Cell biology and Stem cell are frequently intertwined in his study. His Stem cell study frequently links to other fields, such as Cell biology. His Genetics study frequently links to adjacent areas such as Cell growth. Hisashi Harada performs integrative study on Gene and Transfection. His work blends Transfection and Gene studies together. Biochemistry and Phosphorylation are commonly linked in his work. In his works, he performs multidisciplinary study on Phosphorylation and Protein kinase A. His multidisciplinary approach integrates Protein kinase A and Kinase in his work. His Kinase study frequently draws connections between adjacent fields such as Biochemistry.
Other disciplines of study, such as Catalysis and Sonochemistry, are mixed together with his Organic chemistry studies. Hisashi Harada combines Catalysis and Organic chemistry in his research. He integrates Gene and Gene expression in his studies. In his articles, he combines various disciplines, including Gene expression and Gene. Hisashi Harada combines Biochemistry and Enzyme in his studies. Hisashi Harada integrates Enzyme and Biochemistry in his research. While working on this project, he studies both Genetics and Molecular biology. While working in this field, he studies both Molecular biology and Genetics. Cell biology is closely attributed to Kinase in his work.
Hisashi Harada merges Cancer research with Lung cancer in his research. His Lung cancer study frequently links to related topics such as Oncology. In his works, Hisashi Harada undertakes multidisciplinary study on Oncology and Cancer research. He undertakes multidisciplinary studies into Gene and Cisplatin in his work. Cisplatin and Apoptosis are two areas of study in which he engages in interdisciplinary work. He performs integrative study on Apoptosis and Downregulation and upregulation in his works. He performs integrative study on Downregulation and upregulation and Gene in his works. While working on this project, Hisashi Harada studies both Cancer and Cell culture. Hisashi Harada performs integrative study on Cell culture and Cancer.
Phenotype and Druggability are closely tied to his Gene research. In his articles, he combines various disciplines, including Cancer research and Lung cancer. Hisashi Harada performs multidisciplinary studies into Lung cancer and Small-cell carcinoma in his work. His Small-cell carcinoma study typically links adjacent topics like Small Cell Lung Carcinoma. His research links Internal medicine with Small Cell Lung Carcinoma. Internal medicine and Oncology are two areas of study in which Hisashi Harada engages in interdisciplinary research. He undertakes multidisciplinary investigations into Oncology and Cancer research in his work. By researching both Cancer and Immunology, Hisashi Harada produces research that crosses academic boundaries. Hisashi Harada undertakes interdisciplinary study in the fields of Immunology and Cancer through his works.
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Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Serine Phosphorylation of Death Agonist BAD in Response to Survival Factor Results in Binding to 14-3-3 Not BCL-XL
Jiping Zha;Hisashi Harada;Elizabeth Yang;Jennifer Jockel.
Complementary DNA for a novel human interleukin (BSF-2) that induces B lymphocytes to produce immunoglobulin
Toshio Hirano;Kiyoshi Yasukawa;Hisashi Harada;Tetsuya Taga.
Regulated expression of a gene encoding a nuclear factor, IRF-1, that specifically binds to IFN-β gene regulatory elements
Masaaki Miyamoto;Takashi Fujita;Yoko Kimura;Mitsuo Maruyama.
Structurally similar but functionally distinct factors, IRF-1 and IRF-2, bind to the same regulatory elements of IFN and IFN-inducible genes
Hisashi Harada;Takashi Fujita;Masaaki Miyamoto;Yoko Kimura.
Requirement for transcription factor IRF-1 in NO synthase induction in macrophages.
R. Kamijo;H. Harada;T. Matsuyama;Maarten C. Bosland.
Phosphorylation and Inactivation of BAD by Mitochondria-Anchored Protein Kinase A
Hisashi Harada;Brian Becknell;Matthias Wilm;Matthias Mann.
Molecular Cell (1999)
p70S6 kinase signals cell survival as well as growth, inactivating the pro-apoptotic molecule BAD
Hisashi Harada;Jens S. Andersen;Matthias Mann;Naohiro Terada.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Cellular commitment to oncogene-induced transformation or apoptosis is dependent on the transcription factor IRF-1
Nobuyuki Tanaka;Masahiko Ishihara;Motoo Kitagawa;Hisashi Harada.
Anti-Oncogenic and Oncogenic Potentials of Interferon Regulatory Factors-1 and -2
Hisashi Harada;Motoo Kitagawa;Nobuyuki Tanaka;Hitomi Yamamoto.
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