Edwin A. Deitch mainly investigates Bacteria, Pathology, Microbiology, Mesenteric lymph nodes and Chromosomal translocation. His Bacteria research is multidisciplinary, incorporating elements of Surgery and Shock. In general Surgery, his work in Sepsis and Peritoneal cavity is often linked to Glasgow Outcome Scale linking many areas of study.
Edwin A. Deitch combines subjects such as Gastroenterology, Lung injury and Permeability with his study of Pathology. His Microbiology research is multidisciplinary, relying on both Xanthine oxidase, In vivo and Malnutrition. His Chromosomal translocation research includes elements of Internal medicine, Intestinal permeability, Ratón and Bacteria Present.
His primary areas of investigation include Immunology, Internal medicine, Pathology, Lymph and Shock. As a part of the same scientific family, Edwin A. Deitch mostly works in the field of Immunology, focusing on Chromosomal translocation and, on occasion, Mesenteric lymph nodes and Ratón. His work is dedicated to discovering how Internal medicine, Endocrinology are connected with Chemokine and Neutrophile and other disciplines.
His work carried out in the field of Pathology brings together such families of science as Pharmacology and Hemorrhagic shock. The concepts of his Shock study are interwoven with issues in Resuscitation, Multiple organ dysfunction syndrome and Pathogenesis. His studies in Sepsis integrate themes in fields like Microbiology and Intensive care medicine.
His scientific interests lie mostly in Immunology, Internal medicine, Sepsis, Inflammation and Shock. His Immunology research focuses on Human umbilical vein endothelial cell and how it connects with Microcirculation. His Internal medicine research incorporates themes from Endocrinology and Severe trauma.
His research in Sepsis intersects with topics in Proinflammatory cytokine, Adenosine, Adenosine receptor and Bacterial translocation. His study in Inflammation is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha and Resuscitation. His biological study spans a wide range of topics, including Respiratory burst, Lymph, Pathology and Multiple organ dysfunction syndrome.
Edwin A. Deitch focuses on Sepsis, Immunology, Inflammation, Cytokine and Lung injury. Edwin A. Deitch has researched Sepsis in several fields, including Immune system, Cannabinoid receptor type 2, Intensive care medicine, Knockout mouse and Endocannabinoid system. The various areas that Edwin A. Deitch examines in his Immunology study include Apoptosis, Adenosine and Adenosine receptor.
Edwin A. Deitch interconnects Hsp70, TRIF and Lymph in the investigation of issues within Inflammation. The Cytokine study which covers Tumor necrosis factor alpha that intersects with HMGB1 and Tyrosine phosphorylation. His research integrates issues of Heat shock protein, Anesthesia, Toll-like receptor, Bronchoalveolar lavage and Tidal volume in his study of Lung injury.
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Multiple organ failure. Pathophysiology and potential future therapy.
Edwin A. Deitch.
Annals of Surgery (1992)
Animal models of sepsis and shock : A review and lessons learned
Edwin A. Deitch.
Hypertrophic burn scars: analysis of variables.
Edwin A. Deitch;Timothy M. Wheelahan;Mary Paige Rose;Jill Clothier.
Journal of Trauma-injury Infection and Critical Care (1983)
Role of the Gut in Multiple Organ Failure: Bacterial Translocation and Permeability Changes
Gregory M. Swank;Edwin A. Deitch.
World Journal of Surgery (1996)
MicroRNA-145, a novel smooth muscle cell phenotypic marker and modulator, controls vascular neointimal lesion formation.
Yunhui Cheng;Xiaojun Liu;Jian Yang;Ying Lin.
Circulation Research (2009)
The Role of Intestinal Barrier Failure and Bacterial Translocation in the Development of Systemic Infection and Multiple Organ Failure
Edwin A. Deitch.
Archives of Surgery (1990)
The gut as a portal of entry for bacteremia. Role of protein malnutrition
Edwin A. Deitch;John Winterton;M A Li;Rodney Berg.
Annals of Surgery (1987)
Endotoxin promotes the translocation of bacteria from the gut.
Edwin A. Deitch;Rodney Berg;Robert Specian.
Archives of Surgery (1987)
Adenosine inhibits IL-12 and TNF-[alpha] production via adenosine A2a receptor-dependent and independent mechanisms
György Haskó;David G. Kuhel;Jiang Fan Chen;Michael A. Schwarzschild.
The FASEB Journal (2000)
Hemorrhagic shock induces bacterial translocation from the gut.
John Wilder Baker;Edwin A. Deitch;M A Li;Rodney D. Berg.
Journal of Trauma-injury Infection and Critical Care (1988)
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