1987 - Fellow of the American Association for the Advancement of Science (AAAS)
Caleb E. Finch spends much of his time researching Internal medicine, Endocrinology, Neuroscience, Alzheimer's disease and Senescence. His work in Endocrinology addresses issues such as Messenger RNA, which are connected to fields such as RNA, Human brain and Molecular biology. His Neuroscience research incorporates elements of Synaptic plasticity, Cytokine and In situ hybridization.
His Alzheimer's disease study integrates concerns from other disciplines, such as Clusterin, Biochemistry, Amyloid and Degenerative disease. His research in Senescence focuses on subjects like Longevity, which are connected to Inflammation. Caleb E. Finch has researched Astrocyte in several fields, including Hippocampal formation, Microglia and Neuroglia.
His primary areas of study are Internal medicine, Endocrinology, Neuroscience, Disease and Alzheimer's disease. His study focuses on the intersection of Internal medicine and fields such as Messenger RNA with connections in the field of Gene expression. His Endocrinology study focuses mostly on Estrous cycle, Hippocampus, Astrocyte, Hypothalamus and Dopaminergic.
He works mostly in the field of Hippocampus, limiting it down to topics relating to Hippocampal formation and, in certain cases, Molecular biology and Glucocorticoid. As a part of the same scientific study, Caleb E. Finch usually deals with the Astrocyte, concentrating on Neuroglia and frequently concerns with Microglia. Caleb E. Finch has included themes like Inflammation, Degenerative disease and Amyloid in his Alzheimer's disease study.
The scientist’s investigation covers issues in Disease, Internal medicine, Apolipoprotein E, Cell biology and Endocrinology. His studies in Internal medicine integrate themes in fields like Neurogenesis and Cardiology. His work investigates the relationship between Apolipoprotein E and topics such as Cerebral cortex that intersect with problems in Neurite and Hippocampus.
His Cell biology study incorporates themes from Oxidative stress, Caenorhabditis elegans and Microglia. Caleb E. Finch combines subjects such as Long-term potentiation and Neurotrophin with his study of Endocrinology. The study incorporates disciplines such as Neuroscience and Cerebral amyloid angiopathy, Amyloid in addition to Alzheimer's disease.
His primary areas of study are Disease, Internal medicine, Neuroscience, Endocrinology and Cell biology. His study in Disease is interdisciplinary in nature, drawing from both Demography, Cognition, Immunology, Heart disease and Risk factor. The various areas that he examines in his Neuroscience study include Alzheimer's disease, Neurite and Gorilla.
His work deals with themes such as Sex characteristics, Amyloid beta and Cerebral amyloid angiopathy, Amyloid, which intersect with Alzheimer's disease. His study in the field of Apolipoprotein B, Neuroendocrinology and Hypothalamus is also linked to topics like Comparative perspective. His studies deal with areas such as Oxidative stress, Basal, Neuroglia, Microglia and Receptor as well as Cell biology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Inflammation and Alzheimer's disease.
H Akiyama;S Barger;S Barnum;B Bradt.
Neurobiology of Aging (2000)
Diffusible, nonfibrillar ligands derived from Aβ1–42 are potent central nervous system neurotoxins
M. P. Lambert;A. K. Barlow;B. A. Chromy;C. Edwards.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Longevity, senescence, and the genome
Caleb Ellicott Finch.
(1990)
Expression of a noncoding RNA is elevated in Alzheimer's disease and drives rapid feed-forward regulation of β-secretase
Mohammad Ali Faghihi;Mohammad Ali Faghihi;Farzaneh Modarresi;Ahmad M Khalil;Douglas E Wood.
Nature Medicine (2008)
Alzheimer's disease-affected brain: Presence of oligomeric Aβ ligands (ADDLs) suggests a molecular basis for reversible memory loss
Yuesong Gong;Lei Chang;Kirsten L. Viola;Pascale N. Lacor.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Targeting small Aβ oligomers: the solution to an Alzheimer's disease conundrum?
William L Klein;Grant A Krafft;Caleb E Finch.
Trends in Neurosciences (2001)
Handbook of the Biology of Aging
Caleb Ellicott Finch;Edward L. Schneider.
(1990)
Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers
Pascale N. Lacor;Maria C. Buniel;Lei Chang;Sara J. Fernandez.
The Journal of Neuroscience (2004)
Inflammatory Exposure and Historical Changes in Human Life-Spans
Caleb E. Finch;Eileen M. Crimmins.
Science (2004)
Cytokines and cognition--the case for a head-to-toe inflammatory paradigm.
Craig J. Wilson;Caleb E. Finch;Harvey J. Cohen;Harvey J. Cohen.
Journal of the American Geriatrics Society (2002)
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