D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 47 Citations 7,739 117 World Ranking 14718 National Ranking 605

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Cancer

Bernard Perbal spends much of his time researching Cell biology, CTGF, CCN protein, Molecular biology and CYR61. His work deals with themes such as Dermis, Cell adhesion and Melanocyte, which intersect with Cell biology. As part of one scientific family, Bernard Perbal deals mainly with the area of CTGF, narrowing it down to issues related to the Transforming growth factor, and often Cell signaling, Hepatic stellate cell, Endogeny and Fibrosis.

His research investigates the link between CCN protein and topics such as CCN Intercellular Signaling Proteins that cross with problems in Sequence alignment, Function, Peptide sequence, Bone morphogenetic protein and Protein structure. His CYR61 study incorporates themes from Immediate early protein and Connective tissue. Bernard Perbal combines subjects such as Carcinogenesis and Pathology with his study of Immediate early protein.

His most cited work include:

  • CCN proteins: multifunctional signalling regulators. (597 citations)
  • The CCN family of proteins: structure-function relationships. (285 citations)
  • The CCN family of proteins: structure-function relationships. (285 citations)

What are the main themes of his work throughout his whole career to date?

Bernard Perbal mainly investigates Cell biology, Cancer research, Pathology, Molecular biology and Gene. His Cell biology research integrates issues from Endocrinology, Cell growth and CYR61, Growth factor, CTGF. His research integrates issues of Protein kinase B, Tyrosine kinase and MAP2K7, Protein kinase R, Cyclin-dependent kinase 2 in his study of Cancer research.

His work deals with themes such as Cancer, Extracellular matrix, Matricellular protein and Immediate early protein, which intersect with Pathology. The various areas that he examines in his Molecular biology study include Regulation of gene expression, Messenger RNA and Gene expression. His study in Gene is interdisciplinary in nature, drawing from both Kidney and Ccn family.

He most often published in these fields:

  • Cell biology (40.30%)
  • Cancer research (24.63%)
  • Pathology (23.88%)

What were the highlights of his more recent work (between 2008-2019)?

  • Pathology (23.88%)
  • Cell biology (40.30%)
  • Internal medicine (19.40%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Pathology, Cell biology, Internal medicine, Endocrinology and Cancer research. His Pathology study combines topics from a wide range of disciplines, such as Extracellular matrix, Matricellular protein and Experimental pathology. His Cell biology research includes themes of Inflammation, Cell adhesion and Pseudopodia.

His Primary biliary cirrhosis, Hepatic stellate cell, Hepatic fibrosis and Tumor suppressor gene study in the realm of Internal medicine interacts with subjects such as Alcoholic liver disease. The Endocrinology study combines topics in areas such as Regulator, Downregulation and upregulation, CYR61, CTGF and Fibrosis. The concepts of his Cancer research study are interwoven with issues in Cancer and Bone marrow.

Between 2008 and 2019, his most popular works were:

  • CCN3 (NOV) is a negative regulator of CCN2 (CTGF) and a novel endogenous inhibitor of the fibrotic pathway in an in vitro model of renal disease. (101 citations)
  • CCN3 Inhibits Neointimal Hyperplasia Through Modulation of Smooth Muscle Cell Growth and Migration (66 citations)
  • Susceptibility to liver fibrosis in mice expressing a connective tissue growth factor transgene in hepatocytes. (63 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Cancer

The scientist’s investigation covers issues in Internal medicine, Endocrinology, Cell biology, CTGF and Fibrosis. His study in the field of Osteoclast and Metastatic breast cancer is also linked to topics like Osteoprotegerin and RANKL. His Endocrinology study incorporates themes from Breast cancer, Breast disease, Hepatic fibrosis and Primary bone.

His Inflammation research extends to Cell biology, which is thematically connected. Particularly relevant to CYR61 is his body of work in CTGF. He focuses mostly in the field of Fibrosis, narrowing it down to matters related to Endogeny and, in some cases, Downregulation and upregulation, Diabetes mellitus, Diabetic nephropathy, Renal fibrosis and Kidney.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

CCN proteins: multifunctional signalling regulators.

Bernard Perbal.
The Lancet (2004)

960 Citations

NOV (nephroblastoma overexpressed) and the CCN family of genes: structural and functional issues

B Perbal.
Journal of Clinical Pathology-molecular Pathology (2001)

490 Citations

Proviral rearrangements and overexpression of a new cellular gene (nov) in myeloblastosis-associated virus type 1-induced nephroblastomas.

V Joliot;C Martinerie;G Dambrine;G Plassiart.
Molecular and Cellular Biology (1992)

429 Citations

Proposal for a unified CCN nomenclature

D. R. Brigstock;R. Goldschmeding;K. I. Katsube;S. C.T. Lam.
Journal of Clinical Pathology-molecular Pathology (2003)

282 Citations

The Nephroblastoma Overexpressed Gene (NOV/ccn3) Protein Associates with Notch1 Extracellular Domain and Inhibits Myoblast Differentiation via Notch Signaling Pathway

Kei Sakamoto;Shunji Yamaguchi;R. Ando;Atsushi Miyawaki.
Journal of Biological Chemistry (2002)

261 Citations

CCN3 (NOV) Interacts with Connexin43 in C6 Glioma Cells POSSIBLE MECHANISM OF CONNEXIN-MEDIATED GROWTH SUPPRESSION

Christine T. Fu;John F. Bechberger;Mark A. Ozog;Bernard Perbal.
Journal of Biological Chemistry (2004)

228 Citations

A structural approach to the role of CCN (CYR61/CTGF/NOV) proteins in tumourigenesis

Nathalie Planque;Bernard Perbal.
Cancer Cell International (2003)

216 Citations

novH: differential expression in developing kidney and Wilm's tumors.

G. Chevalier;H. Yeger;C. Martinerie;M. Laurent.
American Journal of Pathology (1998)

207 Citations

Ccn Proteins: A New Family of Cell Growth and Differentiation Regulators

Bernard Perbal;Masaharu Takigawa.
(2005)

194 Citations

Connexin43 Interacts with NOV A POSSIBLE MECHANISM FOR NEGATIVE REGULATION OF CELL GROWTH IN CHORIOCARCINOMA CELLS

Alexandra Gellhaus;Xuesen Dong;Sven Propson;Karen Maass.
Journal of Biological Chemistry (2004)

179 Citations

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