D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 100 Citations 47,909 390 World Ranking 437 National Ranking 259
Medicine D-index 113 Citations 54,591 520 World Ranking 2812 National Ranking 1614

Research.com Recognitions

Awards & Achievements

1987 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

Caleb E. Finch spends much of his time researching Internal medicine, Endocrinology, Neuroscience, Alzheimer's disease and Senescence. His work in Endocrinology addresses issues such as Messenger RNA, which are connected to fields such as RNA, Human brain and Molecular biology. His Neuroscience research incorporates elements of Synaptic plasticity, Cytokine and In situ hybridization.

His Alzheimer's disease study integrates concerns from other disciplines, such as Clusterin, Biochemistry, Amyloid and Degenerative disease. His research in Senescence focuses on subjects like Longevity, which are connected to Inflammation. Caleb E. Finch has researched Astrocyte in several fields, including Hippocampal formation, Microglia and Neuroglia.

His most cited work include:

  • Inflammation and Alzheimer's disease. (3451 citations)
  • Diffusible, nonfibrillar ligands derived from Aβ1–42 are potent central nervous system neurotoxins (3068 citations)
  • Longevity, senescence, and the genome (1275 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Internal medicine, Endocrinology, Neuroscience, Disease and Alzheimer's disease. His study focuses on the intersection of Internal medicine and fields such as Messenger RNA with connections in the field of Gene expression. His Endocrinology study focuses mostly on Estrous cycle, Hippocampus, Astrocyte, Hypothalamus and Dopaminergic.

He works mostly in the field of Hippocampus, limiting it down to topics relating to Hippocampal formation and, in certain cases, Molecular biology and Glucocorticoid. As a part of the same scientific study, Caleb E. Finch usually deals with the Astrocyte, concentrating on Neuroglia and frequently concerns with Microglia. Caleb E. Finch has included themes like Inflammation, Degenerative disease and Amyloid in his Alzheimer's disease study.

He most often published in these fields:

  • Internal medicine (31.24%)
  • Endocrinology (30.02%)
  • Neuroscience (14.49%)

What were the highlights of his more recent work (between 2012-2021)?

  • Disease (9.08%)
  • Internal medicine (31.24%)
  • Apolipoprotein E (6.11%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Disease, Internal medicine, Apolipoprotein E, Cell biology and Endocrinology. His studies in Internal medicine integrate themes in fields like Neurogenesis and Cardiology. His work investigates the relationship between Apolipoprotein E and topics such as Cerebral cortex that intersect with problems in Neurite and Hippocampus.

His Cell biology study incorporates themes from Oxidative stress, Caenorhabditis elegans and Microglia. Caleb E. Finch combines subjects such as Long-term potentiation and Neurotrophin with his study of Endocrinology. The study incorporates disciplines such as Neuroscience and Cerebral amyloid angiopathy, Amyloid in addition to Alzheimer's disease.

Between 2012 and 2021, his most popular works were:

  • Atherosclerosis across 4000 years of human history: the Horus study of four ancient populations (226 citations)
  • Vascular dysfunction-The disregarded partner of Alzheimer's disease (175 citations)
  • Particulate air pollutants, APOE alleles and their contributions to cognitive impairment in older women and to amyloidogenesis in experimental models. (153 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

His primary areas of study are Disease, Internal medicine, Neuroscience, Endocrinology and Cell biology. His study in Disease is interdisciplinary in nature, drawing from both Demography, Cognition, Immunology, Heart disease and Risk factor. The various areas that he examines in his Neuroscience study include Alzheimer's disease, Neurite and Gorilla.

His work deals with themes such as Sex characteristics, Amyloid beta and Cerebral amyloid angiopathy, Amyloid, which intersect with Alzheimer's disease. His study in the field of Apolipoprotein B, Neuroendocrinology and Hypothalamus is also linked to topics like Comparative perspective. His studies deal with areas such as Oxidative stress, Basal, Neuroglia, Microglia and Receptor as well as Cell biology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Inflammation and Alzheimer's disease.

H Akiyama;S Barger;S Barnum;B Bradt.
Neurobiology of Aging (2000)

5119 Citations

Diffusible, nonfibrillar ligands derived from Aβ1–42 are potent central nervous system neurotoxins

M. P. Lambert;A. K. Barlow;B. A. Chromy;C. Edwards.
Proceedings of the National Academy of Sciences of the United States of America (1998)

4346 Citations

Longevity, senescence, and the genome

Caleb Ellicott Finch.
(1990)

2073 Citations

Expression of a noncoding RNA is elevated in Alzheimer's disease and drives rapid feed-forward regulation of β-secretase

Mohammad Ali Faghihi;Mohammad Ali Faghihi;Farzaneh Modarresi;Ahmad M Khalil;Douglas E Wood.
Nature Medicine (2008)

1403 Citations

Alzheimer's disease-affected brain: Presence of oligomeric Aβ ligands (ADDLs) suggests a molecular basis for reversible memory loss

Yuesong Gong;Lei Chang;Kirsten L. Viola;Pascale N. Lacor.
Proceedings of the National Academy of Sciences of the United States of America (2003)

1367 Citations

Targeting small Aβ oligomers: the solution to an Alzheimer's disease conundrum?

William L Klein;Grant A Krafft;Caleb E Finch.
Trends in Neurosciences (2001)

1339 Citations

Handbook of the Biology of Aging

Caleb Ellicott Finch;Edward L. Schneider.
(1990)

1222 Citations

Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers

Pascale N. Lacor;Maria C. Buniel;Lei Chang;Sara J. Fernandez.
The Journal of Neuroscience (2004)

1189 Citations

Inflammatory Exposure and Historical Changes in Human Life-Spans

Caleb E. Finch;Eileen M. Crimmins.
Science (2004)

859 Citations

Cytokines and cognition--the case for a head-to-toe inflammatory paradigm.

Craig J. Wilson;Caleb E. Finch;Harvey J. Cohen;Harvey J. Cohen.
Journal of the American Geriatrics Society (2002)

816 Citations

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