What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Gene
- Enzyme
The scientist’s investigation covers issues in Platelet, Immunology, Platelet activation, Inflammation and Molecular biology.
Karlheinz Peter has included themes like Abciximab, Pharmacology and Binding site in his Platelet study.
Karlheinz Peter focuses mostly in the field of Immunology, narrowing it down to topics relating to Thrombosis and, in certain cases, Pulmonary embolism, Thrombolysis and Bleeding time.
The Platelet activation study combines topics in areas such as Platelet adhesiveness, Pathology, Molecular imaging, In vivo and Phage display.
His research integrates issues of Gene expression profiling, microRNA, Macrophage-1 antigen and Cell biology in his study of Inflammation.
His Molecular biology research includes elements of Integrin and Effector.
His most cited work include:
- Integrin cytoplasmic domains mediate inside-out signal transduction (607 citations)
- Integrin cytoplasmic domains mediate inside-out signal transduction (607 citations)
- Engineering multifunctional capsules through the assembly of metal-phenolic networks (360 citations)
What are the main themes of his work throughout his whole career to date?
Karlheinz Peter mainly investigates Platelet activation, Platelet, Internal medicine, Inflammation and Pharmacology.
His Platelet activation research focuses on Thrombosis and how it connects with Radiology.
His study on Platelet is covered under Immunology.
He focuses mostly in the field of Immunology, narrowing it down to matters related to Integrin and, in some cases, Molecular biology.
His studies deal with areas such as Endocrinology and Cardiology as well as Internal medicine.
In his work, Cell adhesion is strongly intertwined with Cell biology, which is a subfield of Inflammation.
He most often published in these fields:
- Platelet activation (34.07%)
- Platelet (38.67%)
- Internal medicine (33.15%)
What were the highlights of his more recent work (between 2017-2021)?
- Internal medicine (33.15%)
- Platelet (38.67%)
- Myocardial infarction (23.02%)
In recent papers he was focusing on the following fields of study:
Karlheinz Peter focuses on Internal medicine, Platelet, Myocardial infarction, Cardiology and Platelet activation.
His Internal medicine research is multidisciplinary, relying on both Endocrinology and microRNA.
His work deals with themes such as Inflammation, Thrombus and Cell biology, which intersect with Platelet.
His Inflammation research incorporates elements of Innate immune system, Immune system, Pathology, Stenosis and Monocyte.
His biological study spans a wide range of topics, including Cardiac function curve, Randomized controlled trial and Opioid.
His research investigates the connection between Platelet activation and topics such as Magnetic resonance imaging that intersect with issues in Molecular imaging and Ultrasound.
Between 2017 and 2021, his most popular works were:
- The Emerging Threat of (Micro)Thrombosis in COVID-19 and Its Therapeutic Implications. (109 citations)
- The Emerging Threat of (Micro)Thrombosis in COVID-19 and Its Therapeutic Implications. (109 citations)
- Current and future antiplatelet therapies: emphasis on preserving haemostasis (89 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Gene
- Enzyme
His primary areas of investigation include Platelet, Internal medicine, Myocardial infarction, Inflammation and Reperfusion injury.
Karlheinz Peter studies Platelet activation which is a part of Platelet.
His Platelet activation study is concerned with the field of Immunology as a whole.
In general Myocardial infarction, his work in Percutaneous coronary intervention is often linked to Digital polymerase chain reaction linking many areas of study.
Karlheinz Peter interconnects Endocrinology, Blood pressure, Innate immune system, Immune system and Cell biology in the investigation of issues within Inflammation.
The study incorporates disciplines such as Cardiac function curve, Lipid raft and Reactive oxygen species in addition to Reperfusion injury.
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