His primary areas of investigation include Molecular biology, Proto-Oncogene Proteins c-rel, Transcription factor, Enhancer and Signal transduction. Dean W. Ballard has included themes like Transcription, Gene expression, Gene, T cell and Receptor in his Molecular biology study. The various areas that Dean W. Ballard examines in his Proto-Oncogene Proteins c-rel study include Serine, Ubiquitin, Lysine and T-cell receptor.
The study incorporates disciplines such as Virus, Leukemia, T-cell leukemia and Virology in addition to Transcription factor. His work deals with themes such as Protein subunit and DNA-binding protein, which intersect with Enhancer. His studies deal with areas such as Cancer research, breakpoint cluster region, Cytokine and Phosphorylation as well as Signal transduction.
His primary areas of study are Molecular biology, Transcription factor, Cell biology, Signal transduction and Phosphorylation. His research in Molecular biology intersects with topics in Transactivation, Enhancer, Proto-Oncogene Proteins c-rel, Gene and Antibody. Dean W. Ballard studied Transcription factor and Virology that intersect with Leukemia.
As a part of the same scientific family, Dean W. Ballard mostly works in the field of Signal transduction, focusing on NFKB1 and, on occasion, Tumor necrosis factor alpha. His Phosphorylation study combines topics in areas such as Proinflammatory cytokine and Ubiquitin. His IκB kinase research includes themes of Cancer research, Protein subunit and Kinase.
Dean W. Ballard focuses on Transcription factor, Signal transduction, Molecular biology, IκB kinase and Phosphorylation. His Transcription factor study integrates concerns from other disciplines, such as Proinflammatory cytokine, Acquired immune system, NF-κB and Cancer research. His study in Signal transduction is interdisciplinary in nature, drawing from both Innate immune system, Kinase and Programmed cell death.
His Molecular biology research is multidisciplinary, incorporating elements of Cell culture and Gene. His biological study deals with issues like Ubiquitin, which deal with fields such as Proteasome. His Cell biology research includes elements of NFKB1 and Immunology.
His scientific interests lie mostly in Molecular biology, Transcription factor, Signal transduction, IκB kinase and NF-κB. His studies in Molecular biology integrate themes in fields like Proteasome inhibitor, Cell culture, Apoptosis, HeLa and NFKB1. His Cell culture research is multidisciplinary, incorporating perspectives in Cell, Cytokine, Carcinogenesis, Oncogene and Programmed cell death.
His studies examine the connections between Signal transduction and genetics, as well as such issues in Cancer research, with regards to I-Kappa-B Kinase, Tyrosine kinase, X-linked agammaglobulinemia, breakpoint cluster region and B-cell receptor. The concepts of his IκB kinase study are interwoven with issues in Cell cycle, IκBα, Phosphorylation and Small interfering RNA. Dean W. Ballard has researched NF-κB in several fields, including Protein subunit and Kinase.
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NF-κB controls expression of inhibitor IκBα : evidence for an inducible autoregulatory pathway
Shao Cong Sun;Parham A. Ganchi;Dean W. Ballard;Warner C. Greene.
Suppression of tumor necrosis factor-induced cell death by inhibitor of apoptosis c-IAP2 is under NF-κB control
Zhi-Liang Chu;Timothy A. McKinsey;Lily Liu;Jennifer J. Gentry.
Proceedings of the National Academy of Sciences of the United States of America (1997)
Coupling of a signal response domain in I kappa B alpha to multiple pathways for NF-kappa B activation.
J. A. Brockman;D. C. Scherer;T. A. Mckinsey;S. M. Hall.
Molecular and Cellular Biology (1995)
Cross-coupling of the NF-kappa B p65 and Fos/Jun transcription factors produces potentiated biological function.
Bernd Stein;Albert S. Baldwin;Dean W. Ballard;Warner C. Greene.
The EMBO Journal (1993)
Signal-induced degradation of I kappa B alpha requires site-specific ubiquitination.
David C. Scherer;Jeffrey A. Brockman;Zhijian Chen;Tom Maniatis.
Proceedings of the National Academy of Sciences of the United States of America (1995)
HTLV-I tax induces cellular proteins that activate the kappa B element in the IL-2 receptor alpha gene.
DW Ballard;E Bohnlein;JW Lowenthal;Y Wano.
The same inducible nuclear proteins regulates mitogen activation of both the interleukin-2 receptor-alpha gene and type 1 HIV.
Ernst Böhnlein;John W. Lowenthal;Miriam Siekevitz;Dean W. Ballard.
Constitutive Activation of NF-κB in Primary Adult T-Cell Leukemia Cells
Naoki Mori;Masahiro Fujii;Shuichi Ikeda;Yasuaki Yamada.
Kappa B-specific DNA binding proteins: role in the regulation of human interleukin-2 gene expression
Beatrice Hoyos;Dean W. Ballard;Ernst Böhnlein;Miriam Siekevitz.
Bruton's Tyrosine Kinase Is Required for Activation of Iκb Kinase and Nuclear Factor κb in Response to B Cell Receptor Engagement
James B. Petro;S.M. Jamshedur Rahman;Dean W. Ballard;Wasif N. Khan.
Journal of Experimental Medicine (2000)
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