His primary areas of investigation include Immunology, Cell biology, Stem cell, Progenitor cell and Intestinal mucosa. Thaddeus S. Stappenbeck combines subjects such as Disease, Inflammatory bowel disease and Microbiology with his study of Immunology. His work deals with themes such as Epithelium and Crypt, which intersect with Cell biology.
The various areas that he examines in his Progenitor cell study include Cell growth and Pathology. His work carried out in the field of Intestinal mucosa brings together such families of science as Intestinal epithelium and Cellular differentiation. His Intestinal epithelium research integrates issues from Autophagy and ATG16L1.
Thaddeus S. Stappenbeck mainly focuses on Cell biology, Immunology, Stem cell, Paneth cell and Intestinal mucosa. The concepts of his Cell biology study are interwoven with issues in Autophagy and Epithelium. His Immunology research is multidisciplinary, relying on both Crohn's disease, Disease and Inflammatory bowel disease.
His Stem cell research also works with subjects such as
Thaddeus S. Stappenbeck focuses on Cell biology, Immune system, Crohn's disease, Stem cell and Cryptosporidium. His studies deal with areas such as Cell culture, Innate immune system, Epithelium and Cell growth as well as Cell biology. His study in Immune system is interdisciplinary in nature, drawing from both Bacteroides thetaiotaomicron and Antigen.
His Crohn's disease research incorporates themes from Gastroenterology and Replication. His Stem cell research is multidisciplinary, incorporating perspectives in In vitro and LGR5. His Dysbiosis study deals with the bigger picture of Immunology.
His main research concerns Cell biology, Stem cell, LGR5, Transgene and Transcriptome. Thaddeus S. Stappenbeck interconnects Macrophage proliferation, Cytokine, Cell growth and MAFB, Transcription factor in the investigation of issues within Cell biology. His Stem cell research incorporates elements of Reporter gene and Conditioned medium.
His work investigates the relationship between LGR5 and topics such as Unfolded protein response that intersect with problems in Colitis and Inflammation. His work focuses on many connections between Transcriptome and other disciplines, such as Tumor necrosis factor alpha, that overlap with his field of interest in Inflammatory bowel disease. The study incorporates disciplines such as Obligate anaerobe, Immunology, Ulcerative colitis, Biopsy and Clostridia in addition to Inflammatory bowel disease.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells
Ken Cadwell;John Y. Liu;Sarah L. Brown;Hiroyuki Miyoshi.
Nature (2008)
Developmental regulation of intestinal angiogenesis by indigenous microbes via Paneth cells.
Thaddeus S. Stappenbeck;Lora V. Hooper;Jeffrey I. Gordon.
Proceedings of the National Academy of Sciences of the United States of America (2002)
Angiogenins: a new class of microbicidal proteins involved in innate immunity
Lora V. Hooper;Thaddeus S. Stappenbeck;Chieu V. Hong;Jeffrey I. Gordon.
Nature Immunology (2003)
A Dietary Fiber-Deprived Gut Microbiota Degrades the Colonic Mucus Barrier and Enhances Pathogen Susceptibility
Mahesh S. Desai;Mahesh S. Desai;Anna M. Seekatz;Nicole M. Koropatkin;Nobuhiko Kamada.
Cell (2016)
Peripheral education of the immune system by colonic commensal microbiota
Stephanie K. Lathrop;Seth M. Bloom;Sindhuja M. Rao;Katherine Nutsch.
Nature (2011)
Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine
Ken Cadwell;Khushbu K. Patel;Nicole S. Maloney;Ta Chiang Liu.
Cell (2010)
Activated macrophages are an adaptive element of the colonic epithelial progenitor niche necessary for regenerative responses to injury.
Sarah L. Pull;Jason M. Doherty;Jason C. Mills;Jeffrey I. Gordon.
Proceedings of the National Academy of Sciences of the United States of America (2005)
Disease-specific alterations in the enteric virome in inflammatory bowel disease.
Jason M. Norman;Scott A. Handley;Megan T. Baldridge;Lindsay Droit.
Cell (2015)
Peripheral CD103+ dendritic cells form a unified subset developmentally related to CD8α+ conventional dendritic cells
Brian T. Edelson;Wumesh Kc;Richard Juang;Masako Kohyama.
Journal of Experimental Medicine (2010)
Multi-omics of the gut microbial ecosystem in inflammatory bowel diseases.
Jason Lloyd-Price;Jason Lloyd-Price;Cesar Arze;Ashwin N. Ananthakrishnan;Melanie Schirmer;Melanie Schirmer.
Nature (2019)
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