What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Gene
- Surgery
His scientific interests lie mostly in Fibrodysplasia ossificans progressiva, Heterotopic ossification, Pathology, ACVR1 and Ossification.
His Fibrodysplasia ossificans progressiva study incorporates themes from Genetic disorder, Disease, Myositis ossificans, Endochondral ossification and Bone morphogenetic protein.
Frederick S. Kaplan studied Heterotopic ossification and Penetrance that intersect with Dermatology, Pseudopseudohypoparathyroidism and STX16.
The Pathology study which covers Stem cell that intersects with Aplastic anemia, Bone marrow, Transplantation and Myocyte.
His ACVR1 research includes themes of Mutation, Chondrogenesis, Connective tissue and Anatomy.
His Ossification study integrates concerns from other disciplines, such as Heart valve, Progressive osseous heteroplasia and Bone remodeling.
His most cited work include:
- A recurrent mutation in the BMP type I receptor ACVR1 causes inherited and sporadic fibrodysplasia ossificans progressiva (840 citations)
- Bone Formation and Inflammation in Cardiac Valves (821 citations)
- Treatment of Osteoporosis: Are Physicians Missing an Opportunity?*† (325 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Fibrodysplasia ossificans progressiva, Heterotopic ossification, ACVR1, Pathology and Bone morphogenetic protein.
Frederick S. Kaplan has included themes like Internal medicine, Myositis ossificans, Endochondral ossification and Endocrinology in his Fibrodysplasia ossificans progressiva study.
The study incorporates disciplines such as Ossification, Genetic disorder, Disease and Progressive osseous heteroplasia in addition to Heterotopic ossification.
His ACVR1 research is multidisciplinary, relying on both Mutation, BMPR2, BMP signaling pathway and Bioinformatics.
His Pathology study combines topics from a wide range of disciplines, such as Progenitor cell, Stem cell and Cartilage.
His Bone morphogenetic protein research is multidisciplinary, incorporating elements of Cancer research, Activin receptor, Signal transduction, Cell biology and Bone morphogenetic protein 2.
He most often published in these fields:
- Fibrodysplasia ossificans progressiva (72.01%)
- Heterotopic ossification (47.78%)
- ACVR1 (29.01%)
What were the highlights of his more recent work (between 2013-2021)?
- Fibrodysplasia ossificans progressiva (72.01%)
- Heterotopic ossification (47.78%)
- ACVR1 (29.01%)
In recent papers he was focusing on the following fields of study:
Frederick S. Kaplan spends much of his time researching Fibrodysplasia ossificans progressiva, Heterotopic ossification, ACVR1, Bone morphogenetic protein and Myositis ossificans.
His Fibrodysplasia ossificans progressiva research includes elements of Dermatology, Natural history study, Internal medicine, Endochondral ossification and Pediatrics.
Frederick S. Kaplan combines subjects such as Ossification and Genetic disorder, Disease, Pathology with his study of Heterotopic ossification.
His work deals with themes such as Mast cell and Progressive osseous heteroplasia, which intersect with Pathology.
His study in ACVR1 is interdisciplinary in nature, drawing from both Mutation, Cancer research, Clinical trial and Connective tissue.
His biological study spans a wide range of topics, including Progenitor cell, Signal transduction, Stem cell, Cell biology and Activin receptor.
Between 2013 and 2021, his most popular works were:
- Palovarotene Inhibits Heterotopic Ossification and Maintains Limb Mobility and Growth in Mice With the Human ACVR1(R206H) Fibrodysplasia Ossificans Progressiva (FOP) Mutation. (90 citations)
- The Natural History of Flare-Ups in Fibrodysplasia Ossificans Progressiva (FOP): A Comprehensive Global Assessment. (86 citations)
- Alk2 regulates early chondrogenic fate in fibrodysplasia ossificans progressiva heterotopic endochondral ossification. (75 citations)
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