His primary areas of investigation include Mitochondrion, Cell biology, mitochondrial fusion, Mitochondrial fission and Translocase of the inner membrane. Hiromi Sesaki has researched Mitochondrion in several fields, including GTPase, Neurodegeneration, DNAJA3, Organelle and Mitophagy. His Cell biology research includes elements of Cardiolipin and Dynamin.
His work deals with themes such as Missense mutation, Mitosis and Saccharomyces cerevisiae, Yeast, which intersect with mitochondrial fusion. Hiromi Sesaki focuses mostly in the field of Mitochondrial fission, narrowing it down to topics relating to Cell division and, in certain cases, Mitochondrial membrane fusion, Organelle fission, Mutant and Lipid bilayer fusion. He interconnects Translocase of the outer membrane and Mitochondrial carrier in the investigation of issues within Translocase of the inner membrane.
Hiromi Sesaki mainly investigates Cell biology, Mitochondrion, Mitochondrial fission, mitochondrial fusion and Inner mitochondrial membrane. The Cell biology study combines topics in areas such as DNAJA3 and Mitochondrial carrier. The concepts of his Mitochondrion study are interwoven with issues in Programmed cell death, Organelle and Mitophagy.
His Programmed cell death study often links to related topics such as Autophagy. His work carried out in the field of Mitochondrial fission brings together such families of science as Oxidative stress, Endocrinology, Saccharomyces cerevisiae, Cell division and Mitochondrial DNA. The various areas that Hiromi Sesaki examines in his Inner mitochondrial membrane study include Cardiolipin and Phosphatidic acid.
Cell biology, Mitochondrion, Mitochondrial fission, Programmed cell death and Inner mitochondrial membrane are his primary areas of study. His Cell biology study frequently draws connections to other fields, such as mitochondrial fusion. His studies in Mitochondrion integrate themes in fields like Cell growth, Apoptosis, Mitochondrial DNA, Organelle and Mitophagy.
His Programmed cell death research includes themes of Embryonic stem cell, Crosstalk and Autolysosome. Dynamin, Translocase of the inner membrane, ATP–ADP translocase and Mitochondrial membrane transport protein is closely connected to Phosphatidic acid in his research, which is encompassed under the umbrella topic of Inner mitochondrial membrane. His Autophagy research integrates issues from Computational biology and DNM1L.
The scientist’s investigation covers issues in Cell biology, Mitochondrion, Mitochondrial fission, Programmed cell death and Mitophagy. In his study, which falls under the umbrella issue of Cell biology, Mitochondrial carrier is strongly linked to mitochondrial fusion. His work in Mitochondrion covers topics such as Mitochondrial DNA which are related to areas like Apoptosis, Inner membrane, Spinal muscular atrophy and Neurodegeneration.
The study incorporates disciplines such as Glycolysis, Flux, KRAS and Homeostasis in addition to Mitochondrial fission. Hiromi Sesaki has included themes like Biophysics, Computational biology and Calcium in his Programmed cell death study. As part of the same scientific family, he usually focuses on Mitophagy, concentrating on Parkin and intersecting with Mitochondrial Size and Ubiquitin ligase.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Division versus Fusion: Dnm1p and Fzo1p Antagonistically Regulate Mitochondrial Shape
Hiromi Sesaki;Robert E. Jensen.
Journal of Cell Biology (1999)
The dynamin-related GTPase Drp1 is required for embryonic and brain development in mice
Junko Wakabayashi;Zhongyan Zhang;Nobunao Wakabayashi;Yasushi Tamura.
Journal of Cell Biology (2009)
Direct Membrane Association Drives Mitochondrial Fission by the Parkinson Disease-associated Protein α-Synuclein
Ken Nakamura;Venu M. Nemani;Farnaz Azarbal;Gaia Skibinski.
Journal of Biological Chemistry (2011)
Mitochondrial Dynamics Controls T Cell Fate through Metabolic Programming
Michael D D. Buck;Michael D D. Buck;David O'Sullivan;Ramon I I. Klein Geltink;Jonathan D D. Curtis.
Cell (2016)
Mitochondrial dynamics in neurodegeneration
Kie Itoh;Ken Nakamura;Miho Iijima;Hiromi Sesaki.
Trends in Cell Biology (2013)
A mitochondrial origin for frontotemporal dementia and amyotrophic lateral sclerosis through CHCHD10 involvement
Sylvie Bannwarth;Samira Ait-El-Mkadem;Annabelle Chaussenot;Emmanuelle C. Genin.
Brain (2014)
Mgm1p, a Dynamin-related GTPase, Is Essential for Fusion of the Mitochondrial Outer Membrane
Hiromi Sesaki;Sheryl M. Southard;Michael P. Yaffe;Robert E. Jensen.
Molecular Biology of the Cell (2003)
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