D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 74 Citations 19,610 233 World Ranking 12490 National Ranking 6619

Overview

What is he best known for?

The fields of study he is best known for:

  • Cancer
  • Gene
  • Internal medicine

His main research concerns Cancer, Internal medicine, DNA repair, Cancer research and Breast cancer. His Cancer research is multidisciplinary, incorporating perspectives in Missense mutation, Antibiotics, Hereditary diffuse gastric cancer, Genetic counseling and Genetic testing. The concepts of his Internal medicine study are interwoven with issues in Pharmacogenetics and Oncology.

His biological study spans a wide range of topics, including Mutation and Molecular biology. His Cancer research study combines topics from a wide range of disciplines, such as Gene expression profiling, Carcinogenesis, Metastatic breast cancer, Single-cell analysis and Metastasis. His studies deal with areas such as Epithelial–mesenchymal transition, Germline mutation, Circulating tumor cell and Pathology as well as Breast cancer.

His most cited work include:

  • Pharmacology of drugs that alter multidrug resistance in cancer. (903 citations)
  • American Society of Clinical Oncology Policy Statement Update: Genetic and Genomic Testing for Cancer Susceptibility (584 citations)
  • Expression of the p48 xeroderma pigmentosum gene is p53-dependent and is involved in global genomic repair (525 citations)

What are the main themes of his work throughout his whole career to date?

Internal medicine, Oncology, Cancer, Breast cancer and Cancer research are his primary areas of study. His research on Internal medicine frequently connects to adjacent areas such as Pathology. While the research belongs to areas of Oncology, James M. Ford spends his time largely on the problem of Iniparib, intersecting his research to questions surrounding Neoadjuvant therapy.

James M. Ford has researched Cancer in several fields, including Genetic counseling and Hereditary diffuse gastric cancer. As part of one scientific family, James M. Ford deals mainly with the area of Breast cancer, narrowing it down to issues related to the Gemcitabine, and often Cisplatin. James M. Ford focuses mostly in the field of Cancer research, narrowing it down to matters related to DNA repair and, in some cases, Molecular biology and Genome instability.

He most often published in these fields:

  • Internal medicine (42.26%)
  • Oncology (34.38%)
  • Cancer (30.71%)

What were the highlights of his more recent work (between 2016-2021)?

  • Internal medicine (42.26%)
  • Oncology (34.38%)
  • Cancer (30.71%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Internal medicine, Oncology, Cancer, Cancer research and Breast cancer. His work in Internal medicine covers topics such as Multiplex which are related to areas like Cancer screening and Prospective cohort study. James M. Ford combines subjects such as Medical record, Microarray, Cancer registry, Stage and Colorectal cancer with his study of Oncology.

His Cancer research is multidisciplinary, relying on both Computational biology, Gene and Genetic testing. His Cancer research research also works with subjects such as

  • PARP inhibitor which is related to area like Talazoparib,
  • DNA damage which is related to area like DNA repair and Cancer cell. The study incorporates disciplines such as Mutation, Ovarian cancer and Chemotherapy in addition to Breast cancer.

Between 2016 and 2021, his most popular works were:

  • Long-read genome sequencing identifies causal structural variation in a Mendelian disease (113 citations)
  • NCCN guidelines® insights: Genetic/Familial High-Risk Assessment: Colorectal, version 3.2017 featured updates to the NCCN Guidelines (111 citations)
  • The Human Tumor Atlas Network: Charting Tumor Transitions Across Space and Time at Single-Cell Resolution (65 citations)

In his most recent research, the most cited papers focused on:

  • Cancer
  • Gene
  • Internal medicine

His primary scientific interests are in Cancer, Cancer research, Internal medicine, Breast cancer and Oncology. His research in Cancer intersects with topics in Precision medicine, Germline mutation, Personalized medicine and Genetic testing. His research integrates issues of DNA damage, PARP inhibitor, Cancer cell, Pathology and In vivo in his study of Cancer research.

James M. Ford studies Medical record, a branch of Internal medicine. His Breast cancer research focuses on Mutation and how it connects with Germline, Mutant, Homologous recombination, Taxane and Chemotherapy. His study in Oncology is interdisciplinary in nature, drawing from both Anthracycline, Cyclophosphamide, Doxorubicin, Cancer registry and Triple-negative breast cancer.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Pharmacology of drugs that alter multidrug resistance in cancer.

J M Ford;W N Hait.
Pharmacological Reviews (1990)

1372 Citations

Expression of the p48 xeroderma pigmentosum gene is p53-dependent and is involved in global genomic repair

Byung Joon Hwang;James M. Ford;Philip C. Hanawalt;Gilbert Chu.
Proceedings of the National Academy of Sciences of the United States of America (1999)

711 Citations

American Society of Clinical Oncology Policy Statement Update: Genetic and Genomic Testing for Cancer Susceptibility

Mark E. Robson;Mark E. Robson;Angela R. Bradbury;Banu K Arun;Susan M. Domchek.
Journal of Clinical Oncology (2010)

584 Citations

Single Cell Profiling of Circulating Tumor Cells: Transcriptional Heterogeneity and Diversity from Breast Cancer Cell Lines

Ashley A. Powell;AmirAli H. Talasaz;Haiyu Zhang;Marc A. Coram.
PLOS ONE (2012)

559 Citations

p53-mediated DNA repair responses to UV radiation: studies of mouse cells lacking p53, p21, and/or gadd45 genes.

Martin L. Smith;James M. Ford;M. Christine Hollander;Rachel A. Bortnick.
Molecular and Cellular Biology (2000)

537 Citations

Germline E-cadherin mutations in hereditary diffuse gastric cancer: assessment of 42 new families and review of genetic screening criteria

A R Brooks-Wilson;P Kaurah;G Suriano;S Leach.
Journal of Medical Genetics (2004)

462 Citations

Clinical Interpretation and Implications of Whole-Genome Sequencing

Frederick E. Dewey;Megan E. Grove;Cuiping Pan;Benjamin A. Goldstein.
JAMA (2014)

453 Citations

Phase I study of stereotactic radiosurgery in patients with locally advanced pancreatic cancer.

Albert C Koong;Quynh T Le;Anthony Ho;Bryan Fong.
International Journal of Radiation Oncology Biology Physics (2004)

452 Citations

Expression of Wild-type p53 Is Required for Efficient Global Genomic Nucleotide Excision Repair in UV-irradiated Human Fibroblasts

James M. Ford;Philip C. Hanawalt.
Journal of Biological Chemistry (1997)

452 Citations

Li-Fraumeni syndrome fibroblasts homozygous for p53 mutations are deficient in global DNA repair but exhibit normal transcription-coupled repair and enhanced UV resistance

James M. Ford;Philip C. Hanawalt.
Proceedings of the National Academy of Sciences of the United States of America (1995)

443 Citations

Editorial Boards

JCO Precision Oncology
(Impact Factor: 5.479)

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