What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His main research concerns Poly ADP ribose polymerase, Molecular biology, Biochemistry, Peroxynitrite and Immunology.
He has included themes like Programmed cell death, DNA damage, NAD+ kinase and DNA repair in his Poly ADP ribose polymerase study.
His biological study spans a wide range of topics, including Adenosine receptor and Interleukin 10.
His Peroxynitrite research incorporates elements of Oxidative stress, Tyrosine, Nitrotyrosine and Nitric oxide.
His studies deal with areas such as Internal medicine, PARP inhibitor and Endocrinology as well as Immunology.
The PARG study combines topics in areas such as Apoptosis and Poly glycohydrolase.
His most cited work include:
- The Therapeutic Potential of Poly(ADP-Ribose) Polymerase Inhibitors (1229 citations)
- Diabetic endothelial dysfunction: the role of poly(ADP-ribose) polymerase activation. (531 citations)
- Peroxynitrite-induced cytotoxicity: mechanism and opportunities for intervention (385 citations)
What are the main themes of his work throughout his whole career to date?
László Virág mainly focuses on Poly ADP ribose polymerase, Internal medicine, Molecular biology, Pharmacology and Biochemistry.
His Poly ADP ribose polymerase study combines topics in areas such as Inflammation, Oxidative stress, DNA damage and Programmed cell death.
His Programmed cell death study incorporates themes from PARP1 and Cell biology.
His research integrates issues of Endocrinology and Cardiology in his study of Internal medicine.
His study in Molecular biology is interdisciplinary in nature, drawing from both HaCaT, Immunology, Kinase, DNA fragmentation and PARG.
As part of the same scientific family, he usually focuses on Pharmacology, concentrating on Repolarization and intersecting with Inward-rectifier potassium ion channel, Dofetilide and Ion channel.
He most often published in these fields:
- Poly ADP ribose polymerase (37.26%)
- Internal medicine (29.66%)
- Molecular biology (29.66%)
What were the highlights of his more recent work (between 2017-2021)?
- Pharmacology (22.81%)
- Internal medicine (29.66%)
- Cell biology (17.87%)
In recent papers he was focusing on the following fields of study:
László Virág focuses on Pharmacology, Internal medicine, Cell biology, Cardiology and Programmed cell death.
His Pharmacology study combines topics from a wide range of disciplines, such as hERG, Electrophysiology, Ventricular myocardium and Mexiletine.
His research in Internal medicine focuses on subjects like Endocrinology, which are connected to Mir 133a.
The concepts of his Cell biology study are interwoven with issues in Inflammation, Promoter, Necroptosis, Regulator and Transcription.
His Programmed cell death study introduces a deeper knowledge of Apoptosis.
In his works, László Virág undertakes multidisciplinary study on Ciclopirox and Poly ADP ribose polymerase.
Between 2017 and 2021, his most popular works were:
- Programmed necrotic cell death of macrophages: Focus on pyroptosis, necroptosis, and parthanatos. (52 citations)
- Redox control of cancer cell destruction. (46 citations)
- Development, calibration, and validation of a novel human ventricular myocyte model in health, disease, and drug block (36 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
László Virág mainly investigates Cell biology, Programmed cell death, Pharmacology, Inflammation and hERG.
His Cell biology research includes elements of Transcription, Gene, Repressor and Cell.
His Programmed cell death study contributes to a more complete understanding of Apoptosis.
The various areas that László Virág examines in his Apoptosis study include Anaerobic glycolysis and Warburg effect.
His Pharmacology research is multidisciplinary, incorporating perspectives in Dofetilide, Wound healing, Lipid peroxidation, Verapamil and Nitrotyrosine.
His studies in Inflammation integrate themes in fields like Peroxynitrite, Phorbol, Antioxidant and AP-1 transcription factor.
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