D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 80 Citations 26,998 516 World Ranking 9498 National Ranking 428

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Disease
  • Gene

The scientist’s investigation covers issues in Cerebrospinal fluid, Dementia, Alzheimer's disease, Pathology and Internal medicine. His studies deal with areas such as Central nervous system disease, Amyloid β peptide, Molecular biology, Apolipoprotein E and Peptide as well as Cerebrospinal fluid. His Dementia research entails a greater understanding of Disease.

His study in Disease is interdisciplinary in nature, drawing from both PICALM and Genetics. His work in Alzheimer's disease covers topics such as Psychiatry which are related to areas like Proportional hazards model. In his work, Neurology is strongly intertwined with Biomarker, which is a subfield of Pathology.

His most cited work include:

  • Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (2556 citations)
  • Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease (1387 citations)
  • Improved discrimination of AD patients using β-amyloid(1-42) and tau levels in CSF (628 citations)

What are the main themes of his work throughout his whole career to date?

Dementia, Internal medicine, Disease, Cerebrospinal fluid and Pathology are his primary areas of study. His Dementia study combines topics in areas such as Alzheimer's disease, Differential diagnosis and Neurochemical, Neuroscience. His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology, Endocrinology and Oncology.

As part of his studies on Disease, Jens Wiltfang often connects relevant subjects like Cognition. Jens Wiltfang interconnects Apolipoprotein E, Tau protein, Amyloid beta and Immunology in the investigation of issues within Cerebrospinal fluid. As part of his studies on Pathology, he often connects relevant areas like Central nervous system disease.

He most often published in these fields:

  • Dementia (51.20%)
  • Internal medicine (42.98%)
  • Disease (37.55%)

What were the highlights of his more recent work (between 2018-2021)?

  • Dementia (51.20%)
  • Internal medicine (42.98%)
  • Disease (37.55%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Dementia, Internal medicine, Disease, Biomarker and Cognitive decline. His biological study spans a wide range of topics, including Alzheimer's disease, Neuropsychology and Neuroscience, Depression. His research in Internal medicine focuses on subjects like Oncology, which are connected to Atrophy, Neuroimaging and Receiver operating characteristic.

Jens Wiltfang works in the field of Disease, focusing on Amyloid in particular. His work in Biomarker addresses subjects such as Cerebrospinal fluid, which are connected to disciplines such as Apolipoprotein E. His Cognitive decline research also works with subjects such as

  • Clinical psychology and related Schizophrenia,
  • Hippocampal formation and related Hippocampus.

Between 2018 and 2021, his most popular works were:

  • The genetic relationship between educational attainment and cognitive performance in major psychiatric disorders (30 citations)
  • The genetic relationship between educational attainment and cognitive performance in major psychiatric disorders (30 citations)
  • The genetic relationship between educational attainment and cognitive performance in major psychiatric disorders (30 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Gene
  • Disease

His primary scientific interests are in Dementia, Disease, Internal medicine, Biomarker and Cognitive decline. His Dementia research includes themes of Microvesicles, Non-coding RNA, microRNA and RNA. Jens Wiltfang is involved in the study of Disease that focuses on Amyloid in particular.

The study incorporates disciplines such as Quetiapine, Escitalopram and Oncology in addition to Internal medicine. His Biomarker study integrates concerns from other disciplines, such as Amyloid beta, Cerebrospinal fluid, Alzheimer's disease biomarkers, Computational biology and Cohort. In his study, Immunology and Lumbar puncture is inextricably linked to Apolipoprotein E, which falls within the broad field of Cerebrospinal fluid.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (Nature Genetics (2009) 41 (1088-1093))

D Harold;R Abraham;P Hollingworth;R Sims.
Nature Genetics (2013)

2731 Citations

Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease.

Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)

2573 Citations

Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease

Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)

1570 Citations

Improved discrimination of AD patients using β-amyloid(1-42) and tau levels in CSF

F Hulstaert;K Blennow;A Ivanoiu;HC Schoonderwaldt.
Neurology (1999)

869 Citations

Genetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's disease.

Lesley Jones;Peter A. Holmans;Marian L. Hamshere;Denise Harold.
PLOS ONE (2010)

769 Citations

Detection of 14‐3‐3 protein in the cerebrospinal fluid supports the diagnosis of Creutzfeldt‐Jakob disease

Inga Zerr;Monika Bodemer;Olaf Gefeller;Markus Otto.
Annals of Neurology (1998)

526 Citations

Presenilin clinical mutations can affect gamma-secretase activity by different mechanisms.

Mostafa Bentahir;Omar Nyabi;Jan Verhamme;Alexandra Tolia.
Journal of Neurochemistry (2006)

511 Citations

The mechanism of γ-Secretase dysfunction in familial Alzheimer disease

Lucía Chávez-Gutiérrez;Leen Bammens;Iryna Benilova;Annelies Vandersteen;Annelies Vandersteen.
The EMBO Journal (2012)

470 Citations

Social cognition in attention-deficit hyperactivity disorder (ADHD).

J. Uekermann;M. Kraemer;M. Abdel-Hamid;B.G. Schimmelmann.
Neuroscience & Biobehavioral Reviews (2010)

439 Citations

Elevated levels of tau-protein in cerebrospinal fluid of patients with Creutzfeldt–Jakob disease

Markus Otto;Jens Wiltfang;Hayrettin Tumani;Inga Zerr.
Neuroscience Letters (1997)

416 Citations

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