D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 59 Citations 12,914 122 World Ranking 8324 National Ranking 595

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Apoptosis

Ikuo Nishimoto focuses on Molecular biology, G protein, Biochemistry, Cell biology and Amyloid precursor protein. His work deals with themes such as Humanin, Mutation, Mutant, Neuroprotection and G protein-coupled receptor kinase, which intersect with Molecular biology. His G protein research is mostly focused on the topic Pertussis toxin.

His study in the fields of Heterotrimeric G protein, Receptor, Acetylcholine receptor and PI3K/AKT/mTOR pathway under the domain of Biochemistry overlaps with other disciplines such as GTP'. His Cell biology research incorporates elements of Internal medicine and Endocrinology. Ikuo Nishimoto interconnects Vesicle and Gene isoform in the investigation of issues within Amyloid precursor protein.

His most cited work include:

  • Molecular Cloning of Caveolin-3, a Novel Member of the Caveolin Gene Family Expressed Predominantly in Muscle (666 citations)
  • Evidence for a Regulated Interaction between Heterotrimeric G Proteins and Caveolin (568 citations)
  • Identification, sequence, and expression of caveolin-2 defines a caveolin gene family (512 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Molecular biology, Cell biology, Programmed cell death, Humanin and Biochemistry. His Molecular biology research includes themes of Adenylyl cyclase, Mutant, Transfection, COS cells and Pertussis toxin. He is involved in the study of Cell biology that focuses on G protein in particular.

Ikuo Nishimoto combines subjects such as Neurotoxicity, Amyloid precursor protein, Gene, Disease and Kinase with his study of Programmed cell death. The study incorporates disciplines such as Mutation and Amyloid in addition to Amyloid precursor protein. His studies deal with areas such as Presenilin, Neuroprotection and Function as well as Humanin.

He most often published in these fields:

  • Molecular biology (45.19%)
  • Cell biology (38.52%)
  • Programmed cell death (31.11%)

What were the highlights of his more recent work (between 2002-2015)?

  • Humanin (23.70%)
  • Programmed cell death (31.11%)
  • Neuroprotection (20.00%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Humanin, Programmed cell death, Neuroprotection, Cell biology and Molecular biology. His study in Humanin is interdisciplinary in nature, drawing from both Receptor, NADPH oxidase, In vivo and Function. His Programmed cell death study integrates concerns from other disciplines, such as Amino acid, Neurotoxicity, Endocrinology and Internal medicine.

The various areas that Ikuo Nishimoto examines in his Neuroprotection study include Neurotrophic factors and Mutant, SOD1. Ikuo Nishimoto has included themes like Apoptosis and Amyloid precursor protein in his Cell biology study. He has researched Molecular biology in several fields, including Small interfering RNA, Pertussis toxin, Gene, Cytotoxicity and MAPK/ERK pathway.

Between 2002 and 2015, his most popular works were:

  • Interaction between the Alzheimer's survival peptide humanin and insulin-like growth factor-binding protein 3 regulates cell survival and apoptosis. (207 citations)
  • D-serine is a key determinant of glutamate toxicity in amyotrophic lateral sclerosis (204 citations)
  • Characterization of Amyotrophic Lateral Sclerosis-linked P56S Mutation of Vesicle-associated Membrane Protein-associated Protein B (VAPB/ALS8) (153 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Apoptosis

His scientific interests lie mostly in Humanin, Neuroprotection, Programmed cell death, Molecular biology and Cell biology. His Humanin study combines topics from a wide range of disciplines, such as Proteasome, Neurotoxicity and Protein family. His Neuroprotection research is multidisciplinary, incorporating perspectives in In vivo, Gene, Function and Amyloid precursor protein.

His Programmed cell death research integrates issues from Endocrinology, Serine, Glutamate receptor, Neurotrophic factors and Internal medicine. His Molecular biology research is multidisciplinary, incorporating elements of Ring finger, Protein degradation, Small interfering RNA, Pertussis toxin and Kinase. As part of his studies on Cell biology, Ikuo Nishimoto frequently links adjacent subjects like Receptor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Molecular Cloning of Caveolin-3, a Novel Member of the Caveolin Gene Family Expressed Predominantly in Muscle

ZhaoLan Tang;Philipp E. Scherer;Takashi Okamoto;Kenneth Song.
Journal of Biological Chemistry (1996)

935 Citations

Evidence for a Regulated Interaction between Heterotrimeric G Proteins and Caveolin

Shengwen Li;Takashi Okamoto;Miyoung Chun;Massimo Sargiacomo.
Journal of Biological Chemistry (1995)

796 Citations

Identification, sequence, and expression of caveolin-2 defines a caveolin gene family

Philipp E. Scherer;Takashi Okamoto;Miyoung Chun;Ikuo Nishimoto.
Proceedings of the National Academy of Sciences of the United States of America (1996)

721 Citations

Regulation of eIF-4E BP1 Phosphorylation by mTOR

Kenta Hara;Kazuyoshi Yonezawa;Mark T. Kozlowski;Tadanori Sugimoto.
Journal of Biological Chemistry (1997)

665 Citations

A rescue factor abolishing neuronal cell death by a wide spectrum of familial Alzheimer's disease genes and Aβ

Yuichi Hashimoto;Takako Niikura;Hirohisa Tajima;Takashi Yasukawa.
Proceedings of the National Academy of Sciences of the United States of America (2001)

624 Citations

Alzheimer amyloid protein precursor complexes with brain GTP-binding protein Go

Ikuo Nishimoto;Ikuo Nishimoto;Takashi Okamoto;Takashi Okamoto;Yoshiharu Matsuura;Shuji Takahashi.
Nature (1993)

555 Citations

Identification of a Gs activator region of the β2-adrenergic receptor that is autoregulated via protein kinase A-dependent phosphorylation

Takashi Okamoto;Yoshitake Murayama;Yujiro Hayashi;Masaki Inagaki.
Cell (1991)

386 Citations

Interaction between the Alzheimer's survival peptide humanin and insulin-like growth factor-binding protein 3 regulates cell survival and apoptosis.

Maaria Ikonen;Bingrong Liu;Yuichi Hashimoto;Liqun Ma.
Proceedings of the National Academy of Sciences of the United States of America (2003)

349 Citations

Detection of G protein-activator regions in M4 subtype muscarinic, cholinergic, and alpha 2-adrenergic receptors based upon characteristics in primary structure.

T Okamoto;I Nishimoto.
Journal of Biological Chemistry (1992)

296 Citations

G Protein-Mediated Neuronal DNA Fragmentation Induced by Familial Alzheimer's Disease-Associated Mutants of APP

Tomoki Yamatsuji;Takashi Matsui;Takashi Okamoto;Katsumi Komatsuzaki.
Science (1996)

275 Citations

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