His main research concerns Immunology, Immune system, Virology, Microbiology and Molecular biology. While the research belongs to areas of Immunology, he spends his time largely on the problem of Cytotoxic T cell, intersecting his research to questions surrounding Peripheral blood mononuclear cell and Receptor. His Immune system research incorporates themes from Chlamydia, Natural killer cell and Antigen.
Martin E. Rottenberg has researched Virology in several fields, including Laminin, Trypanosoma cruzi, Antibody and Blood–brain barrier. As a member of one scientific family, Martin E. Rottenberg mostly works in the field of Trypanosoma cruzi, focusing on Spleen and, on occasion, Cellular immunity and Interferon gamma. His studies deal with areas such as Listeria monocytogenes, Secretion, Flagellum and Chemotaxis as well as Microbiology.
His scientific interests lie mostly in Immunology, Immune system, Microbiology, Virology and Cytokine. The various areas that Martin E. Rottenberg examines in his Immunology study include SOCS3 and Mycobacterium tuberculosis. His Immune system research is multidisciplinary, relying on both Granuloma and Monocyte.
The concepts of his Microbiology study are interwoven with issues in Chlamydia, Mycobacterium, Secretion, Listeria monocytogenes and Nitric oxide. His research integrates issues of Trypanosoma cruzi and CD8 in his study of Virology. Martin E. Rottenberg combines subjects such as Molecular biology, Cytotoxic T cell and Cyclosporin a with his study of T cell.
Martin E. Rottenberg mainly investigates Immunology, Mycobacterium tuberculosis, Cytokine, Immune system and Microbiology. Martin E. Rottenberg regularly links together related areas like Stimulation in his Immunology studies. His work deals with themes such as Tumor necrosis factor alpha and Antigen, which intersect with Mycobacterium tuberculosis.
In his research on the topic of Cytokine, Interleukin 6 and Chemokine is strongly related with SOCS3. He studies Immune system, focusing on FOXP3 in particular. The study incorporates disciplines such as Thioredoxin and Granuloma in addition to Microbiology.
Immunology, Mycobacterium tuberculosis, Cytokine, SOCS3 and Thioredoxin are his primary areas of study. His work on Mycobacterium tuberculosis is being expanded to include thematically relevant topics such as Immune system. His Immune system study incorporates themes from Myeloid and Tumor necrosis factor alpha.
His Tumor necrosis factor alpha research is multidisciplinary, incorporating perspectives in Progenitor cell, Granuloma, CD3 and Virology. His study in SOCS3 is interdisciplinary in nature, drawing from both Chemokine, Interleukin 6, Bronchoalveolar lavage and CCL5. His Thioredoxin study also includes fields such as
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Tuberculosis and HIV co-infection.
Andrzej Pawlowski;Marianne Jansson;Marianne Jansson;Markus Sköld;Martin E. Rottenberg.
PLOS Pathogens (2012)
Conformation-dependent Antibacterial Activity of the Naturally Occurring Human Peptide LL-37
Jan Johansson;Gudmundur H. Gudmundsson;Martı́n E. Rottenberg;Kurt D. Berndt.
Journal of Biological Chemistry (1998)
Expression patterns of NKG2A, KIR, and CD57 define a process of CD56dim NK-cell differentiation uncoupled from NK-cell education
Niklas K. Björkström;Peggy Riese;Frank Heuts;Sandra Andersson.
Neutrophil secretion products pave the way for inflammatory monocytes
Oliver Soehnlein;Alma Zernecke;Einar E. Eriksson;Antonio Gigliotti Rothfuchs.
SOCS3, a major regulator of infection and inflammation
Berit Carow;Martin E. Rottenberg.
Frontiers in Immunology (2014)
The role of IFN-γ in the outcome of chlamydial infection
Martı́n E Rottenberg;Antonio Gigliotti-Rothfuchs;Hans Wigzell.
Current Opinion in Immunology (2002)
Role of innate and adaptive immunity in the outcome of primary infection with Chlamydia pneumoniae, as analyzed in genetically modified mice.
Martín E. Rottenberg;Antonio C. Gigliotti Rothfuchs;Dulceaydee Gigliotti;Cecilia Svanholm.
Journal of Immunology (1999)
Bcl-2, bax and p53 expression in B-CLL in relation to in vitro survival and clinical progression
Miguel Aguilar-Santelises;Martín E. Rottenberg;Nongnit Lewin;Håkan Mellstedt.
International Journal of Cancer (1996)
Differential susceptibilities of mice genomically deleted of CD4 and CD8 to infections with Trypanosoma cruzi or Trypanosoma brucei.
M E Rottenberg;M Bakhiet;T Olsson;K Kristensson.
Infection and Immunity (1993)
IFN-alpha beta-dependent, IFN-gamma secretion by bone marrow-derived macrophages controls an intracellular bacterial infection.
Antonio Gigliotti Rothfuchs;Dulceaydee Gigliotti;Karin Palmblad;Ulf Andersson.
Journal of Immunology (2001)
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