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John L. Telford

John L. Telford

D-Index & Metrics

Microbiology

D-Index
77
Citations
24571
World Ranking
1338
National Ranking
32

Overview

John L. Telford is affiliated with GlaxoSmithKline in Italy, where their research contributions focus primarily on medical science, particularly in the field of infectious diseases.

The main areas of study in which they have published include Medicine, with specific emphasis on Public Health, Environmental and Occupational Health, Epidemiology, and Molecular Biology. Their work extensively covers topics related to Neonatal and Maternal Infections, Streptococcal Infections and Treatments, as well as Pneumonia and Respiratory Infections. Additionally, they have engaged in research on vaccines and immunoinformatics approaches.

Their recent papers include the following:

  • Neonatal invasive disease caused by Streptococcus agalactiae in Europe: the DEVANI multi-center study, 2022, Infection
  • Maternal Streptococcus agalactiae colonization in Europe: data from the multi-center DEVANI study, 2024, Infection

These publications focus on Streptococcus agalactiae, examining both neonatal invasive disease and maternal colonization within a European multi-center context.

The research has been published primarily in the journal Infection, signifying a concentrated presence in this venue.

Frequent collaborators in their research include Florens Lohrmann, Markus Hufnagel, Mirjam Kunze, Baharak Afshar, and Roberta Creti. These coauthors have contributed alongside Telford on multiple occasions, indicating ongoing collaborative efforts within their scientific network.

Best Publications

  • Genome analysis of multiple pathogenic isolates of Streptococcus agalactiae: Implications for the microbial “pan-genome”

    Hervé Tettelin;Vega Masignani;Michael J. Cieslewicz;Claudio Donati

  • Helicobacter pylori virulence and genetic geography.

    Antonello Covacci;John L. Telford;Giuseppe Del Giudice;Julie Parsonnet

  • ANALYSIS OF EXPRESSION OF CAGA AND VACA VIRULENCE FACTORS IN 43 STRAINS OF HELICOBACTER PYLORI REVEALS THAT CLINICAL ISOLATES CAN BE DIVIDED INTO TWO MAJOR TYPES AND THAT CAGA IS NOT NECESSARY FOR EXPRESSION OF THE VACUOLATING CYTOTOXIN

    Zhaoying Xiang;Stefano Censini;Pietro F. Bayeli;John L. Telford

  • Gene structure of the Helicobacter pylori cytotoxin and evidence of its key role in gastric disease.

    J L Telford;P Ghiara;M Dell'Orco;M Comanducci

  • Correction: Corrigendum: Streptococcus agalactiae clones infecting humans were selected and fixed through the extensive use of tetracycline

    Violette Da Cunha;Mark R. Davies;Pierre-Emmanuel Douarre;Isabelle Rosinski-Chupin

  • Identification of a universal Group B streptococcus vaccine by multiple genome screen.

    Domenico Maione;Immaculada Margarit;Cira D. Rinaudo;Vega Masignani

  • T helper 1 effector cells specific for Helicobacter pylori in the gastric antrum of patients with peptic ulcer disease.

    M M D'Elios;M Manghetti;M De Carli;F Costa

  • Pili in Gram-positive pathogens

    John L. Telford;Michèle A. Barocchi;Immaculada Margarit;Rino Rappuoli

  • Complete genome sequence and comparative genomic analysis of an emerging human pathogen, serotype V Streptococcus agalactiae

    Hervé Tettelin;Vega Masignani;Michael J. Cieslewicz;Jonathan A. Eisen;Jonathan A. Eisen

  • Characterization and identification of vaccine candidate proteins through analysis of the group A Streptococcus surface proteome

    Manuel J Rodríguez-Ortega;Nathalie Norais;Giuliano Bensi;Sabrina Liberatori

  • Group A Streptococcus produce pilus-like structures containing protective antigens and Lancefield T antigens.

    Marirosa Mora;Giuliano Bensi;Sabrina Capo;Fabiana Falugi

  • The Helicobacter pylori Vacuolating Toxin Inhibits T Cell Activation by Two Independent Mechanisms

    Marianna Boncristiano;Silvia Rossi Paccani;Silvia Barone;Cristina Ulivieri

  • Genome analysis reveals pili in Group B Streptococcus.

    Peter Lauer;Cira D. Rinaudo;Marco Soriani;Immaculada Margarit

  • Formation of anion-selective channels in the cell plasma membrane by the toxin VacA of Helicobacter pylori is required for its biological activity

    Ildikò Szabò;Sandra Brutsche;Francesco Tombola;Monica Moschioni

  • Molecular mimicry between Helicobacter pylori antigens and H+, K+-adenosine triphosphatase in human gastric autoimmunity

    Amedeo Amedei;Mathijs P. Bergman;Ben J. Appelmelk;Annalisa Azzurri

  • Selective increase of the permeability of polarized epithelial cell monolayers by Helicobacter pylori vacuolating toxin.

    E Papini;B Satin;N Norais;M de Bernard

  • The Design of Vaccines Against Helicobacter Pylori and Their Development

    Del Giudice G;Covacci A;Telford Jl;Montecucco C

  • Different cytokine profile and antigen-specificity repertoire in Helicobacter pylori-specific T cell clones from the antrum of chronic gastritis patients with or without peptic ulcer.

    M M D'Elios;M Manghetti;F Almerigogna;A Amedei

  • Streptococcus pyogenes pili promote pharyngeal cell adhesion and biofilm formation.

    Andrea G. O. Manetti;Chiara Zingaretti;Fabiana Falugi;Sabrina Capo

  • Low pH Activates the Vacuolating Toxin of Helicobacter pylori, Which Becomes Acid and Pepsin Resistant

    Marina de Bernard;Emanuele Papini;Vincenzo de Filippis;Elena Gottardi

Frequent Co-Authors

Cosima T. Baldari
Cosima T. Baldari University of Siena
Rino Rappuoli
Rino Rappuoli Imperial College London
Guido Grandi
Guido Grandi University of Trento
Emanuele Papini
Emanuele Papini University of Padua
Cesare Montecucco
Cesare Montecucco University of Padua
Mario Milco D'Elios
Mario Milco D'Elios University of Siena
Amedeo Amedei
Amedeo Amedei University of Florence
Gianfranco Del Prete
Gianfranco Del Prete University of Florence
Antonello Covacci
Antonello Covacci Novartis (Switzerland)
Romano Dallai
Romano Dallai University of Siena

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