Ikuo Nishimoto focuses on Molecular biology, G protein, Biochemistry, Cell biology and Amyloid precursor protein. His work deals with themes such as Humanin, Mutation, Mutant, Neuroprotection and G protein-coupled receptor kinase, which intersect with Molecular biology. His G protein research is mostly focused on the topic Pertussis toxin.
His study in the fields of Heterotrimeric G protein, Receptor, Acetylcholine receptor and PI3K/AKT/mTOR pathway under the domain of Biochemistry overlaps with other disciplines such as GTP'. His Cell biology research incorporates elements of Internal medicine and Endocrinology. Ikuo Nishimoto interconnects Vesicle and Gene isoform in the investigation of issues within Amyloid precursor protein.
His primary areas of investigation include Molecular biology, Cell biology, Programmed cell death, Humanin and Biochemistry. His Molecular biology research includes themes of Adenylyl cyclase, Mutant, Transfection, COS cells and Pertussis toxin. He is involved in the study of Cell biology that focuses on G protein in particular.
Ikuo Nishimoto combines subjects such as Neurotoxicity, Amyloid precursor protein, Gene, Disease and Kinase with his study of Programmed cell death. The study incorporates disciplines such as Mutation and Amyloid in addition to Amyloid precursor protein. His studies deal with areas such as Presenilin, Neuroprotection and Function as well as Humanin.
His primary areas of study are Humanin, Programmed cell death, Neuroprotection, Cell biology and Molecular biology. His study in Humanin is interdisciplinary in nature, drawing from both Receptor, NADPH oxidase, In vivo and Function. His Programmed cell death study integrates concerns from other disciplines, such as Amino acid, Neurotoxicity, Endocrinology and Internal medicine.
The various areas that Ikuo Nishimoto examines in his Neuroprotection study include Neurotrophic factors and Mutant, SOD1. Ikuo Nishimoto has included themes like Apoptosis and Amyloid precursor protein in his Cell biology study. He has researched Molecular biology in several fields, including Small interfering RNA, Pertussis toxin, Gene, Cytotoxicity and MAPK/ERK pathway.
His scientific interests lie mostly in Humanin, Neuroprotection, Programmed cell death, Molecular biology and Cell biology. His Humanin study combines topics from a wide range of disciplines, such as Proteasome, Neurotoxicity and Protein family. His Neuroprotection research is multidisciplinary, incorporating perspectives in In vivo, Gene, Function and Amyloid precursor protein.
His Programmed cell death research integrates issues from Endocrinology, Serine, Glutamate receptor, Neurotrophic factors and Internal medicine. His Molecular biology research is multidisciplinary, incorporating elements of Ring finger, Protein degradation, Small interfering RNA, Pertussis toxin and Kinase. As part of his studies on Cell biology, Ikuo Nishimoto frequently links adjacent subjects like Receptor.
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Molecular Cloning of Caveolin-3, a Novel Member of the Caveolin Gene Family Expressed Predominantly in Muscle
ZhaoLan Tang;Philipp E. Scherer;Takashi Okamoto;Kenneth Song.
Journal of Biological Chemistry (1996)
Evidence for a Regulated Interaction between Heterotrimeric G Proteins and Caveolin
Shengwen Li;Takashi Okamoto;Miyoung Chun;Massimo Sargiacomo.
Journal of Biological Chemistry (1995)
Identification, sequence, and expression of caveolin-2 defines a caveolin gene family
Philipp E. Scherer;Takashi Okamoto;Miyoung Chun;Ikuo Nishimoto.
Proceedings of the National Academy of Sciences of the United States of America (1996)
Regulation of eIF-4E BP1 Phosphorylation by mTOR
Kenta Hara;Kazuyoshi Yonezawa;Mark T. Kozlowski;Tadanori Sugimoto.
Journal of Biological Chemistry (1997)
A rescue factor abolishing neuronal cell death by a wide spectrum of familial Alzheimer's disease genes and Aβ
Yuichi Hashimoto;Takako Niikura;Hirohisa Tajima;Takashi Yasukawa.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Alzheimer amyloid protein precursor complexes with brain GTP-binding protein Go
Ikuo Nishimoto;Ikuo Nishimoto;Takashi Okamoto;Takashi Okamoto;Yoshiharu Matsuura;Shuji Takahashi.
Nature (1993)
Identification of a Gs activator region of the β2-adrenergic receptor that is autoregulated via protein kinase A-dependent phosphorylation
Takashi Okamoto;Yoshitake Murayama;Yujiro Hayashi;Masaki Inagaki.
Cell (1991)
Interaction between the Alzheimer's survival peptide humanin and insulin-like growth factor-binding protein 3 regulates cell survival and apoptosis.
Maaria Ikonen;Bingrong Liu;Yuichi Hashimoto;Liqun Ma.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Detection of G protein-activator regions in M4 subtype muscarinic, cholinergic, and alpha 2-adrenergic receptors based upon characteristics in primary structure.
T Okamoto;I Nishimoto.
Journal of Biological Chemistry (1992)
G Protein-Mediated Neuronal DNA Fragmentation Induced by Familial Alzheimer's Disease-Associated Mutants of APP
Tomoki Yamatsuji;Takashi Matsui;Takashi Okamoto;Katsumi Komatsuzaki.
Science (1996)
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