Cellular differentiation. Masaharu Takigawa combines subjects such as Extracellular matrix, Gene expression and Immunology with his study of Cartilage.
He is studying CTGF, which is a component of Growth factor. As part of the same scientific family, Masaharu Takigawa usually focuses on CTGF, concentrating on Regulation of gene expression and intersecting with Extracellular. His study in the field of Osteoprotegerin is also linked to topics like Macrophage colony-stimulating factor.
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Role and interaction of connective tissue growth factor with transforming growth factor-beta in persistent fibrosis: A mouse fibrosis model.
Toshifumi Mori;Shigeru Kawara;Mikio Shinozaki;Nobukazu Hayashi.
Journal of Cellular Physiology (1999)
A functional polymorphism in the 5′ UTR of GDF5 is associated with susceptibility to osteoarthritis
Yoshinari Miyamoto;Akihiko Mabuchi;Dongquan Shi;Toshikazu Kubo.
Nature Genetics (2007)
Connective tissue growth factor induces the proliferation, migration, and tube formation of vascular endothelial cells in vitro, and angiogenesis in vivo.
Tsuyoshi Shimo;Tohru Nakanishi;Takashi Nishida;Masahiro Asano.
Journal of Biochemistry (1999)
Nitric oxide mediates interleukin-1-induced gene expression of matrix metalloproteinases and basic fibroblast growth factor in cultured rabbit articular chondrocytes
Kazuhiro Sasaki;Takako Hattori;Takuo Fujisawa;Kojiro Takahashi.
Journal of Biochemistry (1998)
Reduction in Connective Tissue Growth Factor by Antisense Treatment Ameliorates Renal Tubulointerstitial Fibrosis
Hideki Yokoi;Masashi Mukoyama;Tetsuya Nagae;Kiyoshi Mori.
Journal of The American Society of Nephrology (2004)
Serum levels of connective tissue growth factor are elevated in patients with systemic sclerosis: association with extent of skin sclerosis and severity of pulmonary fibrosis.
Shinichi Sato;Tetsuya Nagaoka;Minoru Hasegawa;Takuya Tamatani.
The Journal of Rheumatology (2000)
Effects of CTGF/Hcs24, a product of a hypertrophic chondrocyte-specific gene, on the proliferation and differentiation of chondrocytes in culture.
Tohru Nakanishi;Takashi Nishida;Tsuyoshi Shimo;Kappei Kobayashi.
Proposal for a unified CCN nomenclature
D. R. Brigstock;R. Goldschmeding;K. I. Katsube;S. C.T. Lam.
Journal of Clinical Pathology-molecular Pathology (2003)
Establishment of a Clonal Human Chondrosarcoma Cell Line with Cartilage Phenotypes
Masaharu Takigawa;Koji Tajima;Hai Ou Pan;Motomi Enomoto.
Cancer Research (1989)
Cloning of a mRNA Preferentially Expressed in Chondrocytes by Differential Display-PCR from a Human Chondrocytic Cell Line That Is Identical with Connective Tissue Growth Factor (CTGF) mRNA
Tohru Nakanishi;Yusuke Kimura;Tomoo Tamura;Hiroyuki Ichikawa.
Biochemical and Biophysical Research Communications (1997)
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