What is he best known for?
The fields of study he is best known for:
- Gene
- Immune system
- Cytokine
Simon R. Carding mainly investigates Immunology, T cell, Immune system, T lymphocyte and Cytotoxic T cell.
His research integrates issues of Apoptosis and Inflammatory bowel disease in his study of Immunology.
His work deals with themes such as Antigen and Claudin, Tight junction, Phosphorylation, Cell biology, which intersect with T cell.
Simon R. Carding specializes in Immune system, namely CD8.
His T lymphocyte research is multidisciplinary, incorporating elements of Virology, Streptamer, Gamma/Delta T-Lymphocyte, T-cell receptor and Molecular biology.
In his study, Inflammation is strongly linked to Microbiome, which falls under the umbrella field of Gut flora.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Inflammatory bowel disease: cause and immunobiology (1301 citations)
- Gammadelta T cells: functional plasticity and heterogeneity. (658 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Immunology, Cell biology, Immune system, T cell and Molecular biology.
Immunology and Inflammatory bowel disease are commonly linked in his work.
His Cell biology study integrates concerns from other disciplines, such as Autophagy, Gut flora, Blood–brain barrier, Transcriptome and Paneth cell.
Simon R. Carding has researched T cell in several fields, including Cytotoxic T cell and Virology.
His Molecular biology study which covers Interleukin 2 that intersects with Interleukin 4 and Thymocyte.
His T-cell receptor research includes elements of Receptor and Major histocompatibility complex.
He most often published in these fields:
- Immunology (43.17%)
- Cell biology (35.06%)
- Immune system (29.15%)
What were the highlights of his more recent work (between 2017-2021)?
- Cell biology (35.06%)
- Autophagy (12.55%)
- Immunology (43.17%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Cell biology, Autophagy, Immunology, Immune system and Microbiology.
His biological study spans a wide range of topics, including Transcriptome, Paneth cell, Gut flora and Blood–brain barrier.
Simon R. Carding combines subjects such as Emerging technologies and Inflammatory bowel disease with his study of Immunology.
His studies in Inflammatory bowel disease integrate themes in fields like Cytotoxic T cell, CD8, Memory T cell, Immunity and Humoral immunity.
His study on T cell and Intraepithelial lymphocyte is often connected to Bacterial outer membrane as part of broader study in Immune system.
His Microbiology study incorporates themes from Bacteroides thetaiotaomicron, Bacteroides, Small intestine and Antigen.
Between 2017 and 2021, his most popular works were:
- Microbiome-host systems interactions: protective effects of propionate upon the blood-brain barrier. (108 citations)
- Microbiome-host systems interactions: protective effects of propionate upon the blood-brain barrier. (108 citations)
- The WD40 domain of ATG16L1 is required for its non‐canonical role in lipidation of LC3 at single membranes (92 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Cytokine
Simon R. Carding spends much of his time researching Cell biology, Immune system, Gut flora, Immunology and Bacterial outer membrane.
His Cell biology research incorporates themes from Autophagy, Sequestosome 1, ATG16L1, Receptor and Barrier integrity.
Simon R. Carding has included themes like Increased risk and Gastrointestinal Microbiome in his Immune system study.
His Gut flora research is multidisciplinary, incorporating perspectives in Microbiome, Function, Blood–brain barrier and Brain function.
His work on Vaccine delivery as part of general Immunology research is often related to Unmet needs, thus linking different fields of science.
The concepts of his Inflammatory bowel disease study are interwoven with issues in Cytokine, CD8, Immunity, Humoral immunity and Cytotoxic T cell.
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