2005 - Member of the Royal Irish Academy
His primary scientific interests are in Apoptosis, Programmed cell death, Cell biology, Cell and Molecular biology. The Apoptosis study combines topics in areas such as Oxidative stress, Cancer research, Gene expression and Immunology. His work is dedicated to discovering how Cancer research, Chronic myelogenous leukemia are connected with ABL and other disciplines.
The study incorporates disciplines such as Necrosis, Cell culture and Fragmentation in addition to Programmed cell death. His biological study spans a wide range of topics, including Retinal degeneration and Caspase. His Molecular biology research is multidisciplinary, relying on both Antigen-presenting cell, Natural killer T cell, Interleukin 21, CD8 and ZAP70.
Apoptosis, Cell biology, Programmed cell death, Cancer research and Molecular biology are his primary areas of study. His Apoptosis research integrates issues from Cell, Cytotoxic T cell and Cell culture. His work deals with themes such as Photoreceptor cell, Retina and Biochemistry, which intersect with Cell biology.
His Programmed cell death study combines topics in areas such as Camptothecin, Fragmentation, Immunology, Necrosis and Mitochondrion. His Cancer research study incorporates themes from ABL, Chronic myelogenous leukemia, K562 cells, PI3K/AKT/mTOR pathway and Kinase. Antigen is closely connected to Monoclonal antibody in his research, which is encompassed under the umbrella topic of Molecular biology.
Thomas G. Cotter focuses on Cell biology, Reactive oxygen species, Retina, Internal medicine and Signal transduction. The concepts of his Cell biology study are interwoven with issues in Basic fibroblast growth factor and Biochemistry. His Reactive oxygen species research includes elements of Immunology, Oxidative stress, Cell culture and DNA damage.
Thomas G. Cotter studied Cell culture and Cancer research that intersect with Ellipticines and Acute leukemia. The various areas that Thomas G. Cotter examines in his Retinal study include Apoptosis, Programmed cell death, Neuroscience and Anatomy. Thomas G. Cotter undertakes interdisciplinary study in the fields of Apoptosis and Hypopharyngeal Carcinoma through his research.
Thomas G. Cotter mainly investigates Cell biology, Signal transduction, Reactive oxygen species, Biochemistry and NADPH oxidase. His Cell biology research incorporates themes from Retinal degeneration and Retinal. His Reactive oxygen species research includes themes of Oxidative stress, Oxidation reduction, Programmed cell death and Cellular signal transduction.
His Programmed cell death research is multidisciplinary, incorporating elements of Proinflammatory cytokine, PI3K/AKT/mTOR pathway and Angiogenesis. His research in NADPH oxidase intersects with topics in Molecular biology and Fms-Like Tyrosine Kinase 3. His Neuroscience research incorporates elements of Apoptosis and Hormone.
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Dose-dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli
S. V. Lennon;S. J. Martin;T. G. Cotter.
Cell Proliferation (1991)
Apoptosis and cancer: the genesis of a research field.
Thomas G. Cotter.
Nature Reviews Cancer (2009)
Bax-induced Caspase Activation and Apoptosis via Cytochromec Release from Mitochondria Is Inhibitable by Bcl-xL
Deborah M. Finucane;Ella Bossy-Wetzel;Nigel J. Waterhouse;Thomas G. Cotter.
Journal of Biological Chemistry (1999)
Role for c-myc in activation-induced apoptotic cell death in T cell hybridomas
Yufang Shi;Jacqueline M. Glynn;Lawrence J. Guilbert;Thomas G. Cotter.
Induction of apoptosis (programmed cell death) in human leukemic HL-60 cells by inhibition of RNA or protein synthesis
S J Martin;S V Lennon;A M Bonham;T G Cotter.
Journal of Immunology (1990)
Regulation and measurement of oxidative stress in apoptosis.
James F Curtin;Maryanne Donovan;Thomas G Cotter.
Journal of Immunological Methods (2002)
Heat Shock Proteins Increase Resistance to Apoptosis
Afshin Samali;Thomas G. Cotter.
Experimental Cell Research (1996)
BCR-ABL Maintains Resistance of Chronic Myelogenous Leukemia Cells to Apoptotic Cell Death
Anne McGahon;Anne McGahon;Reid Bissonnette;Reid Bissonnette;Manfred Schmitt;Manfred Schmitt;Kate M. Cotter;Kate M. Cotter.
ROS signalling in the biology of cancer.
Jennifer N. Moloney;Thomas G. Cotter.
Seminars in Cell & Developmental Biology (2017)
BCR-ABL maintains resistance of chronic myelogenous leukemia cells to apoptotic cell death [published erratum appears in Blood 1994 Jun 15;83(12):3835]
A McGahon;R Bissonnette;M Schmitt;KM Cotter.
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