His main research concerns Cancer, Internal medicine, DNA repair, Cancer research and Breast cancer. His Cancer research is multidisciplinary, incorporating perspectives in Missense mutation, Antibiotics, Hereditary diffuse gastric cancer, Genetic counseling and Genetic testing. The concepts of his Internal medicine study are interwoven with issues in Pharmacogenetics and Oncology.
His biological study spans a wide range of topics, including Mutation and Molecular biology. His Cancer research study combines topics from a wide range of disciplines, such as Gene expression profiling, Carcinogenesis, Metastatic breast cancer, Single-cell analysis and Metastasis. His studies deal with areas such as Epithelial–mesenchymal transition, Germline mutation, Circulating tumor cell and Pathology as well as Breast cancer.
Internal medicine, Oncology, Cancer, Breast cancer and Cancer research are his primary areas of study. His research on Internal medicine frequently connects to adjacent areas such as Pathology. While the research belongs to areas of Oncology, James M. Ford spends his time largely on the problem of Iniparib, intersecting his research to questions surrounding Neoadjuvant therapy.
James M. Ford has researched Cancer in several fields, including Genetic counseling and Hereditary diffuse gastric cancer. As part of one scientific family, James M. Ford deals mainly with the area of Breast cancer, narrowing it down to issues related to the Gemcitabine, and often Cisplatin. James M. Ford focuses mostly in the field of Cancer research, narrowing it down to matters related to DNA repair and, in some cases, Molecular biology and Genome instability.
His primary areas of investigation include Internal medicine, Oncology, Cancer, Cancer research and Breast cancer. His work in Internal medicine covers topics such as Multiplex which are related to areas like Cancer screening and Prospective cohort study. James M. Ford combines subjects such as Medical record, Microarray, Cancer registry, Stage and Colorectal cancer with his study of Oncology.
His Cancer research is multidisciplinary, relying on both Computational biology, Gene and Genetic testing. His Cancer research research also works with subjects such as
His primary scientific interests are in Cancer, Cancer research, Internal medicine, Breast cancer and Oncology. His research in Cancer intersects with topics in Precision medicine, Germline mutation, Personalized medicine and Genetic testing. His research integrates issues of DNA damage, PARP inhibitor, Cancer cell, Pathology and In vivo in his study of Cancer research.
James M. Ford studies Medical record, a branch of Internal medicine. His Breast cancer research focuses on Mutation and how it connects with Germline, Mutant, Homologous recombination, Taxane and Chemotherapy. His study in Oncology is interdisciplinary in nature, drawing from both Anthracycline, Cyclophosphamide, Doxorubicin, Cancer registry and Triple-negative breast cancer.
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Pharmacology of drugs that alter multidrug resistance in cancer.
J M Ford;W N Hait.
Pharmacological Reviews (1990)
Expression of the p48 xeroderma pigmentosum gene is p53-dependent and is involved in global genomic repair
Byung Joon Hwang;James M. Ford;Philip C. Hanawalt;Gilbert Chu.
Proceedings of the National Academy of Sciences of the United States of America (1999)
American Society of Clinical Oncology Policy Statement Update: Genetic and Genomic Testing for Cancer Susceptibility
Mark E. Robson;Mark E. Robson;Angela R. Bradbury;Banu K Arun;Susan M. Domchek.
Journal of Clinical Oncology (2010)
Single Cell Profiling of Circulating Tumor Cells: Transcriptional Heterogeneity and Diversity from Breast Cancer Cell Lines
Ashley A. Powell;AmirAli H. Talasaz;Haiyu Zhang;Marc A. Coram.
PLOS ONE (2012)
p53-mediated DNA repair responses to UV radiation: studies of mouse cells lacking p53, p21, and/or gadd45 genes.
Martin L. Smith;James M. Ford;M. Christine Hollander;Rachel A. Bortnick.
Molecular and Cellular Biology (2000)
Germline E-cadherin mutations in hereditary diffuse gastric cancer: assessment of 42 new families and review of genetic screening criteria
A R Brooks-Wilson;P Kaurah;G Suriano;S Leach.
Journal of Medical Genetics (2004)
Clinical Interpretation and Implications of Whole-Genome Sequencing
Frederick E. Dewey;Megan E. Grove;Cuiping Pan;Benjamin A. Goldstein.
JAMA (2014)
Phase I study of stereotactic radiosurgery in patients with locally advanced pancreatic cancer.
Albert C Koong;Quynh T Le;Anthony Ho;Bryan Fong.
International Journal of Radiation Oncology Biology Physics (2004)
Expression of Wild-type p53 Is Required for Efficient Global Genomic Nucleotide Excision Repair in UV-irradiated Human Fibroblasts
James M. Ford;Philip C. Hanawalt.
Journal of Biological Chemistry (1997)
Li-Fraumeni syndrome fibroblasts homozygous for p53 mutations are deficient in global DNA repair but exhibit normal transcription-coupled repair and enhanced UV resistance
James M. Ford;Philip C. Hanawalt.
Proceedings of the National Academy of Sciences of the United States of America (1995)
JCO Precision Oncology
(Impact Factor: 5.479)
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