2023 - Research.com Biology and Biochemistry in Switzerland Leader Award
Beat W. Schäfer focuses on Cancer research, Gene, Genetics, Pathology and Molecular biology. In his study, Immunology and Chemotherapy is inextricably linked to Gene expression profiling, which falls within the broad field of Cancer research. His Gene research incorporates elements of Cell culture, Calcium-binding protein and S100 protein.
Beat W. Schäfer has included themes like Cancer and Cell growth in his Pathology study. His Molecular biology study combines topics from a wide range of disciplines, such as Carcinogenesis, Candidate Tumor Suppressor Gene, Rhabdomyosarcoma, Alveolar rhabdomyosarcoma and Regulation of gene expression. His work focuses on many connections between Rhabdomyosarcoma and other disciplines, such as Ectopic expression, that overlap with his field of interest in Cell biology.
His main research concerns Cancer research, Molecular biology, Rhabdomyosarcoma, Pathology and Cell biology. The various areas that he examines in his Cancer research study include Cell culture, Transcription factor, Immunology, Alveolar rhabdomyosarcoma and Sarcoma. His Molecular biology study integrates concerns from other disciplines, such as Gene expression, Transfection, Complementary DNA, Messenger RNA and Gene.
Gene is a subfield of Genetics that Beat W. Schäfer investigates. His Pathology research includes themes of Cancer and Metastasis. The concepts of his Cell biology study are interwoven with issues in Cell and Calcium-binding protein.
His main research concerns Cancer research, Rhabdomyosarcoma, Alveolar rhabdomyosarcoma, Cell biology and Transcription factor. His Cancer research research is multidisciplinary, incorporating perspectives in Epigenetics, Bromodomain, BRD4, Gene silencing and PI3K/AKT/mTOR pathway. His work deals with themes such as Insulin-like growth factor 1 receptor, Stem cell, Immunology and Furin, which intersect with Rhabdomyosarcoma.
His work carried out in the field of Alveolar rhabdomyosarcoma brings together such families of science as Carcinogenesis, Phenotype, Kinase and Bioinformatics. His study in the field of Myogenic differentiation also crosses realms of Block. His Transcription factor research also works with subjects such as
The scientist’s investigation covers issues in Cancer research, Alveolar rhabdomyosarcoma, Transcription factor, Fusion protein and Cell biology. His studies deal with areas such as Gene expression, Immunology, Signal transduction, Rhabdomyosarcoma and Kinase as well as Cancer research. His Gene expression research integrates issues from PI3K/AKT/mTOR pathway and Transcription.
His research in Alveolar rhabdomyosarcoma intersects with topics in Mutation, Loss function, Mutant and Pax genes. The subject of his Transcription factor research is within the realm of Genetics. His work in Cell biology addresses issues such as Carcinogenesis, which are connected to fields such as Molecular biology, Hedgehog signaling pathway, GLI1, Hedgehog and Stem cell.
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The S100 family of EF-hand calcium-binding proteins: functions and pathology.
Beat W. Schäfer;Claus W. Heizmann.
Trends in Biochemical Sciences (1996)
S100 proteins: structure, functions and pathology.
Claus W Heizmann;Günter Fritz;Beat W Schäfer.
Frontiers in Bioscience (2002)
Minimal residual disease-directed risk stratification using real-time quantitative PCR analysis of immunoglobulin and T-cell receptor gene rearrangements in the international multicenter trial AIEOP-BFM ALL 2000 for childhood acute lymphoblastic leukemia.
T Flohr;T Flohr;A Schrauder;G Cazzaniga;R Panzer-Grümayer.
The MLL recombinome of acute leukemias in 2013
C. Meyer;J. Hofmann;T. Burmeister;D. Gröger.
New insights to the MLL recombinome of acute leukemias
C. Meyer;E. Kowarz;J. Hofmann;A. Renneville.
Isolation of a YAC clone covering a cluster of nine S100 genes on human chromosome 1q21: rationale for a new nomenclature of the S100 calcium-binding protein family
Beat W. Schäfer;Roland Wicki;Dieter Engelkamp;Marie-geneviève Mattei.
The MLL recombinome of acute leukemias in 2017
C Meyer;T Burmeister;D Gröger;G Tsaur.
Late MRD response determines relapse risk overall and in subsets of childhood T-cell ALL: results of the AIEOP-BFM-ALL 2000 study
Martin Schrappe;Maria Grazia Valsecchi;Claus R. Bartram;André Schrauder.
Induction of autophagy-dependent necroptosis is required for childhood acute lymphoblastic leukemia cells to overcome glucocorticoid resistance
Laura Bonapace;Beat C. Bornhauser;Maike Schmitz;Gunnar Cario.
Journal of Clinical Investigation (2010)
Expression pattern of S100 calcium‐binding proteins in human tumors
Evelyn C. Ilg;Beat W. Schäfer;Claus W. Heizmann.
International Journal of Cancer (1996)
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