Genetics, Cell biology, Spinal and bulbar muscular atrophy, Genome-wide association study and Androgen receptor are his primary areas of study. His Genetics research includes elements of Alzheimer's disease and Disease. His Cell biology research incorporates elements of Interpretation and Biochemistry.
His Spinal and bulbar muscular atrophy research is multidisciplinary, relying on both Cancer research, Neurodegeneration, Mutant protein, Mutant and Receptor. Andrew P. Lieberman has researched Androgen receptor in several fields, including Endocrinology, Skeletal muscle, Genetically modified mouse and Polyglutamine tract. His Bioinformatics research integrates issues from Chaperone-mediated autophagy and Autolysosome.
His primary areas of study are Cell biology, Androgen receptor, Spinal and bulbar muscular atrophy, Pathology and Internal medicine. His biological study spans a wide range of topics, including Ubiquitin, Neurodegeneration and Niemann–Pick disease, type C. His studies deal with areas such as Transgene, Mutant, Receptor, Motor neuron and Neuroscience as well as Androgen receptor.
As a part of the same scientific study, Andrew P. Lieberman usually deals with the Neuroscience, concentrating on Disease and frequently concerns with Genetics and Bioinformatics. The study incorporates disciplines such as Neuromuscular disease, Molecular biology, Polyglutamine tract and Muscle atrophy, Skeletal muscle in addition to Spinal and bulbar muscular atrophy. Andrew P. Lieberman combines subjects such as Endocrinology and Oncology with his study of Internal medicine.
His primary scientific interests are in Cell biology, Androgen receptor, Spinal and bulbar muscular atrophy, Disease and Immune system. The various areas that Andrew P. Lieberman examines in his Cell biology study include Neurodegeneration and Skeletal muscle. The Androgen receptor study combines topics in areas such as Transcriptional activity and Transgene.
His research investigates the connection with Spinal and bulbar muscular atrophy and areas like Ubiquitin which intersect with concerns in Gene knockdown and Huntingtin. As part of one scientific family, Andrew P. Lieberman deals mainly with the area of Disease, narrowing it down to issues related to the Bioinformatics, and often Carrier protein, Alzheimer's disease, Cognitive decline and Ischemia. His Immune system research also works with subjects such as
Andrew P. Lieberman mainly focuses on Cell biology, Neurodegeneration, Spinocerebellar ataxia, Spinal and bulbar muscular atrophy and Disease. His Cell biology research is multidisciplinary, incorporating elements of Lewy body, Hsp90 and Androgen receptor. His Neurodegeneration research incorporates themes from Homeostasis, Pathogenesis, Crosstalk, Endoplasmic reticulum and Regulator.
His Spinocerebellar ataxia research is multidisciplinary, incorporating perspectives in Heat shock protein, Hsp70, Carrier protein, Huntington's disease and Cellular homeostasis. His studies in Spinal and bulbar muscular atrophy integrate themes in fields like Cell type, Bioinformatics and Age of onset. His Candidate Disease Gene research extends to Disease, which is thematically connected.
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Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes
Daniel J. Klionsky;Hagai Abeliovich;Patrizia Agostinis;Devendra K. Agrawal.
Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.
Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)
Analysis of shared heritability in common disorders of the brain
Verneri Anttila;Verneri Anttila;Brendan Bulik-Sullivan;Brendan Bulik-Sullivan;Hilary K. Finucane;Raymond K. Walters;Raymond K. Walters.
Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease
Rebecca Sims;Sven J. Van Der Lee;Adam C. Naj;Céline Bellenguez;Céline Bellenguez.
Nature Genetics (2017)
Identification of common variants influencing risk of the tauopathy progressive supranuclear palsy
Günter U. Höglinger;Nadine M. Melhem;Dennis W. Dickson;Patrick M A Sleiman.
Nature Genetics (2011)
Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions
Vivianna M. Van Deerlin;Patrick M A Sleiman;Maria Martinez-Lage;Maria Martinez-Lage;Alice Chen-Plotkin.
Nature Genetics (2010)
Lipid storage disorders block lysosomal trafficking by inhibiting a TRP channel and lysosomal calcium release
Dongbiao Shen;Xiang Wang;Xinran Li;Xiaoli Zhang.
Nature Communications (2012)
Autophagy in lysosomal storage disorders
Andrew P. Lieberman;Rosa Puertollano;Nina Raben;Susan Ann Slaugenhaupt.
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