D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 62 Citations 34,024 161 World Ranking 6838 National Ranking 3170

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Internal medicine

Genetics, Cell biology, Spinal and bulbar muscular atrophy, Genome-wide association study and Androgen receptor are his primary areas of study. His Genetics research includes elements of Alzheimer's disease and Disease. His Cell biology research incorporates elements of Interpretation and Biochemistry.

His Spinal and bulbar muscular atrophy research is multidisciplinary, relying on both Cancer research, Neurodegeneration, Mutant protein, Mutant and Receptor. Andrew P. Lieberman has researched Androgen receptor in several fields, including Endocrinology, Skeletal muscle, Genetically modified mouse and Polyglutamine tract. His Bioinformatics research integrates issues from Chaperone-mediated autophagy and Autolysosome.

His most cited work include:

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
  • Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
  • Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes (1951 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Cell biology, Androgen receptor, Spinal and bulbar muscular atrophy, Pathology and Internal medicine. His biological study spans a wide range of topics, including Ubiquitin, Neurodegeneration and Niemann–Pick disease, type C. His studies deal with areas such as Transgene, Mutant, Receptor, Motor neuron and Neuroscience as well as Androgen receptor.

As a part of the same scientific study, Andrew P. Lieberman usually deals with the Neuroscience, concentrating on Disease and frequently concerns with Genetics and Bioinformatics. The study incorporates disciplines such as Neuromuscular disease, Molecular biology, Polyglutamine tract and Muscle atrophy, Skeletal muscle in addition to Spinal and bulbar muscular atrophy. Andrew P. Lieberman combines subjects such as Endocrinology and Oncology with his study of Internal medicine.

He most often published in these fields:

  • Cell biology (28.48%)
  • Androgen receptor (20.00%)
  • Spinal and bulbar muscular atrophy (17.58%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (28.48%)
  • Androgen receptor (20.00%)
  • Spinal and bulbar muscular atrophy (17.58%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Cell biology, Androgen receptor, Spinal and bulbar muscular atrophy, Disease and Immune system. The various areas that Andrew P. Lieberman examines in his Cell biology study include Neurodegeneration and Skeletal muscle. The Androgen receptor study combines topics in areas such as Transcriptional activity and Transgene.

His research investigates the connection with Spinal and bulbar muscular atrophy and areas like Ubiquitin which intersect with concerns in Gene knockdown and Huntingtin. As part of one scientific family, Andrew P. Lieberman deals mainly with the area of Disease, narrowing it down to issues related to the Bioinformatics, and often Carrier protein, Alzheimer's disease, Cognitive decline and Ischemia. His Immune system research also works with subjects such as

  • Glioma which is related to area like Oncology and Internal medicine,
  • Cancer research which intersects with area such as Dose escalation.

Between 2018 and 2021, his most popular works were:

  • Polyglutamine Repeats in Neurodegenerative Diseases. (63 citations)
  • Pathogenic alpha-synuclein aggregates preferentially bind to mitochondria and affect cellular respiration (51 citations)
  • First-in-human phase I trial of the combination of two adenoviral vectors expressing HSV1-TK and FLT3L for the treatment of newly diagnosed resectable malignant glioma: Initial results from the therapeutic reprogramming of the brain immune system. (16 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Internal medicine

Andrew P. Lieberman mainly focuses on Cell biology, Neurodegeneration, Spinocerebellar ataxia, Spinal and bulbar muscular atrophy and Disease. His Cell biology research is multidisciplinary, incorporating elements of Lewy body, Hsp90 and Androgen receptor. His Neurodegeneration research incorporates themes from Homeostasis, Pathogenesis, Crosstalk, Endoplasmic reticulum and Regulator.

His Spinocerebellar ataxia research is multidisciplinary, incorporating perspectives in Heat shock protein, Hsp70, Carrier protein, Huntington's disease and Cellular homeostasis. His studies in Spinal and bulbar muscular atrophy integrate themes in fields like Cell type, Bioinformatics and Age of onset. His Candidate Disease Gene research extends to Disease, which is thematically connected.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy

Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)

8302 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)

7788 Citations

Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

Daniel J. Klionsky;Hagai Abeliovich;Patrizia Agostinis;Devendra K. Agrawal.
Autophagy (2008)

2790 Citations

Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.

Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)

1885 Citations

Analysis of shared heritability in common disorders of the brain

Verneri Anttila;Verneri Anttila;Brendan Bulik-Sullivan;Brendan Bulik-Sullivan;Hilary K. Finucane;Raymond K. Walters;Raymond K. Walters.
Science (2018)

1200 Citations

Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease

Rebecca Sims;Sven J. Van Der Lee;Adam C. Naj;Céline Bellenguez;Céline Bellenguez.
Nature Genetics (2017)

683 Citations

Identification of common variants influencing risk of the tauopathy progressive supranuclear palsy

Günter U. Höglinger;Nadine M. Melhem;Dennis W. Dickson;Patrick M A Sleiman.
Nature Genetics (2011)

517 Citations

Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions

Vivianna M. Van Deerlin;Patrick M A Sleiman;Maria Martinez-Lage;Maria Martinez-Lage;Alice Chen-Plotkin.
Nature Genetics (2010)

484 Citations

Lipid storage disorders block lysosomal trafficking by inhibiting a TRP channel and lysosomal calcium release

Dongbiao Shen;Xiang Wang;Xinran Li;Xiaoli Zhang.
Nature Communications (2012)

406 Citations

Autophagy in lysosomal storage disorders

Andrew P. Lieberman;Rosa Puertollano;Nina Raben;Susan Ann Slaugenhaupt.
Autophagy (2012)

406 Citations

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